Fig. 5. Phagocytosis of zymosan particles by BV2 cells

Slides:

Advertisements

Similar presentations

Histograms! Histograms group data that is close together into “classes” and shows how many or what percentage of the data fall into each “class”. It.

Advertisements

Fig. 2. The expression of pKr-2 in the substantia nigra (SN) of patients with Parkinson's disease (PD). (A, B) Western blot analysis for pKr-1-2 and pKr-2,

Supplementary Fig. 1 *** Cell viability (%) HepG2 3 (hr) DU145 A549 17

Fig. 3. Expression of Ninj1protein in cortices of the ischemic hemispheres of the rat brain at 4 day post-MCAO. Coronal brain sections were prepared at.

Journal of Surgical Research

Fig. 2. Effect of Ang II on apoptosis with CaMKII oxidation

Fig. 1. Infarct size and behavioral outcomes in response to single and repeated treatment of hUCB-MSCs after MCAO. (A) Schematic of the time schedule.

Fig. 9. Treatment of flagellin increased cell survival in hypoxic conditions. (A) After 2 h OGD, cell cytotoxicity levels were measured by a LDH assay.

CO20 CO 40 CO 80 Figure S1a: HR-TEM images and quantitative particle size distribution analysis of (A-B) CO20 ; (C-D) CO40 ; and (E-F) CO80 gold NPs.

Fig. 2. Infection by lentivirus

Invest. Ophthalmol. Vis. Sci ;45(7): doi: /iovs Figure Legend:

From: Reengineering Human Bruch's Membrane Increases Rod Outer Segment Phagocytosis by Human Retinal Pigment Epithelium Trans. Vis. Sci. Tech ;4(5):10.

Fig. 2. Expression of Ninj1 in cortices of ischemic hemispheres of rat brains at 1 day post-MCAO. Coronal brain sections were prepared at 1 day post-MCAO.

Fig. 6. Akt inhibitor IV reduced the activity of the NF-κB promoter in postconditioning. NF-κB activity was detected by a reporter gene assay in bEnd.3.

1α,25-Dihydroxycholecalciferol (Vitamin D3) Induces NO-Dependent Endothelial Cell Proliferation and Migration in a Three-Dimensional Matrix Cell Physiol.

From: Nerve Injury-related Autoimmunity Activation Leads to Chronic Inflammation and Chronic Neuropathic Pain Anesthes. 2013;118(2): doi: /ALN.0b013e31827d4b82.

Volume 1, Issue 2, Pages (August 2016)

Loss of USP8 or HIF1α impairs endocytic recycling required for ciliogenesis Kinetic analysis of transferrin recycling measured by flow cytometry. Loss.

PMA/THP-1 lipid uptake. PMA/THP-1 lipid uptake. PMA/THP-1 cells were incubated with 10% vol/vol Calogen for 24 h with and without inhibitors and were stained.

Nicotine inhibits hippocampal and striatal acetylcholinesterase activities, and demonstrates dual action on adult neuronal proliferation and maturation

Fig S1 Figure S1. Frequent mutation in clear cell renal cell carcinoma. Figure S1 shows the top 20 genes ordered by the occurrence of mutations from COSMIC.

The rate of hypo-osmotic challenge influences regulatory volume decrease (RVD) and mechanical properties of articular chondrocytes Z. Wang, J. Irianto,

ER stress response and susceptibility to apoptosis are regulated by TFEB and TFE3 ER stress response and susceptibility to apoptosis are regulated by TFEB.

Fig. 7 DMN-Tre labeling of Mycobacterium tuberculosis is inhibited by tuberculosis drug cocktail, unlike auramine staining. DMN-Tre labeling of Mycobacterium.

Fig. 1. Exogenous folic acid and Folr1 rescues the function of a Rho-kinase binding mutation in Shroom3. Exogenous folic acid and Folr1 rescues thefunction.

Fig. 1. Mitochondrial internalization in cardiomyocytes.

Fig. 6. Comparison of Plk4 with Sas-6 localization

Effects of doxorubicin and epirubicin on proliferation and apoptosis in the hyperplastic livers of transgenic zebrafish larvae. Effects of doxorubicin.

Fig. 6. STK35 KO mice show ovary defects.

MET activation increases the density of Syn-GFP/bassoon and Syn-GFP/PSD-95 clusters in neocortical neurons. MET activation increases the density of Syn-GFP/bassoon.

Fig. 6. Mucus analysis of lungs from allergic and tolerized mice

Transbilayer distribution of cholesterol in the plasma membrane is defective in staurosporine-treated cells. Transbilayer distribution of cholesterol in.

Analysis of Cdc7p at the end of meiosis.

Inflammatory stimuli downregulate macropinocytosis of exosomes in microglia. Inflammatory stimuli downregulate macropinocytosis of exosomes in microglia.

Fig. 3. Mice which experienced MS or MSEW did not exhibit impairments in hippocampal and amygdala-dependent fear-related emotional memories. In the acquisition.

Prophase nuclear movements in wild-type, rec8 and rec7 mutant cells.

Fig. 4. Reduction of apoptosis and autophagic stress by treatment with silibinin in the KA-treated hippocampus. (A, B) Double-immunofluorescence staining.

