Fig. 3. Immunostaining for neural differentiation makers of the stage 29 optic cup.Immunoreactive signals are shown in green or red. Immunostaining for.

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inhibits BMP signalling.

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Vegetal RNAs antagonise the dorsalising and BMP-inhibiting activity of Vrtn. Vegetal RNAs antagonise the dorsalising and BMP-inhibiting activity of Vrtn.

Fig. 6. crb_C mutant photoreceptor cells exhibit normal morphology.

Volume 7, Issue 9, Pages (September 1997)

Fig. 5. Prip silencing enhances the co-localization of GABARAP with insulin vesicles and β-tubulin.Co-localization of GABARAP (green) with insulin (red)

Fig. 1. Overview of the nervous system of the adult S. roscoffensis.

Fig. 8. CCA and ChQ treatment induce accumulation of F-actin rings.

Fig. 3. Hts and Dlg are in a complex at the postsynaptic membrane of larval NMJs.(A–B″) PLA with HtsM and Dlg antibodies (green) performed on third instar.

Fig. 1. Representative images of the four cell lines using fluorescence microscopy. Representative images of the four cell lines using fluorescence microscopy.

Fig. 4. E-cadherin expression level affects monomer dynamics.

Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.

Fig. 2. abu/pqn genes are expressed in the pharyngeal cuticle

Fig. 3. Knockdown of cited3 results in increased cell death but it does not affect proliferation.Embryos that are injected with the control MO (A–C), cited3.

Fig. 1. Prestin in mouse outer hair cells is localized along the lateral wall of the cell along with β-catenin and Na/K ATPase.Shown are cartoons of the.

Fig. 7. Knockdown of Meis1 abolishes CR4. 2-GFP expression

Embryonic corneal defects in E18.5 AP-2β NCC KO mutant embryos.

Fig. 1. Exogenous folic acid and Folr1 rescues the function of a Rho-kinase binding mutation in Shroom3. Exogenous folic acid and Folr1 rescues thefunction.

Fig. 4. Expression of p75NTR and time-course of TrkA autophosphorylation at the Y751 and Y490 sites in PC12-27 cells transfected with the vector, empty.

Fig. 4. The model of malate metabolism in fruit cells under different K level conditions. The model of malate metabolism in fruit cells under different.

Fig. 4. Acentriolar mitotic spindle assembly

DHR3 suppresses Phm protein levels.

Fig. 1. γ-Tubulin localizes in close proximity to centriole walls in interphase but within an extended PCM meshwork in mitosis.U2OS cells were fixed and.

Fig. 8. Knockdown of Meis1 reduces the expression of Foxn4 and Lim1+2

Fig. 3. Characterization of unclassified cells (UCs).

Differentiation of neural crest cells into corneal endothelial cells

Fig. 4. Detection of dFMR1 mRNA in dFMRP granules by FISH

Fig. 2. Ex vivo inducible knockout of PDCD2 in ESCs results in loss of S phase entry and increased p53.(A) Growth curve of inducible knockout and WT ESCs.

Wound recruitment of melanocytes requires innate immune cells.

Fig. 2. The kar phenotype arises during metamorphosis

Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.

Fig. 5. The bulk of Cep135 localizes distantly from Sas-6 and STIL

Fig. 6. Comparison of Plk4 with Sas-6 localization

Fig. 3. The checkpoint proteins Mad2 and BubR1 remain associated with the kinetochores of unaligned chromosomes in cenp-metaΔ mutant cells entering anaphase.(A–C)

Fig. 6. Localization of Sma during growth and migration

Fig. 5. GFP fluorescence colocalization of Gcn5.

Fig. 7. Lhx1-RNAi reduces the eye size

Fig. 8. Lhx1-RNAi perturbs NR formation

Fig. 5. UV reflections from the eye cup.

Fig. 4. Co-immunostaining of nocodazole or ASNase treated RPE-1 cells with anti-hASNS and anti-alpha tubulin showed defect in both mitotic spindle formation.

Fig. 6. Differential effects of Lhx2 and Lhx5 on the OV and genetic interactions between Lhx1 and Lhx5.(A) Phylogenetic tree of chicken LIM-homeodomain.

Fig. 6. Apical polarity of primitive endoderm cells on surface of embryoid bodies.Embryoid bodies were analyzed by immunofluorescence microscopy for GATA4.

Fig. 4. SMXL6 is degraded in response to SL treatment.

The idefix phenotype first becomes visible during metamorphosis

Fig. 8. The morphology of the ventral nerve cord in Ror-Myc overexpressing embryos is normal. The morphology of the ventral nerve cord in Ror-Myc overexpressing.

