بسم الله الرحمن الرحيم.

Slides:



Advertisements
Similar presentations
2) Closed wound: Skin is intact (not opened) include crushing injury and contusions. Wounds A) Skin involvement: 1) Open wound: when the whole thickness.
Advertisements

FIRE DEATHS The Role of the Forensic Pathologist Marie Cassidy State Pathologist.
Alyssa Morris, R4 August 5 th, THE CASE THE A-TEAM.
Burns Heat, electricity, radiation, certain chemicals  Burn (tissue damage, denatured protein, cell death) Immediate threat: –Dehydration and electrolyte.
Elsevier items and derived items © 2010, 2006, 2002 by Saunders, an imprint of Elsevier Inc. Chapter 28 Care of Patients with Burns.
Emergency Department Warwick Hospital
Definition: Burn is the loss of epithelium and a varying degree of dermis due to exposure to physical form of energy, certain chemicals or radiation.
Care of the Burn Patient Presented by Annmarie Keck RN, CEN, EMT-B Northwest MedStar Clinical Outreach Educator.
Burns of the Integument tissue damage inflicted by intense heat, electrical, radiation, or certain chemicals all of which denature cell proteins immediate.
Effect of electric current on human body. What is electric current?
Burns By Matthew & Ivan. Anatomy of the Skin The anatomy of the skin is complex, and there are many structures within the layers of the skin. There are.
Infarct: Definition: An infarct is a localized area of ischemic necrosis resulting from sudden and complete occlusion of its arterial blood supply without.
1 Regulation of Body Temperature Regulation of body temperature is vitally important because even slight shifts can disrupt metabolic reactions.
Dr.Adnan Gelidan FRCS( C ), FACS Assistant Professor Of Surgery Plastic Surgery KSU.
Thermoregulation, Healing and Aging. Regulation of Body Temperature  Slight shifts in temperature can disrupt metabolic rates  Stable temperature is.
BURNS IN CHILDREN A Lecture by Dr. B. O. Edelu Department of Paediatrics.
Forensic Medicine & Toxicology. By: Dr. Amina Afzal Rao ASPHYXIA.
Early signs of death. EARLY SIGNS OF DEATH Mate Žaja Mentor: A. Žmegač Horvat early changes rigor mortis cadaveric rigidity post-mortem hypostasis cooling.
1 BURNS Temple College EMS Professions. 2 Anatomy of Skin l Largest body organ l More than just a passive covering.
BURNS. Types of burns Depths of burns Extent of burns General Treatment Others Airway burns Electrical burns Chemical splashes to eyes.
Burns By: Vera Ware.
BURNS Incidence and Causes 8,000-10,00 burns per year in the U.S.A.
Lesson 10: Burns Emergency Reference Guide p
PHYSICAL INJURIES Causes and types: 1- Dry heat application of the hot solid 2- Moist heart e.g. hot liquids scalds 3- Acids and alkalis corrosive burns.
Gas Gangrene A Presentation by Jennifer Kent-Baker.
Burns Degree of Burns 1 st superficial partial-thickness burn 2 nd deep partial- thickness burn 3 rd full-thickness burn.
Interventions for Clients with Burns. Burn Injury 10,000 deaths/year More common in men Death rates high in kids and older adults Most deaths happen in.
Soft Tissue Injuries Burns
Physical Injuries PresentedBy Said Said Elshama Learning Objectives 1- Types of physical injuries 2- Dry burn 3- Moist burn 4- Electrocution 5- Corrosive.
Slide 1 Copyright © 2007 by Mosby, Inc., an affiliate of Elsevier Inc. All rights reserved. Chapter 43 Care of the Patient with an Integumentary Disorder.
Healing of Wounds and Burns & the Aging of Skin Chapter 6 Sections 5 & 6Chapter 6 Sections 5 & 6.
Burns Aaron J. Katz, AEMT-P, CIC
Dr. Maria Auron, Ilembula 2014
FORENSIC PATHOLOGY ELECTROCUTION.
First Aid Burns. Burns Classified as either Thermal (Heat) Chemical Electrical.
Skin Homeostatic Imbalances. Slide 4.23 Copyright © 2003 Pearson Education, Inc. publishing as Benjamin Cummings  Infections  Athletes foot  Caused.
FIRST AID AND EMERGENCY CARE LECTURE 8
BURN & SCALD. BURN –Dry burn is the tissue damage by dry heat of –Open flame, –Hot object, –Molten metal –Electricity –Friction with rapid moving Machine,
4% of fatal industrial accident Personnel at risk: Electricians Operators of high - power electric equipment &maintenance.
First Aid for Divers Burns 1 FAD 09 v1.3 Copyright © BSAC 2009 Burns.
Burn Injuries Rule of Nines
 An injury caused by heat, cold, electricity, chemicals, light, radiation, friction.  Highly variable in terms of the tissue affected, the severity,
Homeostatic Imbalance of the Skin
BURN S.
Integumentary System Chapter 5.
Burns management Ruqayah A Al Hajji.
Burns and scalds Burns = dry burn scald = wet or moist burn
D.Mahamed Hussein General Surgeon Azadi Teaching Hospital
Fundamentals of Anatomy & Physiology
Give 3 examples of Skin cancer
Chapter 7.
Burn Injuries & Its Management
BURNS Dr.Ishara Maduka M.B.B.S. (Colombo)
Pg 30 Burns Protein denaturation and cell death caused by heat, electricity, UV radiation/X-rays, or chemicals 2 main dangers Dehydration–Loss of fluids.
NURSING CARE MANAGEMENT OF BURNS IN ER
Chapter 5.
BURNS Burns can result from dry heat(fire), moist heat (steam,hot liquids), electricity, chemicals, and radiation (sunlight)
Lesson 10: Burns Emergency Reference Guide p
Lesson 10: Burns.
Physical Injuries Dr. Asmaa Fady Ph D., MSC., M.B, B.Ch.
Burn Patient Rehabilitation Prof.Dr: Ehab Kamal Zayed.
Burns and Sunburns. What is a burn? burn = damage to skin or other body parts caused by extreme heat, flame, contact with heated objects, or chemicals.
Presentation transcript:

