Volume 56, Pages S84-S87 (November 1999) Tumor necrosis factor-α during continuous high-flux hemodialysis in sepsis with acute renal failure  Gerhard Lonnemann, M.D., Mikko Bechstein, Silvia Linnenweber, Michael Burg, Karl Martin Koch  Kidney International  Volume 56, Pages S84-S87 (November 1999) DOI: 10.1046/j.1523-1755.56.s72.8.x Copyright © 1999 International Society of Nephrology Terms and Conditions

Figure 1 Endotoxin-induced whole blood production of tumor necrosis factor-alpha (A; TNF-α) and interleukin-10 (B; IL-10) in healthy controls (N = 18) and patients with sepsis and acute renal failure. Kidney International 1999 56, S84-S87DOI: (10.1046/j.1523-1755.56.s72.8.x) Copyright © 1999 International Society of Nephrology Terms and Conditions

Figure 2 Effect of continuous venovenous hemofiltration (▪; CVVH, 1.5 liters/hr) and continuous high flux hemodialysis (• GENIUS®-CHFD, 4.2 liters/hr) on ex vivo whole-blood TNF-α production in septic patients with acute renal failure. Symbols indicate the medians of N = 7 for CVVH and N = 5 for CHFD. Kidney International 1999 56, S84-S87DOI: (10.1046/j.1523-1755.56.s72.8.x) Copyright © 1999 International Society of Nephrology Terms and Conditions

Figure 3 Effect of CVVH (▪; 1.5 liters/hr) and GENIUS®-CHFD (• 4.2 liters/hr) on ex vivo whole-blood production of inhibitory soluble TNF receptor type I (TNFsRI) in septic patients with acute renal failure. Symbols indicate the medians of N = 7 for CVVH and N = 5 for CHFD. Kidney International 1999 56, S84-S87DOI: (10.1046/j.1523-1755.56.s72.8.x) Copyright © 1999 International Society of Nephrology Terms and Conditions