Venous obstruction: An analysis of one hundred thirty-seven cases with hemodynamic, venographic, and clinical correlations  Seshadri Raju, MD, Ruth Fredericks,

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Presentation transcript:

Venous obstruction: An analysis of one hundred thirty-seven cases with hemodynamic, venographic, and clinical correlations  Seshadri Raju, MD, Ruth Fredericks, MD  Journal of Vascular Surgery  Volume 14, Issue 3, Pages 305-313 (September 1991) DOI: 10.1016/0741-5214(91)90081-5 Copyright © 1991 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions

Fig. 1 Anatomic distribution of venous obstruction. Venograms for analysis were available in 127 of 137 limbs studied. Journal of Vascular Surgery 1991 14, 305-313DOI: (10.1016/0741-5214(91)90081-5) Copyright © 1991 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions

Fig. 2 Distribution of venous obstruction according to the number of venous segments involved; 127 venograms were available for analysis. Journal of Vascular Surgery 1991 14, 305-313DOI: (10.1016/0741-5214(91)90081-5) Copyright © 1991 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions

Fig. 3 A and B. Photoplethysmography tracing of digital arterial pulsations in a limb with grade I obstruction and another with grade IV obstruction, respectively. Classic reactive hyperemia response is evident in the former after release of ischemic cuff occlusion. This is absent in the latter. Journal of Vascular Surgery 1991 14, 305-313DOI: (10.1016/0741-5214(91)90081-5) Copyright © 1991 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions

Fig. 4 Postexercise pressure in venous obstruction was elevated in all groups. No difference between the four grades of obstruction was present except for grade III with small numbers (see text). Journal of Vascular Surgery 1991 14, 305-313DOI: (10.1016/0741-5214(91)90081-5) Copyright © 1991 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions

Fig. 5 Valsalva foot venous pressure elevation in venous obstruction with and without stasis ulceration. For the entire group ulcerated limbs had a mean Valsalva pressure elevation of 7.6 ± 4.4 mm Hg and nonulcerated limbs 3.4 mm Hg ± 2.0, p < 0.001. Journal of Vascular Surgery 1991 14, 305-313DOI: (10.1016/0741-5214(91)90081-5) Copyright © 1991 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions

Fig. 6, A and B. A large collateral vessel connecting the profunda femoris and popliteal veins in two different cases. This is frequently seen after femoral vein obstructions (A). Occasionally such a connection is present as a congenital anomaly even when the femoral vein is open and unobstructed (B). The large bore connection to the popliteal vein may suggest a putative embryologic connection (see text). Journal of Vascular Surgery 1991 14, 305-313DOI: (10.1016/0741-5214(91)90081-5) Copyright © 1991 Society for Vascular Surgery and International Society for Cardiovascular Surgery, North American Chapter Terms and Conditions