Fig. 1. E-cadherin localizes in nano-scale clusters.

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Sec1p requires Boi1/2p for proper localization.
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Volume 91, Issue 1, Pages 1-13 (July 2006)
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Volume 111, Issue 12, Pages (December 2016)
Fig. 5. Prip silencing enhances the co-localization of GABARAP with insulin vesicles and β-tubulin.Co-localization of GABARAP (green) with insulin (red)
Fig. 1. Overview of the nervous system of the adult S. roscoffensis.
Fig. 7. Vinculin recruitment enhances the efficiency of barrier formation.(A) TER measurements after a calcium switch in α-catenin-depleted MDCK cells.
Fig. 8. CCA and ChQ treatment induce accumulation of F-actin rings.
Fig. 1. Representative images of the four cell lines using fluorescence microscopy. Representative images of the four cell lines using fluorescence microscopy.
Fig. 4. E-cadherin expression level affects monomer dynamics.
Fig. 4. smc3 regulates the expression of cx43 in regenerating fins.
Fig. 2. Morphological changes of cultured adherent fibroblastic cells after OA treatment related to actin microfilament reorganization.(A) Cells observed.
Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.
Fig. 7. Motion adaptation increases time-dependent response modulations (TDRM) relatively to the average cell response.TDRM normalized to the value obtained.
Fig. 2. abu/pqn genes are expressed in the pharyngeal cuticle
Fig. 4. A primary screen based on scrape closure
Fig. 10. Ratiometric live imaging of di-4-ANEPPDHQ in growing pollen tubes.(A) Higher and lower membrane order distribution in control and BCD treated.
Fig. 7. Knockdown of Meis1 abolishes CR4. 2-GFP expression
Remodeling and maturation of the mouse retinal vasculature.
Fig. 1. Exogenous folic acid and Folr1 rescues the function of a Rho-kinase binding mutation in Shroom3. Exogenous folic acid and Folr1 rescues thefunction.
Fig. 6. Schematic diagram showing distribution and dynamics of four E-cadherin populations within the ROI of a FRAP experiment. Schematic diagram showing.
Fig. 4. The model of malate metabolism in fruit cells under different K level conditions. The model of malate metabolism in fruit cells under different.
Fig. 4. Acentriolar mitotic spindle assembly
PfSec13 does not co-localize with P. falciparum heterochromatin.
Fig. 1. Pigmentation and melanophore counts of rainbow trout parr and smolt caudal fins.Pigmentation of (A) parr and (B) smolt. Pigmentation and melanophore.
Fig. 1. γ-Tubulin localizes in close proximity to centriole walls in interphase but within an extended PCM meshwork in mitosis.U2OS cells were fixed and.
Fig. 2. DDR1 over-expression enhances collagen fibril reorganization
Fig. 3. Mutation of Y520 and Y667 result in increased delivery of prestin to the apical surface of MDCK cells.MDCK cells transiently transfected with wt.
Fig. 1. Ovarian cancer spheroids can bud from a monolayer
Fig. 3. Characterization of unclassified cells (UCs).
Differentiation of neural crest cells into corneal endothelial cells
Fig. 4. Detection of dFMR1 mRNA in dFMRP granules by FISH
Image cross-correlation analysis reveals the emergence of a dynamic steady state actin distribution in the minimal cortex. Image cross-correlation analysis.
Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.
Fig. 6. Comparison of Plk4 with Sas-6 localization
Fig. 2. The localisation of integrins β1 and α6 in the hES cells grown in a colony and as a single-cell culture. hES cells were harvested manually as small.
Fig. 2. Centrosomal proteins display distinct localizations and radial distances from centriole walls.U2OS cells were fixed and stained with the indicated.
Fig. 10. A mutant clone homozygous for mip120LL07629 has greatly diminished Mip120 protein levels. hsFLP/+; FRT42B, mip120LL07629/FRT42B, Ubi-GFP-nls females.
Fig. 7. Representative images of control (Cas9+GFP) and Cas9+gRNA+GFP co-injected embryos on day 4 of culture, showing nuclear-imported GFP (green) and.
Fig. 6. STK35 KO mice show ovary defects.
Fig. 2. Sorting in chimeric embryoid bodies of wildtype with E- or N-cadherin null ES cells.Wildtype, E-cadherin null (9J), and N-cadherin null (Ncad95)
Fig. 3. Weak interaction between E-cadherin and N-cadherin null cells
Fig. 3. Overexpression of wild-type GFP-CPAP, but not GFP-CPAP-377EE (GFP-377EE), induces cilia formation and promotes the growth of cilia.CAD cells (A,B)
Rgs16::GFP expression in pancreatic neoplasia.
Fig. 1. Polarized F-actin cables in the Xenopus neural plate.