Statistical chart of significantly differentially expressed genes

Elevation of ERK downstream signaling is associated with tumor formation in the bladder and in papilloma formation. Elevation of ERK downstream signaling.

Fig. 6. Meis1 protein is present in CR4. 2-GFP+ and Foxn4+ cells

Fig. 2. Examples of changes in angular velocity and correlation coefficient during the O–O test. Examples of changes in angular velocity and correlation.

Fig. 3. Overexpression of wild-type GFP-CPAP, but not GFP-CPAP-377EE (GFP-377EE), induces cilia formation and promotes the growth of cilia.CAD cells (A,B)

Fig. 1. Polarized F-actin cables in the Xenopus neural plate.

Abnormally low or high Tj expression causes a decrease in DEcad expression and shg enhancer activity. Abnormally low or high Tj expression causes a decrease.

Postnatal expression of dominant-negative FGFR2b causes downregulation of FGFR signaling target genes and changes in expression of dental epithelial markers.

Fig. 3. Exogenous folic acid rescues neural epithelial apical constriction and activation of non-muscle myosin upon Rho-kinase inhibition. Exogenous folic.

Fig. 1. PARN is differentially expressed in breast cancer versus non-cancerous tissue/cells. PARN is differentially expressed in breast cancer versus non-cancerous.

Exo70 is recruited to the plasma membrane at sites of mechanical wounding. Exo70 is recruited to the plasma membrane at sites of mechanical wounding. NRK.

Fig. 2. Non-homogeneous subcellular distribution of Vangl2 along the anteroposterior axis. Non-homogeneous subcellular distribution of Vangl2 along the.

Fig. 2. iPSCs produce functional osteoblasts.

Self-organization into organoids, gastruloids and embryoids.

Fig. 12. Overview of the molecular program essential to build mdDA neurons.The genes identified in this study (in red) have been added to the programming.

Fig. 4. CR4.2 may be active in amacrine cells but not in ganglion cells.Chick retinas were electroporated with either the control CAG-GFP construct or.

Fig. 2. Acetylation stiffens primary cilia.

Fig. 1. Cell adhesion molecule expression and the aggregation of wildtype and mutant ES cells.(A) Wildtype (WT), E-cadherin null (9J), and N-cadherin null.

Fig. 1. Rnd2 and Rnd3 induce stress fibres whereas Rnd1 reduces stress fibres in endothelial cells.(A) Rnd mRNAs are expressed in HUVECs. Total RNA was.

Fig. 7. Eye defects in STK35 KO mouse.

Fig. 1. Microarray analyses of genes whose expression is regulated by innervation during synaptogenesis.(A) Schematic drawings of the experimental design.

Fig. 5. Co-expression analyses of disease mutations in YFP-RPGRIP1α1 with wild-type RFP-RPGR1–19 or RFP-RPGRORF15 in COS7 cells.YFP-RPGRIP1α1 with disease-associated.

Fig. 7. Nrf2-dependent enzyme activities in wild-type, Nrf2- and Keap1-deficient tissues.Hepatic (A,C,E) and cortical (B,D,F) enzyme activities of NQO1.

Fig. 1. Lhx1 is expressed in the proximal region of the OV

Fig. 7. Analysis of dFMRP kinetics in dFMRP granules by FRAP

Phenotype of S. pombe cells in the presence of B21P2 or LMB

Presentation transcript:

Fig. 3. Immunostaining for neural differentiation makers of the stage 29 optic cup.Immunoreactive signals are shown in green or red. Immunostaining for neural differentiation makers of the stage 29 optic cup.Immunoreactive signals are shown in green or red. Control (A,C,E,G,I,K) and Lhx1-overexpressing eyes (B,D,F,H,J,L). In these experiments, a bicistronic vector of pCAGGS-RFP-2A-Lhx1 was used. (A,B) 3A10, (C,D) N-cadherin, (E,F) HuC/D RNA-binding protein, (G,H) Islet1, (I,J) visinin (calcium-binding protein), and (K,L) AP2α. N-cadherin is localized to the neuroepithelium, visinin is localized to future cone photoreceptors, and others are localized to early retinal ganglion cells, and AP2α is localized to differentiating amacrine cells. Note that induced neural retina (iNR) is thicker than the authentic NR in this experimental condition. Scale bar: 100 µm. Takumi Kawaue et al. Biology Open 2012;1:1083-1093 © 2012. Published by The Company of Biologists Ltd