Kuo, Soong-Yu IUFRO Division 5 Conference 5.07B Using Plantation And Small- Diameter Timber In Composites.

Slides:

Advertisements

Similar presentations

3 H-Thymidineincorporation after 24 hrs treatment with severel concentrations of Enniatin Anticancer Activity of the Fusarium Toxin Enniatin Enniatin,

Advertisements

Hui-Jing GU IUFRO IUFRO Division 5 Conference D Forest Products / Energy and chemicals from forest biomass / Chemicals from wood.

2007 IUFRO Division 5 Conference Taipei Physiomechanical properties of wood and wood-based materials and their application Chih-Lung Cho Department.

Shih-Hao Lee IUFRO Division 5 Conference B Mechanical Properties of Wood Poster #392.

Tsang-Chyi SHIAH 1, Han Chien LIN 2 1 Assistant Professor, Department of Forest Products Science, NCYU, 300 University Rd., Chiayi 600, Taiwan. NCYU, 300.

Yiren Wang and Shih-Hao Lee IUFRO Division 5 Conference C Measurement methods including NDT Poster #20.

Composition and bioactivities of the leaf essential oils of Cinnamomum subavenium Miq. from Taiwan Chen-Lung Ho 1,2 Eugene I-Chen Wang 2 Yu-Chang Su 1*

Characterization of a potential new drug in cancer therapy Lab 2 Salah Farag.

Supplementary Fig. 1. (a) Cells were treated with DMSO or with 1 µM of SK-7068 or LAQ824 for 18 hours. They were then fixed with 70% ethanol, stained with.

Figure S1 A MRC5 Control Chk1 TdR(h): γH2AX Cleaved casp3 RPA34

Supplementary Figures: Fig. 1, 2, and 3 show H1299 cells overexpressing Set7/9wt or H297A catalytic mutant and U2-OS, U2-OS Set7/9KD, and U2-OS Set7/9.

POTENTIAL ROLE OF CYTOCHROME P450 3A4 (CYP3A4) IN THE PCB104-MEDIATED BARRIER DYSFUNCTION OF HUMAN MICROVASCULAR ENDOTHELIAL CELLS Yong Woo Lee 1, Sung.

Isosteviol derivatives induced apoptosis in Human lung cancer via targeting MEK/MAPK pathway: An in vitro and in vivo study Ahmed M Malki 1,,PhD Stephen.

Thitiporn Miiksombat 1, Narumol Bhummaphan 1, Teerakul Arpornsuwan 2 1 Student in Medical Technology Program, Faculty of Allied Health Sciences, Thammasat.

Fabrication and characterization of Au-Ag alloy thin films resistance random access memory C. C. Kuo 1 and J. C. Huang 1,* 1 Department of Materials and.

Article Title: AKT-aro and HER2-aro, models for de novo resistance to aromatase inhibitors; molecular characterization and inhibitor response studies Journal.

INCREASED EXPRESSION OF PROTEIN KINASE CK2  SUBUNIT IN HUMAN GASTRIC CARCINOMA Kai-Yuan Lin 1 and Yih-Huei Uen 1,2,3 1 Department of Medical Research,

EVALUATION OF THE ANTIGENOTOXIC POTENTIAL OF GARLIC SULFUR COMPOUNDS IN HEPG2 CELLS C. Belloir, M.H. Siess, C. Daurat and A.M. Le Bon INRA, UMR Toxicologie.

(-)-EPIGALLOCATECHIN-3-GALLATE (EGCG) EHNAHCES OSTEOGENESIS IN A BONE MARROW MESENCHYMAL STEM CELL LINE Chung-Hwan Chen 1,3 ; Mei-Ling Ho 2,3 ; Je-Ken.

Daily used nanoparticles increases the incidence of cancer presented by Hanan Ramadan Hamad Mohamed Zoology Department Faculty of Science Cairo University.

