In vitro lymphokine responses of mesenteric LN cells from S

Slides:



Advertisements
Similar presentations
Fig. 3. IL-4, IFN-γ, and IL-17 production from α-GalCer-stimulated iNKT cells following FlaB treatment of PBMC cultures from asthma patients and healthy.
Advertisements

NK cells inhibited the expansion and function of ILC2s in vitro via IFN-γ. NK cells inhibited the expansion and function of ILC2s in vitro via IFN-γ. ILC2s.
Activation of NK cells during the early stage of allergic and fibrotic lung inflammation. Activation of NK cells during the early stage of allergic and.
Enhanced ILC2 expansion after depletion of NK cells during the early stage of allergic and fibrotic lung inflammation. Enhanced ILC2 expansion after depletion.
Synergistic antitumor effect of anti-CD40/CpG and 14
Antitumor effect of the combination of IL-2 IC and anti–CTLA-4.
Anti-CD40 and CpG induce activation of T cells in draining lymph nodes
Control of IL‐6 synthesis by ζPKC and RelA Ser311 phosphorylation.
Inflammasome responses.
The histamine-cytokine network in allergic inflammation
Expression and regulation of MD-2s.
MD-2s fails to mediate LPS-dependent NF-κB activation and IL-8 secretion. MD-2s fails to mediate LPS-dependent NF-κB activation and IL-8 secretion. A,
Volume 30, Issue 3, Pages (March 2009)
LP-BM5–induced increases of cytokine levels in the hind paw skin of B6 mice. LP-BM5–induced increases of cytokine levels in the hind paw skin of B6 mice.
H31m1-PDL1 cells form progressively growing tumors in WT mice.
Tumor development of poorly immunogenic LLC and B16F10 cells modified to produce GM-CSF was markedly inhibited. Tumor development of poorly immunogenic.
FIP200 deficiency alters mitochondria activation and ROS production in T cells. FIP200 deficiency alters mitochondria activation and ROS production in.
DKO CD8+ T cells demonstrate a strong effector response but altered memory differentiation and maintenance after LCMV infection. DKO CD8+ T cells demonstrate.
Maturation, Ag presentation capacity, and IL-12 production of DCs
MP cells established in Rag γc KO mice are Toxoplasma antigen–unspecific T-bet+ population. MP cells established in Rag γc KO mice are Toxoplasma antigen–unspecific.
CD8α+ DC-deficient mice are highly susceptible to Lm infection in the absence of CD169+ macrophages. CD8α+ DC-deficient mice are highly susceptible to.
Enhanced proliferation and cytokine production in DGKζ and Cbl-b deficient mice. Enhanced proliferation and cytokine production in DGKζ and Cbl-b deficient.
IL32 prompts cell activation and cancer-related pathways.
Histological changes in mouse colons after DSS treatment.
IL-10 upregulates IgG4 production by CD27+ B cells.
Effects of E. coli and S. aureus infection on levels of TNF-α and IL-1β in milk. Effects of E. coli and S. aureus infection on levels of TNF-α and IL-1β.
Measurement of a) IL-8, b) IL-6 and c) GM-CSF release by BEAS-2B cells stimulated with deoxycholic acid for 48 h. Measurement of a) IL-8, b) IL-6 and c)
Genetic FIP200 deletion impairs autophagy induction and causes T cell apoptosis. Genetic FIP200 deletion impairs autophagy induction and causes T cell.
β-Glucans do not modulate epithelial IL-33 or AHR.
IL-6 neutralization results in induction of CD161+ FOXP3+ CD127– Tregs in EC–CD4+ T cell cocultures. IL-6 neutralization results in induction of CD161+
MP cells can mediate resistance in infectious models that induce TH1-type immunity. MP cells can mediate resistance in infectious models that induce TH1-type.
IL-6 neutralization decreases T cell proliferation and cytokine production upon restimulation by allogeneic HUVEC. CIITA transduced HUVECs were cocultured.
Ag 85A–specific immune responses.
Tfr cell–derived IL-10 is important for B cell differentiation and the GC response. Tfr cell–derived IL-10 is important for B cell differentiation and.
IL-27 contributes to IR expression by lung T cells during toxoplasmosis. IL-27 contributes to IR expression by lung T cells during toxoplasmosis. (A) Il27p28-GFP.
Neutrophil recruitment to the colonic lamina propria depends on CD4+ T cells. Neutrophil recruitment to the colonic lamina propria depends on CD4+ T cells.
T-bethi MP cells produce IFN-γ in response to IL-12.
PD-1 blockade enhances T-cell function.
