RUMORS: How can they work ? Summer School Math Biology, 2007 Nuno & Sebastian.

Slides:



Advertisements
Similar presentations
Work and play: Disease spread, social behaviour and data collection in schools Dr Jenny Gage, Dr Andrew Conlan, Dr Ken Eames.
Advertisements

Modeling of Complex Social Systems MATH 800 Fall 2011.
R 0 and other reproduction numbers for households models MRC Centre for Outbreak analysis and modelling, Department of Infectious Disease Epidemiology.
Disease emergence in immunocompromised populations Jamie Lloyd-Smith Penn State University.
Epidemics Modeling them with math. History of epidemics Plague in 1300’s killed in excess of 25 million people Plague in London in 1665 killed 75,000.
In biology – Dynamics of Malaria Spread Background –Malaria is a tropical infections disease which menaces more people in the world than any other disease.
Yellow Fever in Senegal HannahIsaac. Outline Disease Background Disease Background Model Model Comparison with Data Comparison with Data Model Predictions.
Probability of a Major Outbreak for Heterogeneous Populations Math. Biol. Group Meeting 26 April 2005 Joanne Turner and Yanni Xiao.
RD processes on heterogeneous metapopulations: Continuous-time formulation and simulations wANPE08 – December 15-17, Udine Joan Saldaña Universitat de.
Modelling Infectious Disease … And other uses for Compartment Models.
Section 10.1 Basic Properties of Markov Chains
Population dynamics of infectious diseases Arjan Stegeman.
Rumour Dynamics Ines Hotopp University of Osnabrück Jeanette Wheeler Memorial University of Newfoundland.
ODE and Discrete Simulation or Mean Field Methods for Computer and Communication Systems Jean-Yves Le Boudec EPFL MLQA, Aachen, September
Nik Addleman and Jen Fox.   Susceptible, Infected and Recovered S' = - ßSI I' = ßSI - γ I R' = γ I  Assumptions  S and I contact leads to infection.
Persistence and dynamics of disease in a host-pathogen model with seasonality in the host birth rate. Rachel Norman and Jill Ireland.
Modelling Two Host Strains with an Indirectly Transmitted Pathogen Angela Giafis 20 th April 2005.
HIV in CUBA Kelvin Chan & Sasha Jilkine. Developing a Model S = Susceptible I = Infected Z = AIDS Patients N = S+I = Active Population.
Code Red Worm Propagation Modeling and Analysis Zou, Gong, & Towsley Michael E. Locasto March 4, 2003 Paper # 46.
Preventing Smallpox Epidemics Using a Computational Model By Chintan Hossain and Hiren Patel.
Joanne Turner 15 Nov 2005 Introduction to Cellular Automata.
1 The epidemic in a closed population Department of Mathematical Sciences The University of Liverpool U.K. Roger G. Bowers.
Modeling the SARS epidemic in Hong Kong Dr. Liu Hongjie, Prof. Wong Tze Wai Department of Community & Family Medicine The Chinese University of Hong Kong.
Network modeling of the Ebola Outbreak Ahmet Aksoy.
Universal Behavior in a Generalized Model of Contagion Peter S. Dodds Duncan J. Watts Columbia University.
How does mass immunisation affect disease incidence? Niels G Becker (with help from Peter Caley ) National Centre for Epidemiology and Population Health.
WHAT IS PERSONALITY? Why would we want to study personality?
Epidemiology modeling with Stella CSCI Stochastic vs. deterministic  Suppose there are 1000 individuals and each one has a 30% chance of being.
Population Biology: PVA & Assessment Mon. Mar. 14
Matrix Algebra and Applications
Extra Slides Unit 4: Classifications of Disease Outbreak Unit 5: Framingham Study.
Informing disease control strategies using stochastic models S.
Nina H. Fefferman, Ph.D. Rutgers Univ. Balancing Workforce Productivity Against Disease Risks for Environmental and Infectious.
Code Red Worm Propagation Modeling and Analysis Cliff Changchun Zou, Weibo Gong, Don Towsley Univ. Massachusetts, Amherst.
CODE RED WORM PROPAGATION MODELING AND ANALYSIS Cliff Changchun Zou, Weibo Gong, Don Towsley.
Directed-Graph Epidemiological Models of Computer Viruses Presented by: (Kelvin) Weiguo Jin “… (we) adapt the techniques of mathematical epidemiology to.
Modelling infectious diseases Jean-François Boivin 25 October
MA354 Mathematical Modeling T H 2:45 pm– 4:00 pm Dr. Audi Byrne.
Data and modeling issues in population biology  Alan Hastings (UC Davis) and many colalborators  Acknowledge support from NSF.
1 Worm Propagation Modeling and Analysis under Dynamic Quarantine Defense Cliff C. Zou, Weibo Gong, Don Towsley Univ. Massachusetts, Amherst.
Markovian susceptible-infectious- susceptible (SIS) dynamics on finite networks: endemic prevalence and invasion probability Robert Wilkinson Kieran Sharkey.
Showcase /06/2005 Towards Computational Epidemiology Using Stochastic Cellular Automata in Modeling Spread of Diseases Sangeeta Venkatachalam, Armin.
Modeling frameworks Today, compare:Deterministic Compartmental Models (DCM) Stochastic Pairwise Models (SPM) for (I, SI, SIR, SIS) Rest of the week: Focus.
Dynamic Random Graph Modelling and Applications in the UK 2001 Foot-and-Mouth Epidemic Christopher G. Small Joint work with Yasaman Hosseinkashi, Shoja.
Epidemic (Compartment) Models. Epidemic without Removal SI Process Only Transition: Infection Transmission SIS Process Two Transitions: Infection and.
Epidemics Pedro Ribeiro de Andrade Gilberto Câmara.
CDC's Model for West Africa Ebola Outbreak Summarized by Li Wang, 11/14.
Evaluating Persistence Times in Populations Subject to Catastrophes Ben Cairns and Phil Pollett Department of Mathematics.
Simulation of Infectious Diseases Using Agent-Based Versus System Dynamics Models Omar Alam.
MA354 An Introduction to Math Models (more or less corresponding to 1.0 in your book)
Predicting the Future To Predict the Future, “all we have to have is a knowledge of how things are and an understanding of the rules that govern the changes.
SS r SS r This model characterizes how S(t) is changing.
Yellow Fever in Senegal: Strategies for Control Nicholas Eriksson, Heather Lynch,
Vocabulary Infectious Diseases. Disease Classifications Emerging Infectious Diseases Re-emerging Infectious Diseases 1. Have not occurred in humans before,
Biao Wang 1, Ge Chen 1, Luoyi Fu 1, Li Song 1, Xinbing Wang 1, Xue Liu 2 1 Shanghai Jiao Tong University 2 McGill University
The Computational Nature of Language Learning and Evolution 10. Variations and Case Studies Summarized by In-Hee Lee
SIR Epidemics: How to model its behavior? Soonmook Lee 1.
Mean Field Methods for Computer and Communication Systems Jean-Yves Le Boudec EPFL Network Science Workshop Hong Kong July
Stochastic Spatial Dynamics of Epidemic Models
Review of Probability Theory
Sangeeta Venkatachalam, Armin R. Mikler
LIABILITY PORTFOLIO MANAGEMENT Diversification of Longevity and Mortality Risk Stuart Silverman, FSA, MAAA, CERA Longevity 12 September 29-30, 2016.
Modelling infectious diseases
Effective Social Network Quarantine with Minimal Isolation Costs
Predicting the Future To Predict the Future, “all we have to have is a knowledge of how things are and an understanding of the rules that govern the changes.
تقویت سورویلانس و بیماریابی
Predicting the Future To Predict the Future, “all we have to have is a knowledge of how things are and an understanding of the rules that govern the changes.
(Hidden) assumptions of simple compartmental ODE models
Anatomy of an Epidemic.
Susceptible, Infected, Recovered: the SIR Model of an Epidemic
Presentation transcript:

