Fig. 2. The kar phenotype arises during metamorphosis

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Volume 16, Issue 1, Pages (January 2009)

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Zebrafish Stripes as a Model for Vertebrate Colour Pattern Formation

Pigment patterns: fish in stripes and spots

CK2 is required for early cell divisions in C. elegans embryos

Fig. 6. Ultrastructural changes in the glycocalyx of N

Fig. 1. Overview of the nervous system of the adult S. roscoffensis.

Fig. 3. Hts and Dlg are in a complex at the postsynaptic membrane of larval NMJs.(A–B″) PLA with HtsM and Dlg antibodies (green) performed on third instar.

Fig. 2. Spectral sensitivity of smolt melanophores and candidate photopigments involved in melanophore photosensitive processes.(A,B) Incident light triggered.

Fig. 4. smc3 regulates the expression of cx43 in regenerating fins.

Fig. 4. Abdominal aortic aneurysm formation in zebrafish embryos following treatment with Angiotensin II or snuff extract. Abdominal aortic aneurysm formation.

Fig. 2. Morphological changes of cultured adherent fibroblastic cells after OA treatment related to actin microfilament reorganization.(A) Cells observed.

Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.

Fig. 6. Hts regulates par-1 and camkII mRNA distribution and levels in the muscle.(A–J) Views of muscles 6/7 in abdominal segment 4, probed for either.

Fig. 1. GFP expression in blood and lymphatic vessels of Tg(Flk1:eGFP) tadpoles.All panels depict lateral views of the tadpoles, head facing left. GFP.

Fig. 4. transparent encodes Mpv17.(A) tra maps to chromosome 20.

Fig. 1. Bj mutant exhibits outflow tract malalignment defects.

Dany is required for normal spermatogenesis to proceed beyond the spermatocyte stage. dany is required for normal spermatogenesis to proceed beyond the.

Altered oligodendrocyte progenitor cell (OPC) marker protein expression in two white matter regions of ShhΔFP/ΔFP and SmoΔNT/ΔNT embryos at E15.5. Altered.

Fig. 7. Characterization of 3KO embryos at E15. 5

Fig. 4. Mutations induced by TALENs are heritable through the germline

Fig. 4. Acentriolar mitotic spindle assembly

Fig. 1. Pigmentation and melanophore counts of rainbow trout parr and smolt caudal fins.Pigmentation of (A) parr and (B) smolt. Pigmentation and melanophore.

DHR3 suppresses Phm protein levels.

Fig. 1. γ-Tubulin localizes in close proximity to centriole walls in interphase but within an extended PCM meshwork in mitosis.U2OS cells were fixed and.

Fig. 3. Genetic interactions between unc-53 and unc-6 and between unc-53 and unc-5 affect intracellular UNC-40::GFP localization.(A–H) Photomicrographs.

Fgfr3 mutation is not the sole driver of tumorigenesis in the bladder.

Fig. 3. Low power hemi retinal image of the RPE surface showing the albino central and equatorial retina and pigment distribution in the periphery. Low.

Fig. 4. Non-autonomous rescue of puc expression in DME cells

Wound recruitment of melanocytes requires innate immune cells.

Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.

Leukocyte infiltration of epidermis and phagocytosis of debris in clint1 mutants. Leukocyte infiltration of epidermis and phagocytosis of debris in clint1.

Fig. 7. Lhx1-RNAi reduces the eye size

Fig. 8. Lhx1-RNAi perturbs NR formation

Transcription factor network of human pancreas development.

Fig. 6. Differential effects of Lhx2 and Lhx5 on the OV and genetic interactions between Lhx1 and Lhx5.(A) Phylogenetic tree of chicken LIM-homeodomain.

Fig. 6. Apical polarity of primitive endoderm cells on surface of embryoid bodies.Embryoid bodies were analyzed by immunofluorescence microscopy for GATA4.

Table 1. Average ± S.E. of level of dissimilarity scores of each feature per stripe per pattern comparison of sides of the same fish (“Same Individual”),

The idefix phenotype first becomes visible during metamorphosis

Fig. 3. Immunostaining for neural differentiation makers of the stage 29 optic cup.Immunoreactive signals are shown in green or red. Immunostaining for.

Fig. 8. The morphology of the ventral nerve cord in Ror-Myc overexpressing embryos is normal. The morphology of the ventral nerve cord in Ror-Myc overexpressing.

The feelgood mutation affects craniofacial skeletal development.

Reb does not play a significant role in regulating TGF-β signaling

Elevation of ERK downstream signaling is associated with tumor formation in the bladder and in papilloma formation. Elevation of ERK downstream signaling.