ChREBPα increases β-cell mitochondrial activity and ATP production in human islets. ChREBPα increases β-cell mitochondrial activity and ATP production.

Association of NM-HA and NM-GFP with SGs is transient.

Fig. 3. Neuroprotective effects of silibinin against KA-induced excitotoxicity. (A) Silibinin was intraperitoneally injected for 8 days, starting 1 day.

Fig. 5. Anti-inflammatory effects of silibinin on KA treatment in the hippocampus. (A) Representative coronal sections of the hippocampal CA1 region following.

Adipocytes sequester daunorubicin (DNR).

Fig. 1. MS and MSEW mice did not exhibit depression-related behavior compared to controls. There was no significant difference in the time spent stationary.

Fig. 3. OPNpt20 binding to αv integrin in BV2 cells

Fig. 2. High density EEG of anterior and posterior spindles

Fig. 2. Induction of F-actin polymerization by OPNpt20

Fig. 4. Activations of FAK, Erk, and Akt pathways by OPNpt20

Fig. 3. Comparing between source localization algorithms

Fig. 3. Inhibition of HIF1α with chrysin induced cell death and suppressed the expressions of glycolysis-regulating genes in GBMs. (A~D) Cell viability.

Fig. 1 EVs transport ODN into recipient cells.

Peripheral CAF model. Peripheral CAF model. A, bright field (top) and fluorescence (bottom) micrographs of aggregates that contain RFP-tagged 344SQ cells.

Perifosine affects membrane distribution of HA-Akt1 and Akt1-PH domain constructs. Perifosine affects membrane distribution of HA-Akt1 and Akt1-PH domain.

Fig. 5. Subcellular distribution (soma, process, and microdomain) of MOR in astrocyte (indicated in blue). MOR is stained with immunogold with silver enhancement.

Fig. 2. TH expression is decreased in mice with HFD-induced obesity

Fig. 1. Induction of microglial cell motility by OPNpt20

Fig. 4. Chronic microwave treatment produced changes in glucose and corticosterone levels in blood. (A) Blood glucose levels of mice subjected to chronic.

Fig. 10. Summary of astrocyte specificity and coverage of hGFAP-CreERT2 in various brain regions. (A) Schematic sagittal section diagram shows heterogeneous.

Fig. 3. KMS99220 inhibits activation of NFκB signaling via HO-1 induction. (A) BV2 cells were treated with 10 µM KMS99220 for 1 h, and then treated with.

Fig. 1. DACs immunolabeled with TH antibodies in the rabbit retina

Fig. 3. LPS induces degeneration of DA neurons and microglial activation in the SN in vivo. LPS (5 µg/3 µl) was unilaterally injected into the SN in the.

Fig. 8. Scriptaid and RGFP966 have no effect on functional recovery, lesion size or demyelinated area after SCI. BALB/c mice were subjected to a T-cut.

Fig. 9. Scriptaid and RGFP966 have no effect on Iba-+1 and CD4+ cell immune cell infiltration after SCI. BALB/c mice were subjected to a T-cut hemisection.

Fig. 3. Ultra-wide-field color FP and AF, spectral-domain OCT, and H&E staining images at one month after intravitreal MNU injection with pars plana vitrectomy.

Fig. 2. Ultra-wide-field color FP, AF, histology with H&E staining, and OCT images at one month after intravitreal injection of MNU without vitrectomy.

Fig. 3. hALDH1L1 promoter-driven Cre recombinase-mediated tdTomato expression in BLA. (A) Low magnification images of injection area in amygdala. Each.

Fig. 8. Electron micrographs taken from 8-week glaucoma retina

Fig. 7. Apoptotic cell death was evaluated via TUNEL

Fig. 1. The brain of Octopus minor. (A) Left: a live specimen of O

Tumor spheroid induces mesothelial cell migration.

Fig. 5 Electrical stimulation of nerve cells powered by BD-TENG.

Presentation transcript:

Fig. 5. Phagocytosis of zymosan particles by BV2 cells Fig. 5. Phagocytosis of zymosan particles by BV2 cells. BV2 cells were incubated with FITC-labeled zymosan particles and monitored by live cell imaging system for 12 hrs. Phagocytosis of zymosan particles was monitored every 5 minutes for 12 hrs (A~H), and percentages of internalized zymosan particles (I), zymosan positive cells (J), and numbers of zymosan particles per cell (K) were determined by counting 10 photographic fields at 0, 1, 2, 6, and 12 hrs and results are presented as means±SEMs. Arrows indicate internalized zymosan particles. **p<0.05 versus PBS-treated control and ##p<0.01, $$p<0.01 between indicated groups. Scale bars represent 125 µm (A) or 50 µm (B~E). Fig. 5. Phagocytosis of zymosan particles by BV2 cells. BV2 cells were incubated with FITC-labeled zymosan particles and monitored by live cell imaging system for 12 hrs. Phagocytosis of zymosan particles was monitored every 5 minutes for 12 hrs (A~H), and percentages of internalized zymosan particles (I), zymosan positive cells (J), and numbers of zymosan particles. . . Exp Neurobiol. 2017 Dec;26(6):339-349. https://doi.org/10.5607/en.2017.26.6.339