RUMORS: How can they work ? Summer School Math Biology, 2007 Nuno & Sebastian

Outline What is a rumor ? Deterministic vs Stochastic Simple models Not so simple models Summary Discrete & Galton-Watson Process

“unverified proposition of belief that bears topical relevance for persons actively involved in its dissemination” “unauthenticated bits of information in that they are deprived of “secure standards of evidence”.” What is a rumor ? ?

Deterministic vs Stochastic How can we model a rumor ? Is a deterministic or stochastic approach better ? How do these approaches differ ?

Deterministic vs Stochastic Discrete Galton-Watson Process Markov Chain Esteban likes it !!!! WHY ?

Simple models Deterministic Natural recoveredForced recovered Mass action interaction between Infectious and the total population Natural recoveredForced recovered Infectious after 1 time step Infectious 1 time step before Recovered 1 time step before Recovered after 1 time step

Simple models Probability of infected someone Probability of doing nothing Number of infected after forced recovery Natural recoveredForced recovered Infectious after 1 time step Recovered 1 time step before Recovered after 1 time step Stochastic Probability of forgetting the rumor

Simple model assumptions 1. Total population size is extremely large; 2. The number of susceptibles remains roughly constant; 3. The size of the epidemic remains quite small; 4. Mass action interaction (homogeneous population); 5. In the stochastic model, forced recovery precedes other events;

SIMPLE MODEL - Deterministic Results Infected 0 2 “types” fixed points (I*,R*) = (0,0) and (I*,R*)=(0,R) eigenvalue of 1 ? “epidemic” if (αN/  ) > 1.

SIMPLE MODEL - Stochastic Results Stochastic model extinction of the rumor!!!

Effect of I 0 on rumor life-time (both models) Rumor life-time is inversely proportional to I 0

“Strange” Results: Effect of α on rumor lifetime αN/  < 1αN/  > 1

Simple model Extinction of the rumor √ × √ × ?

Not so simple models models Deterministic Susceptible after 1 time step Recovered that become susceptible again

Not so simple models models Stochastic Probability that recovered that become susceptible again

Not so simple model assumptions 1. Total population size is constant; 2.Mass action interaction (homogeneous population); 3.In the stochastic model, forced recovery precedes other events;

NOT SO SIMPLE MODEL - Deterministic results ELVIS IS ALIVE!?!?! Model with model extinction and “endemic” rumors None of the fixed points are stable...

NOT SO SIMPLE MODEL - Stochastic results

Effect of population size – stochastic model

Effect of population size – deterministic model For the deterministic case population size only changes the scale of the epidemic In the stochastic model however, increasing the population size generates very different behaviour

Not so simple model – comparison of deterministic & stochastic results For large  (~ p4) coexistence is observed in both deterministic and stochastic For small  deterministic predicts repeated outbreaks of the rumor. This is not possible in the stochastic model (by varying p4) For the deterministic model the population size does not make any difference, but population size affects the predictions of the stochastic model

Summary 1.Rumors can be modelled similarly to infectious diseases; 2. Not so different models can give us very different predictions; 3. Under certain conditions, stochastic models predict very different results from deterministic ones

Acknowledgments Julien Jungmin Thank you very much !! Group