Figure Neurologic, gastrointestinal, and dermatologic findings

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Nat. Rev. Neurol. doi: /nrneurol

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Figure 2. MRI features of patients with MS who had antibodies to myelin oligodendrocyte glycoprotein MRI features of patients with MS who had antibodies.

Figure 1 Initial brain imaging (A–C) patient 1; (D–F) patient 2; (G–I) patient 3; (J–L) patient 4; and (M) patient 2. Initial brain imaging (A–C) patient.

Figure 1 Phenotype and genotype of an undiagnosed family with autosomal recessive spastic ataxia Phenotype and genotype of an undiagnosed family with autosomal.

Nat. Rev. Neurol. doi: /nrneurol

Coronal (above) and axial (below) views of brain imaging findings in selected young onset dementias (images reproduced by kind permission of Dr Hadi Manji.

Reversible posterior leukoencephalopathy syndrome and silent cerebral infarcts are associated with severe acute chest syndrome in children with sickle.

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Figure 1 Brain MRI findings in the present case

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Figure 2 Association of serum IgG reactivity with MRI measures of disease severity Association of serum IgG reactivity with MRI measures of disease severity.

Figure 1 Coronal MRI images showing the evolution of white matter abnormality and atrophy of patient 1 Coronal MRI images showing the evolution of white.

Figure Facial photograph during headache attack and brain and upper cervical cord MRI Facial photograph during headache attack and brain and upper cervical.

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Figure 3 Example of venous narrowing

Figure Case presentation

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Figure Patient 1's ictal EEG and brain MRI and patient 2's ictal EEG and polygraphic recording(A) Patient 1's EEG showing an ictal discharge over right.

Figure 1 MRI head in faciobrachial dystonic seizures (A) Axial fluid-attenuated inversion recovery image from patient 3 in table 2 shows T2-weighted hyperintensity.

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Figure 1 Cerebral MRI during the disease course Cerebral MRI with multiple cerebral supratentorial lesions during the disease course: periventricular lesions.

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Figure 2 7T MRI can differentiate between early PML and MS lesions Two different patterns of brain lesions were observed using 7T MRI: ring-enhancing lesions.

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Figure 1 White matter lesion central vein visibility in MS and absence in small vessel disease (SVD)‏ White matter lesion central vein visibility in MS.

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Figure 3 Longitudinal performance of 2 MS–cohabitant participant pairs on Ishihara color testing Both response speed and response accuracy are provided.

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Figure 5 Autopsy Mycoplasma DNA analysis

Figure MRI brain 6 weeks post admission (A–C) Symmetrical high signal changes on fluid-attenuated inversion recovery sequences predominantly affecting.

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Figure 2 Spectrum of abnormal CT scanning of patients with bacterial meningitis presenting with a minimal Glasgow Coma Scale score Spectrum of abnormal.

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Figure 1 Radiologic features of patients with white matter syndromes in association with NMDA receptor antibodies Radiologic features of patients with.

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Figure 4 Patient 3 MRI evolution over time

Figure 3 Patient 2 MRI evolution over time before relapse

Figure 2 Patient 1 MRI evolution over time

Figure 1 MRI at presentation The axial diffusion-weighted image (A) showed restricted diffusion throughout the cortical ribbon of the right hemisphere.

On follow-up MR examination, 25 days after onset of symptoms, T2-weighted (A) and fluid-attenuated inversion recovery (B) MR images of brain show neuronal.

Figure 1 MRIs MRIs (A and B) Axial FLAIR images of the brain demonstrate multifocal parenchymal lesions including the right hippocampus, right midbrain,

Presentation transcript:

Figure Neurologic, gastrointestinal, and dermatologic findings Neurologic, gastrointestinal, and dermatologic findings (A) Coronal section of the brain. In the right hemisphere, there are multiple hemorrhages in white matter, hippocampus, basal gray matter, and extensive hemorrhagic cortical infarction. (B) MRI fluid-attenuated inversion recovery image shows bilateral subdural collections (arrows) with extensive subacute infarcts and hemorrhage, predominantly on the right. (C) A 4-mm porcelain white papule with central atrophy and an erythematous rim on the patient's left abdomen. (D) Papule with a dull pink gray center and red rim present on the right medial plantar foot. (E) Deposition of membrane attack complex C5b-9 in vessel walls, immunoperoxidase ×10. (F) High-power view of the skin shows increased mucin (arrow) around dermal vessels. The vessel walls have a smudged appearance and 2 are filled with fibrin thrombi. (G) White porcelain lesions (arrow) on the proximal bowel serosa seen at autopsy. Microscopically, these areas show increased mucin, abnormal vessels with degenerating, smudged vessel walls, and thrombi in medium-sized vessels. Sabrina Gmuca et al. Neurol Neuroimmunol Neuroinflamm 2016;3:e206 © 2016 American Academy of Neurology