High Prevalence of Hypertension and End-Organ Damage Late After Coarctation Repair in Normal Arches Melissa G.Y. Lee, MBBS, BMedSci, Sarah L. Allen, MD, BBiomed, Ryo Kawasaki, MD, PhD, Aneta Kotevski, BMedSci, PhD, Jane Koleff, DipMedLabSc, Remi Kowalski, MBBS, Michael M.H. Cheung, MBChB, Igor E. Konstantinov, MD, PhD, Christian P. Brizard, MD, Yves d’Udekem, MD, PhD The Annals of Thoracic Surgery Volume 100, Issue 2, Pages 647-653 (August 2015) DOI: 10.1016/j.athoracsur.2015.03.099 Copyright © 2015 The Society of Thoracic Surgeons Terms and Conditions
Fig 1 Echocardiographic measures of end-organ damage after coarctation repair. (A) Increased carotid intima-media thickness. (B) Left ventricular hypertrophy. (ASE = American Society of Echocardiography; EDV = end-diastolic volume; EF = ejection fraction; %FS = percent fractional shortening; IMT = intima-media thickness; IVSd = interventricular septal end diastole; IVSs = interventricular septal end systole; LVd = left ventricular diastolic; LVIDs = left ventricular internal diameter end systole; LVPT = left ventricular pacing threshold; LVPWs = left ventricular posterior wall at end systole; SV = stroke volume.) The Annals of Thoracic Surgery 2015 100, 647-653DOI: (10.1016/j.athoracsur.2015.03.099) Copyright © 2015 The Society of Thoracic Surgeons Terms and Conditions
Fig 2 Retinal image demonstrating tortuous and kinked vessels after coarctation repair. The Annals of Thoracic Surgery 2015 100, 647-653DOI: (10.1016/j.athoracsur.2015.03.099) Copyright © 2015 The Society of Thoracic Surgeons Terms and Conditions