Fig. 4. CR4.2 may be active in amacrine cells but not in ganglion cells.Chick retinas were electroporated with either the control CAG-GFP construct or.

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Pias3-Dependent SUMOylation Directs Rod Photoreceptor Development
Volume 31, Issue 6, Pages (September 2001)
Fig. 3 hESC-RPE cell sheet transplantation and validation of the surgical method. hESC-RPE cell sheet transplantation and validation of the surgical method.
Volume 15, Issue 6, Pages (March 2005)
Msx1-Positive Progenitors in the Retinal Ciliary Margin Give Rise to Both Neural and Non-neural Progenies in Mammals  Marie-Claude Bélanger, Benoit Robert,
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Jin Wan, Rajesh Ramachandran, Daniel Goldman  Developmental Cell 
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Identification of one type of RGCs with large somas.
Fig. 5. Prip silencing enhances the co-localization of GABARAP with insulin vesicles and β-tubulin.Co-localization of GABARAP (green) with insulin (red)
Fig. 8. CCA and ChQ treatment induce accumulation of F-actin rings.
Fig. 3. Hts and Dlg are in a complex at the postsynaptic membrane of larval NMJs.(A–B″) PLA with HtsM and Dlg antibodies (green) performed on third instar.
Fig. 1. Representative images of the four cell lines using fluorescence microscopy. Representative images of the four cell lines using fluorescence microscopy.
Targeting of endothelial Rbpj during vessel remodeling.
Nephrogenesis in vitro can be rescued in 3D co-cultures by siRNA treatment of Renca cells. Nephrogenesis in vitro can be rescued in 3D co-cultures by siRNA.
Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.
A highly conserved six-amino-acid region in the C-terminal CT domain of MARCH8 is responsible for its ability to downregulate TfR. A highly conserved six-amino-acid.
Fig. 2. abu/pqn genes are expressed in the pharyngeal cuticle
Fig. 10. Ratiometric live imaging of di-4-ANEPPDHQ in growing pollen tubes.(A) Higher and lower membrane order distribution in control and BCD treated.
Fig. 7. Knockdown of Meis1 abolishes CR4. 2-GFP expression
Embryonic corneal defects in E18.5 AP-2β NCC KO mutant embryos.
Fig. 5. Meis1 transcription factor is essential for CR4
Fig. 1. Exogenous folic acid and Folr1 rescues the function of a Rho-kinase binding mutation in Shroom3. Exogenous folic acid and Folr1 rescues thefunction.
Fig. 1. Mitochondrial internalization in cardiomyocytes.
Fig. 1. Morphological and growth characterization of hBMCs and hPDCs
Ectopic sprouting and proliferation in Rbpj veins.
Fig. 1. E-cadherin localizes in nano-scale clusters.
Fig. 1. γ-Tubulin localizes in close proximity to centriole walls in interphase but within an extended PCM meshwork in mitosis.U2OS cells were fixed and.
Fig. 2. DDR1 over-expression enhances collagen fibril reorganization
Fig. 8. Knockdown of Meis1 reduces the expression of Foxn4 and Lim1+2
Differentiation of neural crest cells into corneal endothelial cells
Fig. 6. LR phenotype of plastid translation-defective mutants with/without Spec. LR phenotype of plastid translation-defective mutants with/without Spec.
Fig. 4. Detection of dFMR1 mRNA in dFMRP granules by FISH
Fig. 5. Differentiated cells sort to the outer layer, regardless of E- or N-cadherin status.Wildtype and mutant ES cells were distinguished by GFP expression.
Fig. 6. Comparison of Plk4 with Sas-6 localization
Fig. 3. Cell-autonomous loss of GABAergic interneurons in the OB of newborn and adult conditional GFRα1 mutants. Cell-autonomous loss of GABAergic interneurons.
Fig. 7. Lhx1-RNAi reduces the eye size
Fig. 2. Centrosomal proteins display distinct localizations and radial distances from centriole walls.U2OS cells were fixed and stained with the indicated.
Fig. 3. Rnd proteins induce stronger responses in subconfluent endothelial cells.HUVECs were transfected with Rnd1, Rnd2, Rnd3 or GFP-encoding plasmids.
Fig. 7. Representative images of control (Cas9+GFP) and Cas9+gRNA+GFP co-injected embryos on day 4 of culture, showing nuclear-imported GFP (green) and.
