Fig. 1. MS and MSEW mice did not exhibit depression-related behavior compared to controls. There was no significant difference in the time spent stationary.

Slides:



Advertisements
Similar presentations
Fig. 4-1, p Fig. 4-2, p. 109 Fig. 4-3, p. 110.
Advertisements

P.464. Table 13-1, p.465 Fig. 13-1, p.466 Fig. 13-2, p.467.
Fig. 11-1, p p. 360 Fig. 11-2, p. 361 Fig. 11-3, p. 361.
Table 6-1, p Fig. 6-1, p. 162 p. 163 Fig. 6-2, p. 164.
Figure 1.1 The observer in the truck sees the ball move in a vertical path when thrown upward. (b) The Earth observer views the path of the ball as a parabola.
M May significant figures significant figures to nearest 1000to nearest tenthto nearest hundred thousandth.
Behavioral Effects of SQSTM1/p62 Overexpression in Mice: Support for a Mitochondrial Role in Depression and Anxiety Michael Lamar Seibenhener, Ph.D. Department.
Normal Distribution.
Supplementary Table 1 The tables show the measures of anxiety-like behavior by home-cage emergence (1a) and open-field (1b) tests, food motivation by food.
Fig. 2. Neuroprotective effects of ex-4 through the G protein-coupled GLP-1 receptor. (A) Infarct volume was assessed by TTC staining after tMCAO in rats.
Fig. 4. TGF-α is one of the major cytokines in M2 conditioned media
Fig. 1. In situ hybridization analysis for detecting endogenous mBest1 transcript in whole-brain region. In situ hybridization results from coronal (top.
Fig. 5. The measurement of phosphorylation of p53 level in N2A cells under high glucose condition and agmatine treatment. (A, B) p53 phosphorylation levels.
Fig. 1. Infarct size and behavioral outcomes in response to single and repeated treatment of hUCB-MSCs after MCAO. (A) Schematic of the time schedule.
Fig. 9. Treatment of flagellin increased cell survival in hypoxic conditions. (A) After 2 h OGD, cell cytotoxicity levels were measured by a LDH assay.
Fig. 2. Severities of seizures by durations and daily frequency at different time points. (A) An example of seizure activity in EEG recording. (B) Seizure.
Fig. 6. Akt inhibitor IV reduced the activity of the NF-κB promoter in postconditioning. NF-κB activity was detected by a reporter gene assay in bEnd.3.
Fig. 2. ERK phosphorylation is elevated in dystrophic muscle
كيــف تكتـب خطـة بحـث سيئـة ؟؟
الدكتـور/ عبدالناصـر محمـد عبدالحميـد
قوانين برگزاري مناقصات و آيين نامه مالي و معاملاتي دانشگاه علوم پزشكي و خدمات بهداشتي ،درماني تهران
Behavioral Disturbances in Estrogen-Related Receptor alpha-Null Mice
Experimental timeline.
Mechanical and cold hypersensitivity in nerve-injured C57BL/6J mice is not associated with fear-avoidance- and depression-related behaviour  F.S. Hasnie,
EXP file structure.
Volume 3, Issue 1, Pages (July 2014)
NKTfh cell–driven class switch against polysaccharide Ag is IL-21-influenced. NKTfh cell–driven class switch against polysaccharide Ag is IL-21-influenced.
Fig. 3. Mice which experienced MS or MSEW did not exhibit impairments in hippocampal and amygdala-dependent fear-related emotional memories. In the acquisition.
Fig. 1. Ca2+-dependent spike frequency adaptation and adaptation index
Fig. 5. Anti-inflammatory effects of silibinin on KA treatment in the hippocampus. (A) Representative coronal sections of the hippocampal CA1 region following.
Effect of circadian forced desynchrony on behavioral manifestations of depression. Effect of circadian forced desynchrony on behavioral manifestations.
Fig. 3. OPNpt20 binding to αv integrin in BV2 cells
Fig. 2. High density EEG of anterior and posterior spindles
Fig. 2. Induction of F-actin polymerization by OPNpt20
Fig. 3. Relationship between the generalization of conscious feeling induced by fear conditioning and individual anxiety (or depression) level. To determine.
Fig. 4. Activations of FAK, Erk, and Akt pathways by OPNpt20
Fig. 1. siRNA-mediated suppression of Shank3, Nlgn3, Fmr1, Mecp2 or Tsc1 in the dorsal striatum produced mild to severe autistic-like phenotypes. (A) Experimental.
Fig. 3. Comparing between source localization algorithms
Fig. 2. Autistic-like phenotypes induced by the striatal inhibition of MeCP2 or TSC1 were rescued by rebalancing glutamate or dopaminergic activity. (A)
Fig. 2. Infarct size and mNSS, weight change of animals with oral gavage of HSA and saline. (A) TTC staining of the individual brain was shown that the.
Fig. 3. Inhibition of HIF1α with chrysin induced cell death and suppressed the expressions of glycolysis-regulating genes in GBMs. (A~D) Cell viability.
Fig. 1. Changes in hippocampal matrix metalloproteinase-9 (MMP-9) and angiopoietin-2 (ANG-2) expression over time following bilateral common carotid artery.
