اصول EKG
سیستم هدایتی SA node Pacemaker bpm P waves up in I, II, aVF Internodal Pathways AVN; RA contraction Interatrial Pathways LA to depolarize
سیستم هدایتی AV node Delays impulse by.1s PR segment AV junction AV node & His bpm Purkinje/bundles Ventricular depol bpm
سیستم هدایتی
Repolarization Direction is same as depolarization Autonomic Nervous System (ANS) Sympathetic Nervous System (SNS) Parasympathetic Nervous System (PNS)
اصول EKG
اصول EKG: موج P Normal Width <.11 secs Height.5 to 2.5 mm Morphology Flat Biphasic Absent
اصول EKG: موج P Abnormal Inversions Amplitude P-Pulmonale > 2.5 mm Duration P-Mitrale >.1 sec (or 2 ½ boxes) Absence
اصول EKG: موج QRS Impulse travels quicker down the left bundle branch (LBB) than the right bundle branch (RBB). Septum depolarizes L to R resulting in a downward deflection Both ventricles are activated simultaneously. Since the RV is smaller, depolarizes quicker resulting in the downward deflection LV depolarizes resulting in the R wave
اصول EKG: موج QRS
6 ویژگی موج QRS 1.Duration: Secs 2.Amplitude: > 5mm; < 20 mm in limb, < 25 in anterior leads 3.Presence of Q waves < 0.04 msec and < 2 mm normal in I, aVL, aVF, V5 4.Axis 5.Progression: Zone of transition V3-V4 6.Configuration
اصول EKG: موج T و U T waves occur in Same direction as QRS Height: < 5 mm in limb leads, <10 mm in anterior leads U waves After T wave Best seen in lead III Hypothermia/hypokalemia
اصول EKG
لیدهای قفسه سینه
EKG استاندارد EKG استاندارد 12 leads and rhythm strip Limb leads I, II, III, aVR, aVL, aVF Anterior leads V1-V6 Speed = 25 mm/sec Height = 10 mm
متغیرهای نوار قلب Source Age, Sex Body weight Chest configuration Heart position Food intake Temperature, Exercise Smoking, Hyperventilation Position of precordial leads
متغیرهای نوار قلب Ideal time for EKG Bayes’ Theorem Normal hearts have abnl EKGs Normal EKG does not r/o heart disease
رویکرد منظم Rate Rhythm Axis Wave Morphology P, T, and U waves and QRS complex Intervals PR, QRS, QT ST Segment
تعیین ریت قلب On 6 sec strip, count QRS complexes, X 10 QRS on dark line of tracing, count large boxes, ÷ into 300
تعیین ریت قلب
دپلاریزاسیون
دپلاریزاسیون
اصول EKG
موج P در لید II
دپلاریزاسیون بطن ها در لید قفسه سینه
محور قلب Find the quadrant Isolate the isoelectric lead Smallest QRS voltage Isolate the perpendicular lead Isolate the vector Double check your findings
پیدا کردن ربع
پیدا کردن لید ایزوالکتریک
پیدا کردن محور
ریتم / آریتمی Sinus Atrial Junctional Ventricular
آریتمی سینوسی : معیار و انواع P waves upright in I, II, aVF Constant P-P/R-R interval Rate Narrow QRS complex P:QRS ratio 1:1 P-R interval is normal and constant
آریتمی سینوسی : معیار و انواع Normal Sinus Rhythm Sinus Bradycardia Sinus Tachycardia Sinus Arrhythmia
Normal Sinus Rhythm Rate is 60 to 100
Sinus Bradycardia Can be normal variant Can result from medication Look for underlying cause
Sinus Tachycardia May be caused by exercise, fever, hyperthyroidism Look for underlying cause, slow the rate
Sinus Arrhythmia Seen in young patients Secondary to breathing Heart beats faster
آریتمی دهلیزی : معیار و انواع P waves inverted in I, II and aVF Abnormal shape Notched Flattened Biphasic Narrow QRS complex
آریتمی دهلیزی : معیار و انواع Premature Atrial Contractions Ectopic Atrial Rhythm Wandering Atrial Pacemaker Multifocal Atrial Tachycardia Atrial Flutter Atrial Fibrillation
Premature Atrial Contraction QRS complex narrow RR interval shorter than sinus QRS complexes P wave shows different morphology than sinus P wave
Ectopic Atrial Rhythm Narrow QRS complex P wave inverted
Wandering Atrial Pacemaker 3 different P wave morphologies possible with ventricular rate < 100 bpm
Multifocal Atrial Tachycardia 3 different P wave morphologies with ventricular rate> 100 bpm
Atrial Flutter Regular ventricular rate 150 bpm Varying ratios of F waves to QRS complexes, most common is 4:1 Tracing shows 2:1 conduction
Atrial Flutter Tracing shows 6:1 conduction
Atrial Fibrillation Tracing shows irregularly irregular rhythm with no P waves Ventricular rate usually > 100 bpm