Fig. 4. Co-immunostaining of nocodazole or ASNase treated RPE-1 cells with anti-hASNS and anti-alpha tubulin showed defect in both mitotic spindle formation.
Simultaneous perturbations to shuttling result in defects in the Dl gradient and in the sna domain. Simultaneous perturbations to shuttling result in defects.
Fig. 1. Prediction of Foxn4 cis-regulatory elements and experimental design for functional verification.(A) Comparative sequence analysis between mouse.
Fig. 2. Histological resin section of the seal retina and TL
Fig. 3. Immunostaining for neural differentiation makers of the stage 29 optic cup.Immunoreactive signals are shown in green or red. Immunostaining for.
Fig. 6. Meis1 protein is present in CR4. 2-GFP+ and Foxn4+ cells
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Fig. 2. Immunostaining for ClC-5 in isolated hog gastric mucosa
Expression analysis of periostin in DRG
Sox2 regulates dental epithelial stem cell proliferation and differentiation. Sox2 regulates dental epithelial stem cell proliferation and differentiation.
Fig. 2. Non-homogeneous subcellular distribution of Vangl2 along the anteroposterior axis. Non-homogeneous subcellular distribution of Vangl2 along the.
Histological analyses for limb-specific Uhrf1 KO mice.
Fig. 2. Acetylation stiffens primary cilia.
Fig. 7. Eye defects in STK35 KO mouse.
Fig. 6. Bj mutants show stereocilia patterning defects and biliary duct (BD) malformations. Bj mutants show stereocilia patterning defects and biliary.
Integrins are required to strengthen Notch signaling.
Adult Acvr1;Trp53DCKO lenses form vascularized, tumor-like structures at their posterior poles. 4-month-old Trp53CKO and Acvr1;Trp53DCKO lenses were sectioned.
WT MARCH8, but not the CS mutant, specifically ubiquitinates and downregulates TfR. WT MARCH8, but not the CS mutant, specifically ubiquitinates and downregulates.
Fig. 7. Analysis of dFMRP kinetics in dFMRP granules by FRAP
Disruption of the tubule formation by immortalized cells during nephrogenesis in vitro. Disruption of the tubule formation by immortalized cells during.
Effects of Smo and Math5 double mutations on RGC and cone cell production. Effects of Smo and Math5 double mutations on RGC and cone cell production. A–P,
Enhanced retinal ganglion cell production in Smo mutant retinas.
Expression of Chx10-cre and phenotypes of Chx10-cre induced Smo ablation. Expression of Chx10-cre and phenotypes of Chx10-cre induced Smo ablation. A–G,
Specificity of the WDR81 antibody for the antigen.
Quantification of TUNEL staining and OS length among the rab11a constructs. Quantification of TUNEL staining and OS length among the rab11a constructs.
Fig. 1. DACs immunolabeled with TH antibodies in the rabbit retina
Fig. 8. Electron micrographs taken from 8-week glaucoma retina
Fig. 7. Apoptotic cell death was evaluated via TUNEL
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Fig. 4. CR4.2 may be active in amacrine cells but not in ganglion cells.Chick retinas were electroporated with either the control CAG-GFP construct or CR4.2-βGP-GFP (CR4.2-GFP) construct at embryonic day 4 (E4). CR4.2 may be active in amacrine cells but not in ganglion cells.Chick retinas were electroporated with either the control CAG-GFP construct or CR4.2-βGP-GFP (CR4.2-GFP) construct at embryonic day 4 (E4). Transfected retinas were harvested at E6, E7, and E8, sectioned, and immunostained for GFP (green), Brn3a (red, panels A–F), and NeuN (red, panels G–L). (A–F) Double labeled cells (boxed regions) are shown in higher magnification on the right (indicated by arrowheads; arrows point to Brn3a-negative cells). (M,N) Quantification of double labeled cells (GFP+/Brn3a+ or GFP+/NeuN+). Error bars represent standard error of the mean. Data represent the mean ± s.d.; n≥3. ONBL, outer neuroblastic layer; INBL, inner neuroblastic layer; ONL, outer nuclear layer; INL, inner nuclear layer; GCL, ganglion cell layer. Scale bar: 20 µm. Mohammed M. Islam et al. Biology Open 2013;bio.20132279 © 2013. Published by The Company of Biologists Ltd