Effect of bypassing the proximal gut on gut hormones involved with glycemic control and weight loss Dimitri J. Pournaras, M.R.C.S., Erlend T. Aasheim, M.D., Ph.D., Marco Bueter, M.D., Ahmed R. Ahmed, F.R.C.S., Richard Welbourn, M.D., F.R.C.S., Torsten Olbers, M.D., Ph.D., Carel W. le Roux, M.R.C.P., Ph.D. Surgery for Obesity and Related Diseases Volume 8, Issue 4, Pages 371-374 (July 2012) DOI: 10.1016/j.soard.2012.01.021 Copyright © 2012 American Society for Metabolic and Bariatric Surgery Terms and Conditions
Fig. 1 Schematic illustration of gastrointestinal glucose route after oral (black arrows) and after gastrostomy load (empty arrows). Surgery for Obesity and Related Diseases 2012 8, 371-374DOI: (10.1016/j.soard.2012.01.021) Copyright © 2012 American Society for Metabolic and Bariatric Surgery Terms and Conditions
Fig. 2 Plasma levels of (A) glucose, (B) insulin, (C) GLP-1, and (D) PYY, (E) GIP, and (F) glucagon after oral (black circles) and gastrostomy (open circles) glucose load. Data presented as mean values ± SEM. When 2-way analysis of variance revealed a significant group × time interaction, post hoc Bonferroni test was used for point to point analysis between 2 groups (*P <.05, ***P <.001). Surgery for Obesity and Related Diseases 2012 8, 371-374DOI: (10.1016/j.soard.2012.01.021) Copyright © 2012 American Society for Metabolic and Bariatric Surgery Terms and Conditions