Overexpression of BTG2 resulted in an increase in hepatic gluconeogenesis. Overexpression of BTG2 resulted in an increase in hepatic gluconeogenesis. A: qPCR of gluconeogenic gene expression. B: Blood glucose levels from Ad-GFP– and Ad-Btg2–infected mice for 7 days (n = 4–6). C and D: Glucose and insulin tolerance tests from mice infected with Ad-Btg2 at day 4 intravenously. *P < 0.05; **P < 0.01 vs. Ad-GFP. AUC, area under the curve. Yong Deuk Kim et al. Diabetes 2014;63:1870-1880 ©2014 by American Diabetes Association