Fig. 5. Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region.

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Structure of abbreviated dystrophins.

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Fig. 2. Effect of endurance training on gene expression, and protein content and activity in heart muscle. Effect of endurance training on gene expression,

Fig. 2. Outline of the two types of stimulus sequences employed in the analysis.(A) Environment information stimuli; (B) adaptation stimuli. Outline of.

Fig. 3. External work and kinetic and potential energy of the whole body for the mean individual (individual 10) in the mean population (population 1)

Representative illustrations of a sagittal lung section stained with H&E in a control and a CS-exposed mouse after 6 months. Representative illustrations.

Fig. 2. Morphological changes of cultured adherent fibroblastic cells after OA treatment related to actin microfilament reorganization.(A) Cells observed.

Fig. 7. Motion adaptation increases time-dependent response modulations (TDRM) relatively to the average cell response.TDRM normalized to the value obtained.

Fig. 2. Proportion of motile objects and track length

Fig. 1. Muscle-specific PITX1 over-expression in the Pitx1 transgenic mice.(A) Detection of Pitx1 mRNA expression in muscles of the Pitx1 transgenic mice.

Fig. 4. A primary screen based on scrape closure

Fig. 6. Cross-section of the stomach wall and spiral intestine of the embryo, stained with PAS. (A) Surface of the stomach wall (SW) and ingested material.

Fig. 6. The cholinergic receptor subunits a6 (Chrna6) and b3 (Chrnb3) are (subset) specifically expressed in mdDA neurons during development.(A) The Chrna6.

Fig. 6. Comparison between the response against transformed tissues and capsule formation.At the cellular level the two responses share many similarities.

Fig. 7. Knockdown of Meis1 abolishes CR4. 2-GFP expression

Fig. 2. Mapping of the interaction domain on coilin for association with the dyskerin complex. Mapping of the interaction domain on coilin for association.

Embryonic corneal defects in E18.5 AP-2β NCC KO mutant embryos.

Fig. 2. Body weight and size analysis of A1/A2-KO mice

Fig. 6. Schematic diagram showing distribution and dynamics of four E-cadherin populations within the ROI of a FRAP experiment. Schematic diagram showing.

Fig. 1. Lack of Hmga1 and Hmga2 expression in A1/A2-KO mice

Fig. 4. The model of malate metabolism in fruit cells under different K level conditions. The model of malate metabolism in fruit cells under different.

Fig. 1. Pigmentation and melanophore counts of rainbow trout parr and smolt caudal fins.Pigmentation of (A) parr and (B) smolt. Pigmentation and melanophore.

Fig. 1. γ-Tubulin localizes in close proximity to centriole walls in interphase but within an extended PCM meshwork in mitosis.U2OS cells were fixed and.

Fig. 8. Knockdown of Meis1 reduces the expression of Foxn4 and Lim1+2

Fig. 3. Characterization of unclassified cells (UCs).

Fig. 7. VCBP-C and chitin localization in stomach sections of adult DSS- and CMP-treated animals. VCBP-C and chitin localization in stomach sections of.

Fig. 2. Two signal-producing behaviours of wild-type Canton-S males and per mutant males relative to whether the wild-type female is moving or immobile.

Expression data confirm and extend existing knowledge

Fig. 2. Morphological analysis of acentriolar mitotic spindles

Fig. 6. Effect of SAHA and ML on histone acetylation, BAX, and p21CDKN1A expression.PANC-1 and BxPC-3 cells were incubated for 48 hours with 5 µM.

Fig. 7. Interaction between BAF60c and cardiac transcription factors.

Fig. 2. Centrosomal proteins display distinct localizations and radial distances from centriole walls.U2OS cells were fixed and stained with the indicated.

Fig. 4. One-dimensional gel electrophoresis of DIMs

Fig. 2. Soluble sugar and organic acid levels with different K fertilization during fruit development. Soluble sugar and organic acid levels with different.

Fig. 1. Generation of WNK3 knockout mice

Fig. 7. Representative images of control (Cas9+GFP) and Cas9+gRNA+GFP co-injected embryos on day 4 of culture, showing nuclear-imported GFP (green) and.

Fig. 1. TSA at 165 nM induces maximal acetylation of histone H3 and near-maximal acetylation of histone H4.Immunoblot analysis of acetylated histones H3.

Fig. 6. STK35 KO mice show ovary defects.

Fig. 4. SMXL6 is degraded in response to SL treatment.

Fig. 2. TSA impairs growth and disrupts morphogenesis of exocrine pancreas in zebrafish larvae with hyperacetylation of nucleosomal histones.WT zebrafish.

