Fig. 8. Expression of other genomic-clustered Chrn subunits in the mesodiencephalon.(A) Schematic representation illustrating the assembly of the Chrnb4,

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Wnt-Drl/Ryk mediates axon repulsion in the Drosophila nerve cord and vertebrate cortico-spinal tract. Wnt-Drl/Ryk mediates axon repulsion in the Drosophila.
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Scanning electron microscopy analysis of EGK-I to -V chick embryos.
Fig. 2. Outline of the two types of stimulus sequences employed in the analysis.(A) Environment information stimuli; (B) adaptation stimuli. Outline of.
Fig. 8. Esco2-dependent cis-DNA looping model underlying the etiology of RBS. (A) Schematic representation of the cohesin ring complex. Esco2-dependent.
Fig. 4. smc3 regulates the expression of cx43 in regenerating fins.
Fig. 2. Proportion of motile objects and track length
Fig. 4. transparent encodes Mpv17.(A) tra maps to chromosome 20.
Fig. 2. abu/pqn genes are expressed in the pharyngeal cuticle
Altered oligodendrocyte progenitor cell (OPC) marker protein expression in two white matter regions of ShhΔFP/ΔFP and SmoΔNT/ΔNT embryos at E15.5. Altered.
Fig. 6. The cholinergic receptor subunits a6 (Chrna6) and b3 (Chrnb3) are (subset) specifically expressed in mdDA neurons during development.(A) The Chrna6.
Fig. 2. Body weight and size analysis of A1/A2-KO mice
Fig. 5. Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region.
Fig. 1. Lack of Hmga1 and Hmga2 expression in A1/A2-KO mice
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Fig. 4. The model of malate metabolism in fruit cells under different K level conditions. The model of malate metabolism in fruit cells under different.
Fig. 1. γ-Tubulin localizes in close proximity to centriole walls in interphase but within an extended PCM meshwork in mitosis.U2OS cells were fixed and.
Fig. 2. Validating the efficiency of smc3 MOs
Fig. 3. Genetic interactions between unc-53 and unc-6 and between unc-53 and unc-5 affect intracellular UNC-40::GFP localization.(A–H) Photomicrographs.
Fig. 4. Targeted disruption of STK35 transcripts in mouse.
Table 3. Penetrance of cuticle defects seen in ush2 mutants embryos and in the progeny from rescue experiments (genotypes indicated in the table) with.
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Fig. 10. Expression of the cholinergic receptor subunits Chrna6 and Chrnb3 depends on Pitx3.In situ hybridization for Th, Chrna6 and Chrnb3 on sagittal.
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Fig. 6. The cholinergic receptor subunits a6 (Chrna6) and b3 (Chrnb3) are (subset) specifically expressed in mdDA neurons during development.(A) The Chrna6.
Fig. 4. Spectra of monochromatic light from the OLS
Fig. 9. Loss of the cholinergic receptor subunit Chrna6 does not alter the development and organization of the mdDA system.(A) In situ hybridization experiments.
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Fig. 12. Overview of the molecular program essential to build mdDA neurons.The genes identified in this study (in red) have been added to the programming.
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Fig. 7. Eye defects in STK35 KO mouse.
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Fig. 5. EGL-20 inhibits anterior and posterior orientation of UNC-40 asymmetric localization and the formation of axons from these sites.(A–D) HSN neurons.
Fig. 1. Expression of a Ror-eGFP fusion protein under control of the endogenous Ror promoter in Drosophila embryos. Expression of a Ror-eGFP fusion protein.
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Fig. 8. Expression of other genomic-clustered Chrn subunits in the mesodiencephalon.(A) Schematic representation illustrating the assembly of the Chrnb4, Chrna3 and Chrna5 genes on mouse chromosome 9. Expression of other genomic-clustered Chrn subunits in the mesodiencephalon.(A) Schematic representation illustrating the assembly of the Chrnb4, Chrna3 and Chrna5 genes on mouse chromosome 9. (B) Schematic representation of the sagittal plane of the in situ hybridization experiments as shown in (C). The position of mdDA neurons is indicated in red. (C) In addition to Chrna6 and Chrnb3 (Fig. 6) the cholinergic receptor subunits Chrna4 and Chrnb2 are expressed within the mdDA region. A, anterior; D, dorsal; P, posterior; V, ventral. Koushik Chakrabarty et al. Biology Open 2012;1:693-704 © 2012. Published by The Company of Biologists Ltd