بسم الله الرحمن الرحيم

Definition Dr. Doaa Abdel Wahab Degree of burn MEDICOLEGAL ASPECTS M.L. aspects of thermal injuries Degree of burn Definition Types of thermal injuries MEDICOLEGAL ASPECTS OF THERMAL INJURIES Old classification Recent classification A thermal injury is a tissue injury resulting from the application of heat in any form to the external or internal surface of the body. First degree burn Epidermal degree 1st & 2nd degree Dry or simple burn. Scald or moist burn. Chemical or corrosive burn. Electric burn. Radiation burn. Second degree burn By Third degree burn Dermo - epidermal burn 3rd & 4th degree Fourth degree burn Dr. Doaa Abdel Wahab Lecturer of Forensic Medicine & Clinical Toxicology Fifth degree burn Deep burn 5th & 6th degree Sixth degree burn

Eaten Singed Wet Wet With mineral acid Present Absent M.L. aspects of thermal injuries Dry burn Scald Corrosive Degree Any degree 1st, 3rd, 4th 1st, 2nd & 3rd Air passage Contains soot No soot No soot Eaten Hair Singed Wet Blood Thick, viscid & contains COHb Skin Dry & charred Sodden & bleached Stained & corroded Flame or heated body Cause Steam or hot liquid Corrosive acid or alkali Much disfigurement Thick with disfigurement Less disfigurement Scar Eaten Clothes Burnt Wet From below upward From above downward At & below site of contact Site & spread With mineral acid Charring Present Absent At circumference of burnt area Over burnt area Vesicles Rarely found

M.L. aspects of thermal injuries Factors affecting degree of burn A) Extent of burnt area: is determined by rule of nine of Wallace. B) Depth of burn: The 3rd degree burn is the most serious one. C) Site of burn. Neck, abdominal wall or genitalia are more dangerous than those of the extremities. D) Age E) Sex F) General health.

