Figure 2 Seizure outcomes

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Figure Pedigrees of the SCA42 families identified in this study

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Figure Flow of funds for a 340B agreement for fingolimod

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Figure 3 Pedigree of familial idiopathic transverse myelitis

Figure 1 Box plot of the venous diameter in lesions

Figure 2 Needle biopsy of the left vastus lateralis

Figure 2 Spinal cord lesions

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Figure Sural nerve electron microscopy

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Figure Pedigree of the family

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Figure Family tree with the HLA haplotyping of 6 members of the family

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Figure 6 Cellular composition after tissue dissociation

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Figure 2 Changes in fatigue under treatment

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Figure 1 Stacked bar chart depicts the proportion of patients with diffusion-weighted imaging (DWI)+ and DWI− scans categorized by index event type TIA.

Figure 1 Annualized percentage brain volume change

Figure 2 BVL according to on-study disability worsening

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Figure 2 Frequency of the proportion of total WMLs with central veins in PPMS, RRMS, and SVD Frequency of the proportion of total WMLs with central veins.

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Figure 2 Pedigrees of families and segregation analysis of variants c

Figure 1 Analysis of inflammatory cells in muscle biopsies

Figure 3 Freedom from clinical disease activity during 36 months of fingolimod treatment Freedom from clinical disease activity during 36 months of fingolimod.

Figure 3 C5B3 blocked MAC formation

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Figure 3 Within-group comparisons (before–after)‏

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Figure 2 Time from incident ADS event to MS diagnosis

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Figure 2 Seizure outcomes Seizure outcomes (A) Response rate over time from the start of everolimus. Bars represent 95% confidence intervals obtained using Clopper-Pearson method. (B) Median percentage reduction in seizure frequency over time from the start of everolimus. Bars represent 95% confidence intervals based on bootstrap percentiles. David N. Franz et al. Neurol Clin Pract 2018;8:412-420 Copyright © 2018 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of the American Academy of Neurology.