Fig. 7. Rho family GTPase signaling is required for cyst formation in Ptp4E Ptp10D mutants.(A–C) Expression of DN Rho1 and Rac1 mutants in wild-type (w1118)

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Scanning electron microscopy analysis of EGK-I to -V chick embryos.

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CK2 is required for early cell divisions in C. elegans embryos

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Fig. 8. In vitro effect of CMPs on adult DSS-treated stomach explants.

Fig. 3. Hts and Dlg are in a complex at the postsynaptic membrane of larval NMJs.(A–B″) PLA with HtsM and Dlg antibodies (green) performed on third instar.

Fig. 1. Representative images of the four cell lines using fluorescence microscopy. Representative images of the four cell lines using fluorescence microscopy.

Fig. 4. Expression of oxt is undetectable in the med12 mutant but is unaffected by treatment with Wnt inhibitors. Expression of oxt is undetectable in.

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Fig. 4. A primary screen based on scrape closure

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Embryonic corneal defects in E18.5 AP-2β NCC KO mutant embryos.

Fig. 1. Mitochondrial internalization in cardiomyocytes.

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Fig. 8. p85β regulates active Cdc42 and Rac localization to deep adhesions.(A) NIH3T3 cell lines expressing p85α or p85β were incubated without serum (2 h),

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eb1a-2 eb1b-3 double-mutant plants display skewed and shorter roots

Fig. 1. γ-Tubulin localizes in close proximity to centriole walls in interphase but within an extended PCM meshwork in mitosis.U2OS cells were fixed and.

Fig. 8. Knockdown of Meis1 reduces the expression of Foxn4 and Lim1+2

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Fig. 1. Polarized F-actin cables in the Xenopus neural plate.

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Fig. 5. Co-expression analyses of disease mutations in YFP-RPGRIP1α1 with wild-type RFP-RPGR1–19 or RFP-RPGRORF15 in COS7 cells.YFP-RPGRIP1α1 with disease-associated.

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Fig. 1. lgl interacts genetically with Argonaute 1 (AGO1) in the eye.

Fig. 8. Enhancement of the Ptp4E Ptp10D cyst phenotype by elevation of Rho activity.(A) Rho1-CA expression in a wild-type (w1118) background produced strong.

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Fig. 7. Rho family GTPase signaling is required for cyst formation in Ptp4E Ptp10D mutants.(A–C) Expression of DN Rho1 and Rac1 mutants in wild-type (w1118) embryos produced defects in tracheal branching, including failures of GB extension (B,C, arrows). Rho family GTPase signaling is required for cyst formation in Ptp4E Ptp10D mutants.(A–C) Expression of DN Rho1 and Rac1 mutants in wild-type (w1118) embryos produced defects in tracheal branching, including failures of GB extension (B,C, arrows). In the Ptp4E Ptp10D background (D), expression of Rho1-DN (E) or Rac1-DN (not shown) produced complete suppression of the TC/LT diameter phenotype, and partial suppression of the GB cyst number phenotype. Cdc42-DN (F) and Rho1 dsRNA (not shown) produced partial suppression of the TC/LT phenotype, but did not affect GB cyst number. In all panels, arrowheads indicate the positions of TC/LT junctions. (G,H) Quantitative analysis of TC/LT (G) and GB cyst number (H) phenotypes. Scale bar: 10 µm. Mili Jeon et al. Biology Open 2012;bio.2012471 © 2012. Published by The Company of Biologists Ltd