External iliac artery endofibrosis: a new possible predisposing factor Vincent Scavèe, MD, Laurent Stainier, MD, Thierry Deltombe, MD, Serge Theys, PhD, Monique Delos, MD, Jean-Paul Trigaux, MD, Jean-Claude Schoevaerdts, MD Journal of Vascular Surgery Volume 38, Issue 1, Pages 180-182 (July 2003) DOI: 10.1016/S0741-5214(03)00123-X
Fig 1 A, Digital substraction angiogram demonstrates a normal aorta and peripheral run-off vessels. B, Digital substraction angiogram obtained 6 weeks later shows left external iliac artery occlusion with collateral flow to the common femoral artery (arrow). Journal of Vascular Surgery 2003 38, 180-182DOI: (10.1016/S0741-5214(03)00123-X)
Fig 2 Computed tomography scan reveals left psoas muscle (LPM) hypertrophy, external iliac artery occlusion (arrow), and a patent hypogastric artery (dotted arrow). Journal of Vascular Surgery 2003 38, 180-182DOI: (10.1016/S0741-5214(03)00123-X)
Fig 3 A, Photomicrograph shows luminal thrombus (T) and endofibrosis (E) in the resected artery (hematoxylin-eosin; magnification 40×). M, media. B, Photomicrograph shows intimal thickening from proliferation of myofibroblasts (MF) (smooth muscle actine; magnification 100×). IEL, Internal elastic lamina; M, media. Journal of Vascular Surgery 2003 38, 180-182DOI: (10.1016/S0741-5214(03)00123-X)