Statistical chart of significantly differentially expressed genes

Fig. 2. Sufficient rate of recombination in embryonic NSCs and NPCs of Emx1cre knock-in mice.(A) Breeding schemes to generate mice with the Emx1cre locus.

Fig. 11. Chrna6 and Chrnb3 expression depends Pitx3 and Nurr1 but through a different mechanism reported for Vmat2.(A) QPCR analysis of Chrna6 and Chrnb3.

Fig. 4. Expression analysis of Onecut transcription factors during mdDA neuron development.Adjacent coronal sections of E11.5, E12.5 and E13.5 mouse brains.

Fig. 10. Expression of the cholinergic receptor subunits Chrna6 and Chrnb3 depends on Pitx3.In situ hybridization for Th, Chrna6 and Chrnb3 on sagittal.

Fig. 6. The cholinergic receptor subunits a6 (Chrna6) and b3 (Chrnb3) are (subset) specifically expressed in mdDA neurons during development.(A) The Chrna6.

EpiDEG efficiently degrades GFP-tagged proteins that localize to different subcellular localizations. epiDEG efficiently degrades GFP-tagged proteins that.

Fig. 9. Loss of the cholinergic receptor subunit Chrna6 does not alter the development and organization of the mdDA system.(A) In situ hybridization experiments.

Fig. 3. Enhanced EGFR signaling in Rhbdf2P159L/P159L mice.

Fig. 5. Flatworm density and activity on coral polyps

Amplicon sequencing analysis of on-target sites in trβ crispants

Analysis of mutant Best1 protein (Y227N) in testis and sperm function

Fig. 4. Visual and RPE phagocytic function in Best1Y227N mice.

Fig. 12. Overview of the molecular program essential to build mdDA neurons.The genes identified in this study (in red) have been added to the programming.

Fig. 2. Coupling of actin to cell–cell junctions requires α-catenin and is necessary for the establishment of the barrier.(A) Effect of Cytochalasin D.

Fig. 2. Acetylation stiffens primary cilia.

Fig. 1. Generation of induced pluripotent stem cells (iPSCs) from urine cells (UC). Generation of induced pluripotent stem cells (iPSCs) from urine cells.

Fig. 8. Compared mobilities of passenger proteins in G2/M-prophase, metaphase and anaphase.FRAP experiments were performed on HeLa cells stably expressing.

Fig. 2. Schematic representation of the interactions within the three-party system. Schematic representation of the interactions within the three-party.

Fig. 5. Testis defects in STK35 KO mice.

Expression of smc3 in whole-mount and cryosectioned regenerating fins

Fig. 3. Mean force and velocity during jumping

Fig. 2. Temporal map of genes clustering with the expression profile of Lmx1a.(A) Heat-map visualization obtained by HCL of genes clustering with Lmx1a.

Fig. 1. Microarray analyses of genes whose expression is regulated by innervation during synaptogenesis.(A) Schematic drawings of the experimental design.

Table 1. Measurement of ring diameters of proteins localizing in ring-like patterns around centrioles.Consideration of the size of IgG (about 8 nm) raises.

Fig. 1. Lhx1 is expressed in the proximal region of the OV

Fig. 5. Behaviours of the wild-types Oregon-R at two temperatures.

Fig. 7. Rho family GTPase signaling is required for cyst formation in Ptp4E Ptp10D mutants.(A–C) Expression of DN Rho1 and Rac1 mutants in wild-type (w1118)

Fig. 1. lgl interacts genetically with Argonaute 1 (AGO1) in the eye.

Fig. 8. Expression of other genomic-clustered Chrn subunits in the mesodiencephalon.(A) Schematic representation illustrating the assembly of the Chrnb4,

Expression of PCNA, K10, and K5 in skin lesions from Stat3+/−:HPV8 and Stat3+/+:HPV8 mice. Expression of PCNA, K10, and K5 in skin lesions from Stat3+/−:HPV8.

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Fig. 5. Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region analyzed for Th immunohistochemistry as shown in (B). Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region analyzed for Th immunohistochemistry as shown in (B). (B) Sagittal sections showing the organization of the mdDA neuronal population through the presence of Th protein. The arrow indicates a region where a severe flattening of the neuronal population is present in the Oc1−/− and Oc1/2−/− mice. (C) Coronal sections from rostral (R) to caudal (C) encompassing the mdDA neuronal population as shown by Th protein expression. The flattening of the mdDA neuronal pool as observed in the sagittal sections (B) is confirmed in these sections as indicated by arrows. Koushik Chakrabarty et al. Biology Open 2012;bio.20121230 © 2012. Published by The Company of Biologists Ltd