Reb does not play a significant role in regulating TGF-β signaling

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Volume 20, Issue 1, Pages (January 2010)
CK2 is required for early cell divisions in C. elegans embryos
Fig. 1. Overview of the nervous system of the adult S. roscoffensis.
Fig. 4. Clinically defined SMN mutants show alterations in the association with telomerase complex proteins. Clinically defined SMN mutants show alterations.
Fig. 1. Representative images of the four cell lines using fluorescence microscopy. Representative images of the four cell lines using fluorescence microscopy.
Arterial Notch signaling is not required for artery patterning.
Fig. 4. Expression of oxt is undetectable in the med12 mutant but is unaffected by treatment with Wnt inhibitors. Expression of oxt is undetectable in.
Fig. 2. Morphological changes of cultured adherent fibroblastic cells after OA treatment related to actin microfilament reorganization.(A) Cells observed.
Fig. 6. Cross-section of the stomach wall and spiral intestine of the embryo, stained with PAS. (A) Surface of the stomach wall (SW) and ingested material.
Fig. 2. p85β regulates cell adhesion
Fig. 7. Knockdown of Meis1 abolishes CR4. 2-GFP expression
Tbx1 regulates the Mef2c AHF enhancer in vivo.
Fig. 1. Exogenous folic acid and Folr1 rescues the function of a Rho-kinase binding mutation in Shroom3. Exogenous folic acid and Folr1 rescues thefunction.
Fig. 1. Mitochondrial internalization in cardiomyocytes.
Fig. 3. Inactivation of the Wnt/β-catenin signaling pathway inhibited cell proliferation and induced apoptosis in A549 and SPC-A-1 cells. Inactivation.
Fig. 1. Loss of PC following INT depletion
Fig. 4. Mutations induced by TALENs are heritable through the germline
Fig. 1. Pigmentation and melanophore counts of rainbow trout parr and smolt caudal fins.Pigmentation of (A) parr and (B) smolt. Pigmentation and melanophore.
DHR3 suppresses Phm protein levels.
eb1a-2 eb1b-3 double-mutant plants display skewed and shorter roots
Fig. 1. γ-Tubulin localizes in close proximity to centriole walls in interphase but within an extended PCM meshwork in mitosis.U2OS cells were fixed and.
Fig. 2. DDR1 over-expression enhances collagen fibril reorganization
Fig. 3. Genetic interactions between unc-53 and unc-6 and between unc-53 and unc-5 affect intracellular UNC-40::GFP localization.(A–H) Photomicrographs.
Fig. 8. Knockdown of Meis1 reduces the expression of Foxn4 and Lim1+2
Fig. 6. LR phenotype of plastid translation-defective mutants with/without Spec. LR phenotype of plastid translation-defective mutants with/without Spec.
Overexpression of Nedd4 is able to rescue miR-1-mediated actin fiber disarray in larval hearts. Overexpression of Nedd4 is able to rescue miR-1-mediated.
Fig. 4. Non-autonomous rescue of puc expression in DME cells
Table 3. Penetrance of cuticle defects seen in ush2 mutants embryos and in the progeny from rescue experiments (genotypes indicated in the table) with.
Fig. 3. The checkpoint proteins Mad2 and BubR1 remain associated with the kinetochores of unaligned chromosomes in cenp-metaΔ mutant cells entering anaphase.(A–C)
Fig. 7. Lhx1-RNAi reduces the eye size
Fig. 3. Rnd proteins induce stronger responses in subconfluent endothelial cells.HUVECs were transfected with Rnd1, Rnd2, Rnd3 or GFP-encoding plasmids.
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Fig. 8. The morphology of the ventral nerve cord in Ror-Myc overexpressing embryos is normal. The morphology of the ventral nerve cord in Ror-Myc overexpressing.
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Abnormally low or high Tj expression causes a decrease in DEcad expression and shg enhancer activity. Abnormally low or high Tj expression causes a decrease.
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Loss or increase of Tj causes a reduction in Slbo expression.
Expression analysis of periostin in DRG
Fig. 1. Loss of PC following INT depletion
Attenuated STM expression in drn and drnl mutants.
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Fig. 2. Expression of Cx43 mutant T154A resulted in non-radial spreading and formation of protrusions in J558µm3 cells spreading in response to BCR signaling.(A)
Fig. 6. RhoB is required for Rnd3-induced stress fibre formation
Fig. 3. MO-mediated smc3 knockdown results in reduced regenerate length, segment length and cell proliferation. MO-mediated smc3 knockdown results in reduced.
Fig. 5. EGL-20 inhibits anterior and posterior orientation of UNC-40 asymmetric localization and the formation of axons from these sites.(A–D) HSN neurons.
mip120 null egg chambers have a condensed nurse cell DNA phenotype
Fig. 6. Bj mutants show stereocilia patterning defects and biliary duct (BD) malformations. Bj mutants show stereocilia patterning defects and biliary.
Integrin-mediated signaling is required for PFC maturation.
Fig. 5. Co-expression analyses of disease mutations in YFP-RPGRIP1α1 with wild-type RFP-RPGR1–19 or RFP-RPGRORF15 in COS7 cells.YFP-RPGRIP1α1 with disease-associated.
Integrins modulate the Notch pathway by regulating its intracellular trafficking and/or processing. Integrins modulate the Notch pathway by regulating.
Volume 22, Issue 19, Pages (October 2012)
Fig. 5. Behaviours of the wild-types Oregon-R at two temperatures.
Fig. 7. Rho family GTPase signaling is required for cyst formation in Ptp4E Ptp10D mutants.(A–C) Expression of DN Rho1 and Rac1 mutants in wild-type (w1118)
Phenotypic analysis of the CNS in mutants for Ror, otk and otk2
Fig. 1. lgl interacts genetically with Argonaute 1 (AGO1) in the eye.
Fig. 8. Enhancement of the Ptp4E Ptp10D cyst phenotype by elevation of Rho activity.(A) Rho1-CA expression in a wild-type (w1118) background produced strong.
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Fig. 4. Reb does not play a significant role in regulating TGF-β signaling. Reb does not play a significant role in regulating TGF-β signaling. Stage 15 embryos were stained with anti-Uif to label the apical membrane. (A) DT and DB form normally in wild-type trachea. (B) DB did not migrate out in zygotic punt135 mutants. (C) Similar to punt135 mutants, HA-Reb overexpression in trachea did not rescue the DB migration defects. (D) Similar to punt135 mutants, HA overexpression in trachea did not rescue the DB migration defects. (E) Mad overexpression in punt135 mutant trachea significantly rescued DB migration defect. (F) TkvQD overexpression in trachea led to DT formation defect. (G) Coexpression of HA-Reb and TkvQD in trachea had similar DT formation defect as TkvQD expression alone. (H) Coexpression of HA and TkvQD in trachea had similar DT formation defect as TkvQD expression alone. (I) Coexpression of Dad and TkvQD in trachea significantly rescued DT formation defect. Arrowheads, dorsal branch (DB); arrows, DT. Scale bar: 10 μm. Ten embryos of each genotype were imaged. Rachana R. Chandran et al. Biology Open 2018;7:bio036848 © 2018. Published by The Company of Biologists Ltd