Fig. 2. Examples of changes in angular velocity and correlation coefficient during the O–O test. Examples of changes in angular velocity and correlation.

Mutant phenotypes and expression patterns of Drosophila segmentation genes. Mutant phenotypes andexpression patterns of Drosophila segmentation genes.

Fig. 1. Polarized F-actin cables in the Xenopus neural plate.

Fig. 3. idefix mutant chromatophores contribute to a wild-type pattern in chimeric animals. idefix mutant chromatophores contribute to a wild-type pattern.

Depleting CK2 restores centrosomal ZYG-1 levels in zyg-1(it25) embryos

Male and female Tg(fli1a:EGFP) blood vessel regeneration.

V-ATPase and proteins involved in late endosomal protein sorting are required for efficient utilization of LDs during growth resumption. V-ATPase and proteins.

Amplicon sequencing analysis of on-target sites in trβ crispants

Analysis of mutant Best1 protein (Y227N) in testis and sperm function

Fig. 12. Overview of the molecular program essential to build mdDA neurons.The genes identified in this study (in red) have been added to the programming.

Fig. 6. Gross morphological inner ear defects in Tbx1;Jag1 compound mutant embryos at E15.5. Gross morphological inner ear defects in Tbx1;Jag1 compound.

Fig. 3. transparent is required cell-autonomously in iridophores

Fig. 3. Mean force and velocity during jumping

Fig. 5. EGL-20 inhibits anterior and posterior orientation of UNC-40 asymmetric localization and the formation of axons from these sites.(A–D) HSN neurons.

Fig. 1. Expression of a Ror-eGFP fusion protein under control of the endogenous Ror promoter in Drosophila embryos. Expression of a Ror-eGFP fusion protein.

mip120 null egg chambers have a condensed nurse cell DNA phenotype

Fig. 1. Microarray analyses of genes whose expression is regulated by innervation during synaptogenesis.(A) Schematic drawings of the experimental design.

Fig. 5. Co-expression analyses of disease mutations in YFP-RPGRIP1α1 with wild-type RFP-RPGR1–19 or RFP-RPGRORF15 in COS7 cells.YFP-RPGRIP1α1 with disease-associated.

Fig. 7. Nrf2-dependent enzyme activities in wild-type, Nrf2- and Keap1-deficient tissues.Hepatic (A,C,E) and cortical (B,D,F) enzyme activities of NQO1.

Fig. 1. Lhx1 is expressed in the proximal region of the OV

Fig. 1. Photomicrograph of an epizoic acoelomorph flatworm (Waminoa sp

Fig. 5. Behaviours of the wild-types Oregon-R at two temperatures.

Fig. 7. Rho family GTPase signaling is required for cyst formation in Ptp4E Ptp10D mutants.(A–C) Expression of DN Rho1 and Rac1 mutants in wild-type (w1118)

Phenotypic analysis of the CNS in mutants for Ror, otk and otk2

Fig. 8. Expression of other genomic-clustered Chrn subunits in the mesodiencephalon.(A) Schematic representation illustrating the assembly of the Chrnb4,

Phenotype of S. pombe cells in the presence of B21P2 or LMB

Genetic interaction between e(y)1 and Notch in wing development.

Presentation transcript:

Fig. 2. The kar phenotype arises during metamorphosis Fig. 2. The kar phenotype arises during metamorphosis.Developmental series of body pigment pattern metamorphosis of wild-type (A–D), kar (E–H) and rsetLF802 mutant fish (I–L). The kar phenotype arises during metamorphosis.Developmental series of body pigment pattern metamorphosis of wild-type (A–D), kar (E–H) and rsetLF802 mutant fish (I–L). Posterior trunk regions at the level of dorsal and anal fins are shown. The series of wild type and strong rse were published by Frohnhöfer et al. (Frohnhöfer et al., 2013). At stages PB and PR, kar mutants show only slight reductions in iridophore numbers (compare panels A,B to panels E,F), they still form a continuous sheet just ventral to the horizontal myoseptum. From PR to SP however, this reduction of iridophores becomes more apparent. They do not extend properly into dorsal and ventral interstripe regions (compare panel C and panel G), occasionally, remnants or patches develop (interstripe X1V in panel H). From stage SP onwards the number of melanophores is reduced compared to wild type (compare panels C,D to panels G,H). The early phase of pigment pattern development of strong rse mutants is similar to kar, with slightly stronger reductions in iridophores (I,J). In contrast to kar, however, iridophores do not form dense ridges in interstripe regions and rather are dispersed (K,L). ao: aorta. Scale bars: 250 µm. Jana Krauss et al. Biology Open 2014;3:503-509 © 2014. Published by The Company of Biologists Ltd