EpiDEG efficiently degrades GFP-tagged proteins that localize to different subcellular localizations. epiDEG efficiently degrades GFP-tagged proteins that.
Fig. 3. Exogenous folic acid rescues neural epithelial apical constriction and activation of non-muscle myosin upon Rho-kinase inhibition. Exogenous folic.
Folic acid increases apical junctional pMLCK localization in vivo
The actin organization and N-cadherin dynamics in migrating cells.
Fig. 2. Non-homogeneous subcellular distribution of Vangl2 along the anteroposterior axis. Non-homogeneous subcellular distribution of Vangl2 along the.
dcn1-deletion results in attenuated cohesin cleavage at anaphase
IGF1R is present in endometrial luminal epithelium at the receptive phase. IGF1R is present in endometrial luminal epithelium at the receptive phase. Endometrial.
Lysine residues in the cytoplasmic region of TfR are involved in the MARCH8-induced downregulation of TfR. Lysine residues in the cytoplasmic region of.
Fig. 4. CR4.2 may be active in amacrine cells but not in ganglion cells.Chick retinas were electroporated with either the control CAG-GFP construct or.
Fig. 1. Cell adhesion molecule expression and the aggregation of wildtype and mutant ES cells.(A) Wildtype (WT), E-cadherin null (9J), and N-cadherin null.
Fig. 8. Compared mobilities of passenger proteins in G2/M-prophase, metaphase and anaphase.FRAP experiments were performed on HeLa cells stably expressing.
Fig. 5. Testis defects in STK35 KO mice.
Fig. 1. Rnd2 and Rnd3 induce stress fibres whereas Rnd1 reduces stress fibres in endothelial cells.(A) Rnd mRNAs are expressed in HUVECs. Total RNA was.
Fig. 5. EGL-20 inhibits anterior and posterior orientation of UNC-40 asymmetric localization and the formation of axons from these sites.(A–D) HSN neurons.
The BRCA1 aggregates exclude large nuclear structures.
Fig. 1. Expression of a Ror-eGFP fusion protein under control of the endogenous Ror promoter in Drosophila embryos. Expression of a Ror-eGFP fusion protein.
mip120 null egg chambers have a condensed nurse cell DNA phenotype
Fig. 5. Co-expression analyses of disease mutations in YFP-RPGRIP1α1 with wild-type RFP-RPGR1–19 or RFP-RPGRORF15 in COS7 cells.YFP-RPGRIP1α1 with disease-associated.
Fig. 3. Inclusion of E-cadherin into stationary clusters requires cis-, trans-, and cytoplasmic interactions. Inclusion of E-cadherin into stationary clusters.
Fig. 8. Enhancement of the Ptp4E Ptp10D cyst phenotype by elevation of Rho activity.(A) Rho1-CA expression in a wild-type (w1118) background produced strong.
Fig. 7. Analysis of dFMRP kinetics in dFMRP granules by FRAP
Ezrin phenotypes in the MVID intestine.
Subcellular distribution of Mst4 and aPKCι in in vivo enterocytes.
Nuclear invaginations observed in thin sections are 3D tunnels and crevices, as shown by FIB-SEM and STORM. (A) In thin section TEM, large nuclear invaginations.
Effect of myosin Vb shRNA on ezrin phosphorylation and microvilli organization in Caco-2 cells. Effect of myosin Vb shRNA on ezrin phosphorylation and.
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Fig. 1. E-cadherin localizes in nano-scale clusters. E-cadherin localizes in nano-scale clusters. (A) Ecad-GFP localization appears continuous in the junctions of pancreatic cancer cells when imaged using confocal microscopy. Bar: 10 µm. (B) Fixed cells were imaged by serial confocal sectioning before and after ROI photobleaching (top and bottom panels respectively, arrow in bottom panel highlights region of photobleaching). 3D data sets were reconstructed as projections to visualize the cell junction along the z, y, and x-axes. The x-axis projection was cropped to the photobleached region, and shows that junctions were vertical and did not undercut adjacent cells. Bar: 2 µm. (C) At higher resolution it was apparent that E-cadherin was localized in clusters. (D) Examination of cells expressing lower levels of Ecad-GFP revealed that E-cadherin clusters maintained the same average size and number of monomers per cluster, but that the spacing between clusters increased. In 3D-STORM images the z-position of each molecule is color-coded and its intensity indicates positional accuracy according to the look-up table in each panel. Color bar in lower left of panels indicates the z-position range from −375 to +375 nm (left to right) and probability per nm2 from 1.2×10−2 to 1.4×10−5 (top to bottom). Bar in C,D: 200 nm. Zahra Erami et al. Biology Open 2015;bio.014159 © 2015. Published by The Company of Biologists Ltd