II) Rapid death "within 6-48 hours”: M.L. aspects of thermal injuries Causes of death from burn IV) Death after one week: II) Rapid death "within 6-48 hours”: III) Death within 2-7 days: Immediate causes "within 6 hours“ Neurogenic shock. Associated accidental serious injuries to vital organs. Asphyxia (suffocation). Traumatic asphyxia. Secondary oligaemic shock. Secondary toxaemic shock. Acute oedema of glottis (in the neck). Pulmonary fat embolism (burn in a fatty area.( Supra-renal haemorrhage. Bronchopneumonia. Inflammation of serous membranes. Septicemia. - Tetanus, gangrene and erysipelas.: - Rupture of an acute duodenal ulcer (at 12th day) (curling's ulcer) (stress ulcer): may be due to hypovolaemia causing devitalization of mucous membrane in addition to the absorbed burn toxins which are excreted by the liver into the duodenum. -Liver, kidney or heart damage: cell degeneration and necrosis of these organs due to the effect of burn toxins metabolism.

M.L. aspects of thermal injuries Burn vitality Age of Burn Antemortem burn Postmortem burn Appears immediately Erythema 36 hs 48 hs Line of hyperemia Present Absent Appears 2-3hs Vesicles 1 w 2 ws Presence of COHb Vesicles Albuminous fluid rich in chloride Present Absent absent poor in albumin and chloride Sepsis 2 – 3 days 36 hs Vital reaction Infl., pus healing Nil Superficial slough 1 w Nothing But burn Other causes Cause of death Deep slough 3 ws Soot in URT Present Absent Red Coppery Scar 6 m 2 m

M.L. aspects of thermal injuries Postmortem Picture of Burn Complications of Burn - Particles of soot are present in the air passage. Subcutaneous or subserous peticheal hge may be present . Haemoconcentration of blood due to loss of plasma and cherry red colour of the skin. - Skull may show thermal fracture "fissure fracture“. - The body shows evidence of antemortem burns of various degrees. The body presents a peculiar attitude; attitude of defence, boxing or "Pugilistic attitude". Crimson red colour of hypostasis. Generalized visceral congestion and Hge in the internal organs. Sepsis Deformities Keloid formation

Postmortem signs of burn Other postmortem signs of death M.L. aspects of thermal injuries Points of differentiation () thermal & traumatic fracture Traumatic Fracture Thermal Fracture The fracture is displaced Postmortem signs of burn The fracture is not displaced The brain is edematous Other postmortem signs of death & associated with contused wound in scalp The brain is shrunken Extradural haematoma fill the space Extradural haematoma doesn’t fill the space

Electric burn Physical Physiological Resistance of body tissue Voltage M.L. aspects of thermal injuries Electric burn Factors influencing the effect of electric current Physical Physiological Resistance of body tissue Voltage Humidity Nature of current Pathway of current Anticipation of shock Duration of contact General health

M.L. aspects of thermal injuries Ventricular fibrillation Causes of Death Ventricular fibrillation Central Respiratory failure Peripheral Cardiac arrest Cerebral anoxia Electric burn Hyperthermic effect

Rapid onset of rigor mortis Compression of the stratum corni which M.L. aspects of thermal injuries Current Marks P.M. Picture of Electric Burn N / E appearance Microscopic appearance Rapid onset of rigor mortis Compression of the stratum corni which stains deeply with superficial carbonization. Separation of the cells in the form of slits (electric channels). - Elongation of both cells and nuclei. It is of the same size and shape as the conductor. Grayish-white painless areas of aseptic necrosis. - They vary from superficial circumscribed lesions to severe burns with full thickness tissue necrosis. Marked hypostasis Presence of current mark Internal signs of asphyxia

M.L. aspects of thermal injuries Lightining Lightning syndrome Definition The electric current is DC not AC. About 20.000 amperes. About 100 - 1000 million volts. - A single flash lasts 1/1000th of a second. Characterized by: Loss of consciousness. Skin burn (similar to 1st degree burn. Conductive deafness. It is the discharge of the atmosphere potential () clouds, or () clouds and Earth.