Big Ideas Technological applications that affect biological processes and cellular functions are used in the food, pharmaceutical, and medical industries.

Inhibition of PDCD6 Induces Cell Proliferation and Reduces Apoptosis in Human Epithelial Ovarian Cancer Cells Yan Huang, Xiaohua Wu Department of Gynecology,

VCU/HHMI Summer Scholars program

Goran Gajski Institute for Medical Research and Occupational Health

Antioxidant and Anticancer Properties of Essential Oils of Satureja thymbra and Satureja parnassica Fitsiou Eleni 1, Anestopoulos Ioannis 1, Kourkoutas.

Targeting of reactive oxygen species can be a potential therapeutic strategy for cancer treatment Ying-Ray Lee 1, San-Yuan Chen 2, and Hau-Ren Chen 3 1.

European Journal of Cancer Prevention 2013 R4 김유진 / Prof. 정재헌.

Elucidate the mechanism of anticancer activity of Piper nigrum extract on colon cancer cell line Akila Prashant, MVSST Subbarao, Devananda D, Prashant.

Volume 314, Issue 1, Pages (January 2012)

J. -F. Zhang, P. -Q. Liu, G. -H. Chen, M. -Q. Lu, C. -J. Cai, Y

Supplementary Figure 1 Sensitive Resistant Cell Count Propidium Iodide

Inhibitory effect of (-)-epigallocatechin-3-gallate and bleomycin on human pancreatic cancer MiaPaca-2 cell growth Bimonte S1, Leongito M1, Barbieri.

DNA repair kinetics: the effect of dose and radiation quality

NSAIDs and esophageal cancer

Cell death following DNA damage occurs from cells arrested in G1 or at the G1/S boundary. Cell death following DNA damage occurs from cells arrested in.

Volume 10, Issue 2, Pages (February 2003)

Figure 1. Artemisinin derivatives inhibited EOC growth and arrested the cell cycle at the G2/M phase (A) SKOV3 and primary epithelial ovarian cancer (EOC)

The Chemopreventive Effect of Tanacetum Polycephalum Against LA7-Induced Breast Cancer in Rats and the Apoptotic Effect of a Cytotoxic Sesquiterpene.

NSAIDs and esophageal cancer

Volume 118, Issue 4, Pages (April 2000)

AZD7762 abrogated the G2 checkpoint in a novel hollow fiber assay.

TNF enhances dose‐dependent cell death following doxorubicin‐induced DNA damage with minimal affect on dose‐dependent cell‐cycle arrest. TNF enhances dose‐dependent.

SUPPLEMENTAL TABLE 1. Cell cycle profiles of HeLa cells treated

2. Chemistry Department, Faculty of Science, Cairo University.

3.2.5 Cell Cycle L.O: to state and describe the stages in the cell cycle to apply knowledge of the cell cycle to cancer treatment.

Prediction of novel cell cycle inhibitors based on CODIM1.

Targeting Mitochondrial DNA with a Platinum-Based Anticancer Agent

A novel class I histone deacetylase inhibitor, I-7ab,

Targeting Mitochondrial DNA with a Platinum-Based Anticancer Agent

FLC treatment results in an increase in ploidy in a significant fraction of cells. FLC treatment results in an increase in ploidy in a significant fraction.

Yu-Fen Wang1, Ya-Chi Chan1, Dar-Ren Chen1, 2, Hui-Yi Lin3

Selective Induction of Apoptosis by Histone Deacetylase Inhibitor SAHA in Cutaneous T- Cell Lymphoma Cells: Relevance to Mechanism of Therapeutic Action

Palbociclib increases proteasome activity and the clearance of protein aggregates without significant effects on autophagy Palbociclib increases proteasome.

Valproic acid (VPA) synergises with VAC (mafosfamide, doxorubicin and vindesine) to induce apoptosis in small cell lung carcinoma (SCLC) cell lines. Valproic.

Effect of expression of constitutively active CAMKK2 on AMPK activation and the cell cycle in G361 cells. Effect of expression of constitutively active.

SW fraction induces S phase arrest and apoptosis.

Aplf downregulation does not induce cellular arrest.

Induction of apoptosis by NS398.

Protracted temozolomide (TMZ) treatment leads to acquired TMZ resistance in glioblastoma (GBM) cells in vivo. Protracted temozolomide (TMZ) treatment leads.

Effects of knockdown of ANRIL on NSCLC cell cycle and apoptosis in vitro. Effects of knockdown of ANRIL on NSCLC cell cycle and apoptosis in vitro. A,

Effect of sulindac and atorvastatin on the cell cycle.

LY induces cytotoxicity in hMECs via inhibition of Akt/p70S6K kinase activities. LY induces cytotoxicity in hMECs via inhibition of Akt/p70S6K.

WP1066 inhibits the progression of OCIM2 cells through the cell cycle.

BEZ235 inhibits DNA-PK and enhances G2–M growth arrest after radiation

Increased apoptotic levels after combined treatment with vincristine (VCR) and imatinib. Increased apoptotic levels after combined treatment with vincristine.

Increased apoptotic levels after combined treatment with DXR and imatinib. Increased apoptotic levels after combined treatment with DXR and imatinib. A–D.

Induction of apoptosis by statins in NB4 cells and NB4 variant cell lines. Induction of apoptosis by statins in NB4 cells and NB4 variant cell lines. A,

Sorafenib inhibits cell proliferation and induces apoptosis in HCC cells. Sorafenib inhibits cell proliferation and induces apoptosis in HCC cells. A,

WEE1 inhibition followed by TRAIL treatment results in apoptotic cell death in MB231 cells. WEE1 inhibition followed by TRAIL treatment results in apoptotic.

Presentation transcript:

Kuo, Soong-Yu IUFRO Division 5 Conference 5.07B Using Plantation And Small- Diameter Timber In Composites

Chemical and Cytotoxic Constituents from the Leaves of Cinnamomum subavenium 1 Basic Medical Science Education Center, Fooyin University; Kaohsiung County 83101, Taiwan (R.O.C.); 2 Department of Forest Products Science, National Chia-Yi university, Chiayi, Taiwan (R.O.C.) Soong-Yu Kuo 1, Wen-Li Lo 1, Chung-Yi Chen 1, Jin-Cherng Huang 2 and Hsien-Toung Huang 2

Motive ： The chemical constituents and the biological activity of the leaves of Cinnamomum subavenium Miq. (Lauraceae) have not yet been reported. Purpose ： To elucidate the cytotoxic mechanism of subamolides (1 and 2), the capacity of subamolide D (1) and subamolide E (2) to inhibit cell cycle phase distribution. Method ： Propidium iodide staining and cytometry analysis were used to evaluate the cell cycle progression of the treated SW480 cells. Aim of Work And Methods

Results The marked increase in sub-G1 apoptotic fraction appeared in cells treated with μM subamolide D (1) or subamolide E (2) in a concentration-dependent manner. Additionally, incubation with 25, 50 and 75 μM subamolide D (1) for 24 h resulted in the accumulation of cells in G2/M phase (25.4, 29.8 and 23.0 %, respectively) compared with that in control (15.4 %). When cells were treated with 100 μM subamolide D (1) or subamolide E (2), > 25 %, > 78 %, and > 85 % sub-G1 fraction was noted for treated cells after 8 h, 16 h, and 24 h exposure, respectively.

Conclusion Subamolides D (1) and E (2) caused DNA damage in a dose- and time-dependent manners. These findings suggest that compounds present in C. subavenium Miq. (e.g. subamolides D (1) and E (2)) may have chemopreventive properties. This study is the first to investigate the effect of subamolides D (1) and E (2) on cell cycle phase distribution in human colorectal cancer cells.