Gene expression in islets isolated from diabetic mice.

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Chun-Ching Shih, Cheng-Hsiu Lin, Wei-Li Lin

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Supplemental Figure 2. (A) AtplaIVA-1 and AtplaIVA-2 null transcription lines for AtPLAIVA mRNA. RNAs from the relevant wild type Col were isolated.

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Volume 23, Issue 5, Pages (May 2015)

Microarray analysis of phospho-Twist1–responsive genes.

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Quantitative RT-PCR results of relative gene expression normalized to wild-type (WT) expression levels for the LjMYB14 transgene (A) and genes specific.

Inhibition of β-catenin by the Smad4/BMP pathway.

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Diabetic FXR KO kidney showed increased lipid accumulation.

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Overexpression of BTG2 resulted in an increase in hepatic gluconeogenesis. Overexpression of BTG2 resulted in an increase in hepatic gluconeogenesis. A:

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β-Cell–specific deletion of Phb2 renders mice diabetic.

RT-PCR quantification of ubiquitination gene transcripts in rat islets exposed to 2.8 or 16.7 mmol/l glucose for 2 h (A) or 24 h (B). RT-PCR quantification.

Increased pancreatic insulin content and islet size and protection against HFD- and palmitate-induced cell death in PKCδKN-overexpressing mice. Increased.

ADCY5 is expressed in isolated human islets and affected by T2D risk alleles. ADCY5 is expressed in isolated human islets and affected by T2D risk alleles.

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Enhanced expression of Cap43 gene by nickel in breast cancer cell lines. Enhanced expression of Cap43 gene by nickel in breast cancer cell lines. Expression.

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Gene expression in islets isolated from diabetic mice. Gene expression in islets isolated from diabetic mice. Real-time RT-PCR analysis of mRNA expression of insulin I (A), insulin II (B), Pdx-1 (C), LC3 (D), DNAJC3 (E), and PCNA (F). Results were standardized to β-actin mRNA levels and then expressed as fold changes vs. the lean-PBS mice. Data are mean ± SEM. n = 3 animals/group. *P < 0.05, **P < 0.01, ***P < 0.001 vs. the lean-PBS group; +P < 0.05, ++P < 0.01, +++P < 0.001 vs. the db/db-PBS group. Yi Lin, and Zhongjie Sun Diabetes 2015;64:1444-1458 ©2015 by American Diabetes Association