Differential effects of a gram-negative and a gram-positive infection on autogenous and prosthetic grafts Kevin J. Geary, MD, Zygmunt M. Tomkiewicz, MD, FACP, Howard N. Harrison, PhD, William M. Fiore, MD, FACS, Joseph E. Geary, MD, FACS, Richard M. Green, MD, FACS, James A. DeWeese, MD, FACS, Kenneth Ouriel, MD Journal of Vascular Surgery Volume 11, Issue 2, Pages 339-347 (February 1990) DOI: 10.1016/0741-5214(90)90278-I Copyright © 1990 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions
Fig. 1 A, Histologic appearance of a PTFE graft in the uninfected group with sparse distribution of inflammatory cells (grade I). (Hematoxylin-eosin stain; original magnification × 100.) B, PTFE graft in S. epidermidis group with more inflammatory cells (grade II). (Hematoxylin-eosin stain; original magnification × 100.) C, PTFE graft infected with P. aeruginosa demonstrating dense infiltrate of inflammatory cells (grade III) (Hematoxylineosin stain; original magnification × 100.) Journal of Vascular Surgery 1990 11, 339-347DOI: (10.1016/0741-5214(90)90278-I) Copyright © 1990 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions
Fig. 2 Scanning electron micrograph of PTFE graft infected with S. epidermidis. Neutrophils and bacteria (arrow) are seen within the graft interstices. (Original magnification × 2000.) Journal of Vascular Surgery 1990 11, 339-347DOI: (10.1016/0741-5214(90)90278-I) Copyright © 1990 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions