EPIGENETICS AND CANCER JILLIAN FROELICK, GRACE LEMPP, NIKHIL UMESH, PAIGE TUMMONS.

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EPIGENETICS AND CANCER JILLIAN FROELICK, GRACE LEMPP, NIKHIL UMESH, PAIGE TUMMONS

THERE ARE 3 MAIN EPIGENETIC MECHANISMS. THEY ARE… DNA METHYLATION HISTONE ACETYLATION AND NON-CODING RNA

IN THE METHYLATION OF A TUMOR-SUPPRESSOR GENE…

1. A methyl group attaches directly to the gene.

2. The tumor-suppressor gene can’t be transcribed because the transcription complex can’t bind.

3. With the tumor- suppressor gene inactivated, cell division isn’t regulated.

4.4. Cancer cells are able to multiply.

WHEN DNA HISTONES OF A TUMOR-SUPPRESSOR GENE ARE DEACETYLATED…

1. An acetyl group is removed from the lysine of a histone tail.

2. Chromatin, which was previously loose, is now tightly compacted.

3. The tumor-suppressor gene cannot be transcribed.

4.4. Cancer cells are able to multiply.

WHEN MICRO RNA PREVENTS TRANSLATION OF SPECIFIC GENES…

1. It downregulates methyltransferases, preventing the methylation of tumor-suppressor genes.

2. A decrease in microRNA allows methyltransferase to methylate DNA.

3. Methylation of a tumor- suppressor gene allows for uncontrolled cell growth.

4.4. Cancer cells are able to multiply.

EPIGENETIC THERAPY

Utilizes DNA methylation inhibitors to reverse DNA methylation in tumor- suppressor genes. 1.

2. Uses histone deacetylase (HDAC) inhibitors to prevent the deacetylation of histones bound to tumor suppressor genes.

CONCLUSION