Cavus Foot N. Craig Stone M.D. F.R.C.S.(C) Discipline of Orthopedic Surgery Sept 29, 2003.

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Presentation transcript:

Cavus Foot N. Craig Stone M.D. F.R.C.S.(C) Discipline of Orthopedic Surgery Sept 29, 2003

Cavus Foot Introduction Definition Anatomy and Pathomechanics Etiology and differential diagnosis Evaluation Clinical and radiographic Treatment

Cavus Foot Definition Abnormal elevation of the medial arch in weight bearing Fore foot equinus relative to hindfoot ?what’s normal/abnormal

Normal Anatomy and Biomechanics Forefoot deformity and the windlass mechanism of the plantar fascia causative Plantar fascia Calcaneal tuberosity – Transverse metatarsal lig – slips to base of prox phalanx Medial and central portions strongest Stabilizes arch and inverts (with tib post) the hindfoot

Anatomy and Biomechanics Chopart’s joint supple when hindfoot everted Heel strike – hindfoot inverted Midstance – hindfoot everted Shock absorption – now hindfoot supple Toe off

Anatomy and Biomechanics Toe off Toes dorsiflex Tib post fires All to lock hindfoot Gives a rigid, long lever for triceps surae

Pathomechanics Foot musculature unbalanced Usually intrinsic muscle weakness Lumbrical weakness allows EDL to hyperextend the MCP’s and FDL to flex the PIP and DIP’s Exaggeration of the windlass mechanism

Pathomechanics

Same applied to EHL and FHL 1 st ray more mobile – makes it worse, forefoot supinates and may become fixed Secondary hindfoot varus Tripod effect

Pathomechanics

So why does it hurt? Inverted hindfoot loses shock absorption ability Recurrent ankle sprains Tripod effects (less surface area) Clawing of toes

Etiology CNS Spinal Peripheral Nerves Other Idiopathic

Etiology - CNS CP esp. hemiplegia Spastic tib post Friedreich’s Ataxia (A. Recessive chrom 9) Triad – ataxia, downgoing Babinski, areflexia

Etiology - Spinal Myelodysplasia Syringomyelia Polio Spinal cord tumors Tethered cord Guillain-Barre syndrome

Etiology – Peripheral Nerves Hereditary Sensorimotor Neuropathy (HSMN) Charcot Marie Tooth

Etiology - Other Traumatic Isolated Tendon Injuries Partial Sciatic Nerve injury Volkman’s Contracture

Etiology - Idiopathic 20-50% of cases - mostly bilateral

Clinical Evaluation History Other neuro symptoms Ulcers, numbness, bowel, bladder, Dev. Delay Family History Ankle Instability Metatarsalgia

Clinical Evaluation Physical Dysraphism Neuro exam Coleman Block Test

Radiographic Assessment Standing AP and Lateral of Foot and Ankle Assess angles (severity) and any evidence of degenerative change Spinal Imaging as required

A – Meary’s Angle N = 0 – 5 Degrees B – Calcaneal Pitch Angle N = 30 degrees C – Hibbs Angle N = <45 degrees D – Weight Bearing Tibioplantar Angle N = 90 degrees

Management Blah, Blah, Blah Orthotics For mild, non progressive deformity Lateral forefoot and hindfoot posting Large toe box shoes

Management Surgical Treat underlying problem Must decide if hindfoot is supple Everyone gets a plantar fascial release Fixed – Supple is often subjective Combination of procedures

Management Hindfoot supple Toe deformity correction Girdlestone-Taylor Forefoot correction Metatarsal osteotomies Midfoot osteotomy Is there any role for a Jones procedure?

Management Hindfoot supple Tendon Transfers If identifiable muscle imbalance Split Tib post to peroneus brevis Peroneus longus to brevis Be careful in progressive disease

Management Rigid Hindfoot

If deformity severe or Degenerative Changes exist Triple Arthrodesis

Summary Rare problem Know causes and clinical assessment Principles of treatment