This Month in Gastroenterology Eugene B Chang  Gastroenterology  Volume 125, Issue 2, Pages 285-287 (August 2003) DOI: 10.1016/S0016-5085(03)00974-0

Figure 1 Effect of dietary calcium and ETEC infection on relative fecal dry weight of the subjects. Volunteer human subjects were orally infected with ETEC on day 0. The value at day 0 is the mean of the relative fecal dry weight in the pre-infection period. ∗P = 0.02. Gastroenterology 2003 125, 285-287DOI: (10.1016/S0016-5085(03)00974-0)

Figure 2 Macroscopic appearance of the livers of representative rats of Experiment B. CDAA alone resulted in the development of fatty cirrhotic liver (A), whereas other groups did not show any gross changes (B: CDAA+JTE-522 10mg/kg/day, C: CDAA+JTE-522 30 mg/kg/day, D: CSAA). Gastroenterology 2003 125, 285-287DOI: (10.1016/S0016-5085(03)00974-0)

Figure 3 H&E sections from livers of representative rats from each group of Experiment B (36 weeks). (A) CDAA induced cirrhotic nodule (left) and HCC (right). Note the marked infiltration of mononuclear cells in the fibrous septa. (B) CDAA plus JTE-522 at 10 mg · kg−1 · day−1 resulted in massive fatty change and prominent fibrogenesis. Some inflammatory cells are present in the fibrous tissues. (C) CDAA plus JTE-522 at 30 mg · kg−1 · day−1 resulted in mild fatty changes and mild fibrogenesis. (D) Normal liver of a representative control CSAA rat. Magnification: A-D, 100×. Gastroenterology 2003 125, 285-287DOI: (10.1016/S0016-5085(03)00974-0)