Cardiovascular and metabolic profiles amongst different polycystic ovary syndrome phenotypes: who is really at risk? Nadine M.P. Daan, M.D., Yvonne V. Louwers, M.D., Ph.D., Maria P.H. Koster, M.D., Ph.D., Marinus J.C. Eijkemans, Ph.D., Yolanda B. de Rijke, Ph.D., Eef W.G. Lentjes, Ph.D., Bart C.J.M. Fauser, M.D., Ph.D., Joop S.E. Laven, M.D., Ph.D. Fertility and Sterility Volume 102, Issue 5, Pages 1444-1451.e3 (November 2014) DOI: 10.1016/j.fertnstert.2014.08.001 Copyright © 2014 American Society for Reproductive Medicine Terms and Conditions
Figure 1 Distribution of PCOS phenotypes in study population. AE = androgen excess; OD = ovulatory dysfunction; PCOM = polycystic ovarian morphology. Fertility and Sterility 2014 102, 1444-1451.e3DOI: (10.1016/j.fertnstert.2014.08.001) Copyright © 2014 American Society for Reproductive Medicine Terms and Conditions
Supplemental Figure 1 Plot graphs showing the LH, FSH, E2, and T assay conversion formulas between centers. Fertility and Sterility 2014 102, 1444-1451.e3DOI: (10.1016/j.fertnstert.2014.08.001) Copyright © 2014 American Society for Reproductive Medicine Terms and Conditions
Supplemental Figure 2 Plot graphs showing the DHEAS, A, SHBG, and insulin assay conversion formulas between centers. Fertility and Sterility 2014 102, 1444-1451.e3DOI: (10.1016/j.fertnstert.2014.08.001) Copyright © 2014 American Society for Reproductive Medicine Terms and Conditions