Fig. 3. Weak interaction between E-cadherin and N-cadherin null cells

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Scanning electron microscopy analysis of EGK-I to -V chick embryos.

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Fig. 6. crb_C mutant photoreceptor cells exhibit normal morphology.

Aggregation is not required for cytoplasmic relocalization induced by misfolding mutations. Aggregation is not required for cytoplasmic relocalization.

Fig. 5. Prip silencing enhances the co-localization of GABARAP with insulin vesicles and β-tubulin.Co-localization of GABARAP (green) with insulin (red)

Fig. 1. Overview of the nervous system of the adult S. roscoffensis.

Fig. 7. Vinculin recruitment enhances the efficiency of barrier formation.(A) TER measurements after a calcium switch in α-catenin-depleted MDCK cells.

Fig. 2. Outline of the two types of stimulus sequences employed in the analysis.(A) Environment information stimuli; (B) adaptation stimuli. Outline of.

Fig. 1. Representative images of the four cell lines using fluorescence microscopy. Representative images of the four cell lines using fluorescence microscopy.

Fig. 4. Abdominal aortic aneurysm formation in zebrafish embryos following treatment with Angiotensin II or snuff extract. Abdominal aortic aneurysm formation.

Fig. 2. Morphological changes of cultured adherent fibroblastic cells after OA treatment related to actin microfilament reorganization.(A) Cells observed.

Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.

Fig. 4. Seeding density modulates cell shape in both cell types

Fig. 2. Proportion of motile objects and track length

Fig. 2. abu/pqn genes are expressed in the pharyngeal cuticle

Fig. 4. A primary screen based on scrape closure

Fig. 6. Cross-section of the stomach wall and spiral intestine of the embryo, stained with PAS. (A) Surface of the stomach wall (SW) and ingested material.

Fig. 10. Ratiometric live imaging of di-4-ANEPPDHQ in growing pollen tubes.(A) Higher and lower membrane order distribution in control and BCD treated.

Fig. 1. Exogenous folic acid and Folr1 rescues the function of a Rho-kinase binding mutation in Shroom3. Exogenous folic acid and Folr1 rescues thefunction.

Fig. 1. Mitochondrial internalization in cardiomyocytes.

Fig. 4. Increased adipocyte differentiation in Cbx7-KO ES cells

Fig. 1. Morphological and growth characterization of hBMCs and hPDCs

Fig. 2. Transfection and clonal selection of rat pluripotent stem cells to generate stable transgenic lines. Transfection and clonal selection of rat pluripotent.

Table 2. Cell surface abundance of β1 integrin measured by flow cytometry.DDR wild type and DDR over-expressing cells were treated with deoxymannojirimycin.

Fig. 1. E-cadherin localizes in nano-scale clusters.

Fig. 2. DDR1 over-expression enhances collagen fibril reorganization

Fig. 1. Ovarian cancer spheroids can bud from a monolayer

Fig. 3. Characterization of unclassified cells (UCs).

Differentiation of neural crest cells into corneal endothelial cells

Fig. 4. Detection of dFMR1 mRNA in dFMRP granules by FISH

Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.

Fig. 5. The bulk of Cep135 localizes distantly from Sas-6 and STIL

Fig. 6. Comparison of Plk4 with Sas-6 localization

Fig. 2. The localisation of integrins β1 and α6 in the hES cells grown in a colony and as a single-cell culture. hES cells were harvested manually as small.

Fig. 6. CBX7 expression in adipocyte differentiation of adipose-derived stem cells.(A) The adipose-derived stem cells, ADS1, were analyzed for the capability.

Morphological defects of hi559 GI tract.

Fig. 7. Lhx1-RNAi reduces the eye size

Fig. 7. Representative images of control (Cas9+GFP) and Cas9+gRNA+GFP co-injected embryos on day 4 of culture, showing nuclear-imported GFP (green) and.

Table 1. Relative levels of tRNAMet and tRNASer in human pancreatic adenocarcinoma cell lines.Quantification of tRNAMet and tRNASer in pancreatic adenocarcinoma.

The effect of TDP-43A315T expression on dendrite spine development.

Fig. 6. STK35 KO mice show ovary defects.

Fig. 2. Sorting in chimeric embryoid bodies of wildtype with E- or N-cadherin null ES cells.Wildtype, E-cadherin null (9J), and N-cadherin null (Ncad95)

Fig. 5. Combination of SAHA and ML produces augmented suppression of cellular proliferation with impaired cell cycle progression, enhanced apoptotic.