Fig. 3. GABA content was increased in reactive astrocytes from 3D culture and hippocampal brain slice. (A) Immunostaining of neuron-astrocyte 3D co-culture.
A, B, Transcription factor p65 (A) and p50 (B) DNA binding activity in hippocampal nuclear extracts of LD and DD mice. A, B, Transcription factor p65 (A)
Fig. 2. Scriptaid and RGFP966 alter macrophage phenotype
Fig. 5. Subcellular distribution (soma, process, and microdomain) of MOR in astrocyte (indicated in blue). MOR is stained with immunogold with silver enhancement.
Fig. 1. Schematic diagram of behavioral procedures
Fig. 3. Effects of RP on SPS-CF stress-induced memory impairment of spatial memory. (A) Latency to find hidden platform in Morris water maze during 5 daily.
Fig. 1. HFD-induced obesity in C58BL/6 mice and decrease of general and motor behavior in HFD-fed mice. (A) HFD-fed, 5-week-old C57BL/6 mice (n=6/group)
Fig. 2. TH expression is decreased in mice with HFD-induced obesity
Fig. 1. Induction of microglial cell motility by OPNpt20
Fig. 6. The expression pattern of calretinin in the bouton endings of CAFs. (Aa~Bc) Single confocal slice images representing the pillar-(Aa~Ac) and modiolar-(Ba~Bc)
Fig. 2. Disease state-specific alteration of activity-dependent BDNF secretion from cortical axons. (A) Average fluorescence changes with time (ΔFt/F0)
Fig. 4. Chronic microwave treatment produced changes in glucose and corticosterone levels in blood. (A) Blood glucose levels of mice subjected to chronic.
Fig. 10. Summary of astrocyte specificity and coverage of hGFAP-CreERT2 in various brain regions. (A) Schematic sagittal section diagram shows heterogeneous.
Fig. 4. A theoretical model of a neural interfacing system to recover neural functions after brain injuries by neuro-prosthetic treatments. A schematic.
Fig. 2. GABAergic signaling in pathological states
Fig. 1. Three main axes of brain disease categories during a lifespan
Fig. 3. LPS induces degeneration of DA neurons and microglial activation in the SN in vivo. LPS (5 µg/3 µl) was unilaterally injected into the SN in the.
Fig. 8. Scriptaid and RGFP966 have no effect on functional recovery, lesion size or demyelinated area after SCI. BALB/c mice were subjected to a T-cut.
Fig. 9. Scriptaid and RGFP966 have no effect on Iba-+1 and CD4+ cell immune cell infiltration after SCI. BALB/c mice were subjected to a T-cut hemisection.
Fig. 3. Ultra-wide-field color FP and AF, spectral-domain OCT, and H&E staining images at one month after intravitreal MNU injection with pars plana vitrectomy.
Fig. 4. Dopaminergic signaling pathways are impaired in Ewsr1 KO (−/−) mice at 3 weeks of age. (A) qPCR analysis showed that Th mRNA was significantly.
Fig. 2. Ultra-wide-field color FP, AF, histology with H&E staining, and OCT images at one month after intravitreal injection of MNU without vitrectomy.
Fig. 3. hALDH1L1 promoter-driven Cre recombinase-mediated tdTomato expression in BLA. (A) Low magnification images of injection area in amygdala. Each.
Fig. 2. E-LTP is impaired in Rev-erbα KO mice during the subjective night. (A) The input-output relationships at SC-CA1 synapses showed no significant.
Fig. 8. Electron micrographs taken from 8-week glaucoma retina
Fig. 1. The brain of Octopus minor. (A) Left: a live specimen of O
Fig. 5. Phagocytosis of zymosan particles by BV2 cells
Fig. 1. Genetic confimation of hGFAP-CreERT2 mouse line and its tamoxifen inducible Cre expression. (A) Genetic map of hGFAP-CreERT2. (B) The primer sequences.
Presentation transcript:

Fig. 1. MS and MSEW mice did not exhibit depression-related behavior compared to controls. There was no significant difference in the time spent stationary on the tail suspension test between (A) MS mice and MC mice and (B) MSEW and MC mice. One mouse from each group in the MS cohort was excluded for excessive climbing and three mice from the MC group in the MSEW cohort were excluded for severe injuries. There was no significant difference in the immobility time during the second trial of a forced swim test between (C) MS and MC mice and (D) MSEW and MC mice. One mouse in the MS cohort was excluded for excessive hyperactivity and two mice in the MC group in the MSEW cohort were excluded for severe injuries. All values are means±SEM. Fig. 1. MS and MSEW mice did not exhibit depression-related behavior compared to controls. There was no significant difference in the time spent stationary on the tail suspension test between (A) MS mice and MC mice and (B) MSEW and MC mice. One mouse from each group in the MS cohort was excluded for excessive climbing and three mice from the MC group in the MSEW . . . Exp Neurobiol. 2017 Dec;26(6):390-398. https://doi.org/10.5607/en.2017.26.6.390