TSG-6 suppressed APC activation in vitro and in vivo.
BAP1 is required for homeostatic and antigen-driven expansion of peripheral T cells. BAP1 is required for homeostatic and antigen-driven expansion of peripheral.
IRS-1 and -2 expression in four mesangial cell lines from different D-NOD (A and B) and ND-NOD (C and D) mice. IRS-1 and -2 expression in four mesangial.
PEDF blockage improved the reduced numbers of circulating CD34+/CD133+/Flk-1+ cells and impaired EPC functions, including migration, adhesion, and tube.
Transgenic c-Maf mediates upregulation of type 2 cytokines in NOD CD4, but not CD8, cells. Transgenic c-Maf mediates upregulation of type 2 cytokines in.
Spontaneous and strong Tfh cell but not Tfr cell development in IL-2 KO mice. Spontaneous and strong Tfh cell but not Tfr cell development in IL-2 KO mice.
Loss of Tfh and GC B cells in 2KO-Bcl6TC mice.
Increased anti-DNA Abs in 2KO-Bcl6TC mice.
M-MDSC:mast cell cross-talk in the regulation of TNFα production.
Gab3 is required for IL-2– and IL-15–induced priming and expansion of NK cells. Gab3 is required for IL-2– and IL-15–induced priming and expansion of NK.
Vaginal CD11c+ DCs from IL-17A−/− mice are impaired in potentiating Th17 responses because of diminished IL-1β production. Vaginal CD11c+ DCs from IL-17A−/−
Kinetic of NFAT-CBR expression.
PD-1 inhibition stimulates the proliferation and cytokine secretion of exhausted/senescent CD8+ T cells in vitro. PD-1 inhibition stimulates the proliferation.
PD-L1 expressed on edited T3 sarcoma cells prevents their immune elimination. PD-L1 expressed on edited T3 sarcoma cells prevents their immune elimination.
T cell effector cytokines and staphylococcal products regulate CCL18.
Activation of Vγ9 T cells by three distinct stimuli is CD277 dependent
Expression of B7-H1, B7-DC, and PD-1 on B cells.
Characterization of mutant flagellin proteins.
p110δ activity is important for autoantibody responses.
Local proliferation of infiltrating circulating memory T cells.
VEGF165 stimulates migration of HMVECs
SPARC is required for spontaneous metastasis
Expression of IFN-λs, IFN-λR1, and IL-10R2 in Xenopus tissues after stimulation in vivo with poly(I:C). Expression of IFN-λs, IFN-λR1, and IL-10R2 in Xenopus.
Analysis of absolute CD4+ cytokine+ cell numbers pre- and post-ART.
ADU-S100 primes the innate immune system in tolerant, tumor bearing neu/N mice. ADU-S100 primes the innate immune system in tolerant, tumor bearing neu/N.
IL-33 is not critical for initiation of allergic airways disease phenotype. IL-33 is not critical for initiation of allergic airways disease phenotype.
Supplementary Figure 1. The extrinsic acquisition of CD80 does not affect the cytotoxicity of Ag-specific memory CD8+ T cells. The LG and SP were obtained.
IL-1β expression is increased in MM cell lines by the co-culture with platelets in vitro. IL-1β expression is increased in MM cell lines by the co-culture.
Fig. 5 IL-5–mediated signaling is critical for the development of CD1dintCD5+ Breg precursor cells and IL-10+ Breg cells. IL-5–mediated signaling is critical.
Acute circadian disruption alters ILC3 cytokine secretion.
Meningeal γδ T cell homeostasis is independent of inflammatory signals
Fig. 1 TH17 cell differentiation was severely impaired in Cxxc1-deficient mice. TH17 cell differentiation was severely impaired in Cxxc1-deficient mice.
Presentation transcript:

In vitro lymphokine responses of mesenteric LN cells from S In vitro lymphokine responses of mesenteric LN cells from S. mansoni-infected wt, IL-4 KO, IL-4Rα KO, and IL-4/IL-4Rα KO mice in response to SEA. Mesenteric LN cells were stimulated with SEA for 72 h, and lymphokine levels were measured in supernatants by ELISA. Results shown are means of duplicate values (±SD) for one of five experiments performed. In vitro lymphokine responses of mesenteric LN cells from S. mansoni-infected wt, IL-4 KO, IL-4Rα KO, and IL-4/IL-4Rα KO mice in response to SEA. Mesenteric LN cells were stimulated with SEA for 72 h, and lymphokine levels were measured in supernatants by ELISA. Results shown are means of duplicate values (±SD) for one of five experiments performed. Dragana Jankovic et al. J Immunol 1999;163:337-342 Copyright © 1999 by The American Association of Immunologists