Readthrough GPx1 Ter–Neptune proteins colocalize with UPF1 under cytochalasin D treatment. 6CFSMEo- cells transfected with plasmids encoding YFP–UPF1 (green),

Fig. 6. Apical polarity of primitive endoderm cells on surface of embryoid bodies.Embryoid bodies were analyzed by immunofluorescence microscopy for GATA4.

Fig. 4. CIZ1 reduces the impact of injury to the heart.

Fig. 2. mip120 null mutants have abnormal egg chamber development.

Fig. 3. Overexpression of wild-type GFP-CPAP, but not GFP-CPAP-377EE (GFP-377EE), induces cilia formation and promotes the growth of cilia.CAD cells (A,B)

Fig. 1. Polarized F-actin cables in the Xenopus neural plate.

Fig. 9. Mip120 CXC and HCH domains are not sufficient for nuclear localization in mip120 null egg chambers. Mip120 CXC and HCH domains are not sufficient.

Fig. 3. idefix mutant chromatophores contribute to a wild-type pattern in chimeric animals. idefix mutant chromatophores contribute to a wild-type pattern.

Fig. 3. Exogenous folic acid rescues neural epithelial apical constriction and activation of non-muscle myosin upon Rho-kinase inhibition. Exogenous folic.

Loss or increase of Tj causes a reduction in Slbo expression.

Fig. 2. iPSCs produce functional osteoblasts.

V-ATPase and proteins involved in late endosomal protein sorting are required for efficient utilization of LDs during growth resumption. V-ATPase and proteins.

The expression of two forms of N-cadherin.

Loss of Graf results in plasmatocyte overproliferation.

Fig. 4. CR4.2 may be active in amacrine cells but not in ganglion cells.Chick retinas were electroporated with either the control CAG-GFP construct or.

Fig. 1. Cell adhesion molecule expression and the aggregation of wildtype and mutant ES cells.(A) Wildtype (WT), E-cadherin null (9J), and N-cadherin null.

Misfolding mutations impair the BRCA1 nuclear aggregation in yeast and the BRCA1 nuclear localization in human cells. Misfolding mutations impair the BRCA1.

Fig. 1. Rnd2 and Rnd3 induce stress fibres whereas Rnd1 reduces stress fibres in endothelial cells.(A) Rnd mRNAs are expressed in HUVECs. Total RNA was.

Fig. 3. MO-mediated smc3 knockdown results in reduced regenerate length, segment length and cell proliferation. MO-mediated smc3 knockdown results in reduced.

Fig. 7. Eye defects in STK35 KO mouse.

mip120 null egg chambers have a condensed nurse cell DNA phenotype

Fig. 5. Co-expression analyses of disease mutations in YFP-RPGRIP1α1 with wild-type RFP-RPGR1–19 or RFP-RPGRORF15 in COS7 cells.YFP-RPGRIP1α1 with disease-associated.

The expression pattern of marker proteins for sensory neuron subgroups changes during postnatal maturation. The expression pattern of marker proteins for.

Failure of chromosome disassembly in mip120 null ovarian nurse cells

Fig. 3. Changes in the total EPS/Chl a ratio and bend interval of trichomes before and after the removal of polysaccharide from the BG11-cultured N. flagelliforme.

The regulatory domain of HSF1 is involved in the pro-apoptotic response to TNF. (A) Upper panel, functional domains and potential DAPK phosphorylation.

Fig. 7. Analysis of dFMRP kinetics in dFMRP granules by FRAP

Exosomal transfer of sortilin to a target cell.

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Fig. 3. Weak interaction between E-cadherin and N-cadherin null cells Fig. 3. Weak interaction between E-cadherin and N-cadherin null cells.(A) Equal numbers of two different types of ES cells were allowed to aggregate at 37°C at a cell density of 2×106 cells/ml. Weak interaction between E-cadherin and N-cadherin null cells.(A) Equal numbers of two different types of ES cells were allowed to aggregate at 37°C at a cell density of 2×106 cells/ml. The rate of aggregation of the mixture was measured using a Coulter Counter, and the reduction of particle number is presented. The aggregation of wildtype (WT) ES cells at the same density is used for comparison. Particle number was determined in triplicate samples and the difference in the value was smaller than 5%. N(−/−): N-cadherin null; E(−/−): E-cadherin null. (B) N-cadherin null ES cells were first labeled with the CellVue Claret (CVC) procedure, and then were mixed with E-cadherin null ES cells to form chimeric embryoid bodies following maintaining in suspension culture for two days. The location of CVC-labeled cells in the embryoid bodies was visualized by confocal fluorescence microscopy (bright white spots). A confocal section near the equatorial region is shown. Propidium iodide (red) was added to mark dead cells. Scale bar: 20 µm. Robert Moore et al. Biology Open 2014;3:121-128 © 2014. Published by The Company of Biologists Ltd