A–F, Schematic drawings illustrating subarachnoid hemosiderosis and superficial cortical hemosiderosis. A–F, Schematic drawings illustrating subarachnoid.

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High-signal-intensity lesions on T2-weighted MR images
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A–F, Schematic drawings illustrating subarachnoid hemosiderosis and superficial cortical hemosiderosis. A–F, Schematic drawings illustrating subarachnoid hemosiderosis and superficial cortical hemosiderosis. A–D, Coronal schematic drawings, illustrating the time-dependent development of subarachnoid hemosiderosis and superficial cortical hemosiderosis (black, subarachnoid space; white line, pia mater; orange line, arachnoid layer; light blue, dura mater). E,F, Horizontal schematic drawings corresponding to the areas marked with black bars in B and C, demonstrating the visual appearance of subarachnoid hemosiderosis and superficial cortical hemosiderosis on axial sections. A, Normal appearance of the subarachnoid space. B,E, Subarachnoid hemorrhage (red) presenting as a linear signal intensity in the subarachnoid space (E). C, Residues of blood penetrating the pia mater and deposit in the superficial layers of the cerebral cortex. D,F, Superficial cortical hemosiderosis, defined as linear residues of blood in the superficial layers of the cerebral cortex (dark red). Superficial cortical hemosiderosis typically has a bilinear “tracklike” appearance on axial sections, caused by the signal intensity of the normal-appearing subarachnoid space in the middle, which is bordered bilaterally by linear deposits of hemosiderin in the superficial layers of the adjacent cortex (F). J. Linn et al. AJNR Am J Neuroradiol 2008;29:184-186 ©2008 by American Society of Neuroradiology