Atrial Fibrillation Tracing shows irregularly irregular rhythm with no P waves Ventricular rate is 40
Atrial Tachycardia Tracing shows regular ventricular rate with P waves that are different from sinus P waves Ventricular rate is usually 150 to 250 bpm
P wave May be absent Buried in QRS If present inverted in leads I, II, and aVF Inverted after QRS آریتمی جانکشنال : معیار
PR interval < 0.12 Secs Rate: Varies Narrow QRS complex آریتمی جانکشنال : معیار و انواع
آریتمی جانکشنال : انواع Premature Junctional Contractions Junctional Escape Rhythm Accelerated Junctional Tachycardia Junctional Tachycardia Reentrant Tachycardia AVNRT
Premature Junctional Contractions R-R interval is shorter Beat is early, narrow QRS complex Inverted P wave P wave can be buried in QRS complex
Junctional Escape Rhythm Junctional origin Rate is 40 to 60
Accelerated Junctional Tachycardia Junctional origin Rate is 60 to 100
Junctional Tachycardia Junctional origin Rate is > 100
Secondary to bypass tract within AV node Premature Atrial Contraction (PAC) depolarizes AV Nodal Reentrant Tachycardia (AVNRT) AV Nodal Reentrant Tachycardia (AVNRT)
Rate Summary Sinus Tachycardia BPM Atrial Tachycardia BPM Atrial Flutter BPM Junctional Tachycardia BPM
بلوک های گره AV Delay conduction of impulses from sinus node If AV node does not let impulse through, no QRS complex is seen AV nodal block classes: 1 st, 2 nd, 3 rd degree
1 st Degree AV Block PR interval constant >.2 sec All impulses conducted
2 nd Degree AV Block Type 1 AV node conducted each impulse slower and finally no impulse is conducted Longer PR interval, finally no QRS complex
2 nd Degree AV Block Type 2 Constant PR interval AV node intermittently conducts no impulse
AV node conducts no impulse Atria and ventricles beat at intrinsic rate (80 and 40 respectively) No association between P waves and QRS complexes 3 rd Degree AV Block
Caused by bypass tract AV node is bypassed, delay EKG shows short PR interval <.11 sec Upsloping to QRS complex (delta wave) Wolfe-Parkinson-White (WPW)
Delta wave, short PR interval WPW
آریتمی های بطنی : معیار و انواع Wide QRS complex Rate : variable No P waves Premature Ventricular Contractions Idioventricular Rhythm Accelerated IVR Ventricular Tachycardia Ventricular Fibrillation
Occurs earlier than sinus beat Wide, no P wave Premature Ventricular Contraction
Escape rhythm Rate is 20 to 40 bpm Idioventricular Rhythm
Rate is 40 to 100 bpm Accelerated Idioventricular Rhythm
Rate is > than 100 bpm Ventricular Tachycardia
Torsades de Pointes Occurs secondary to prolonged QT interval
Unorganized activity of ventricle Ventricular Tachycardia/Fibrillation
Ventricular Fibrillation
هایپرتروفی بطن ها و دهلیزها
Differential Diagnosis Hypertension (HTN) Aortic Stenosis (AS) Aortic Insufficiency (AI) Hypertrophic Cardiomyopathy (HCM) Mitral Regurgitation (MR) Coarctation of the Aorta (COA) Physiologic Left Ventricular Hypertrophy (LVH)
False positive Thin chest wall Status post mastectomy Race, Sex, Age Left Bundle Branch Block (LBBB) Acute MI Left Anterior Fascicular Block Incorrect standardization Left Ventricular Hypertrophy (LVH)
Estes Criteria: Diagnosis of LVH
Right Ventricular Hypertrophy: Causes Chronic Obstructive Pulmonary Disease Pulmonary HTN Primary Pulmonary Embolus Mitral Stenosis Mitral Regurgitation Chronic LV failure
Right Ventricular Hypertrophy: Causes Tricuspid Regurgitation Atrial Septal Defect Pulmonary Stenosis Tetralogy of Fallot Ventricular Septal Defect
Right Ventricular Hypertrophy
Reversal of precordial pattern R waves prominent in V1 and V2 S waves smaller in V1 and V2 S waves become prominent in V5 and V6
Strain
Mitral Stenosis Mitral Regurgitation Left ventricular hypertrophy Hypertension Aortic Stenosis Aortic Insufficiency Hypertrophic Cardiomyopathy بزرگی دهلیز چپ : علل
بزرگی دهلیز چپ : معیار P wave Notch in P wave Any lead Peaks > 0.04 secs V1 Terminal portion of P wave > 1mm deep and > 0.04 sec wide
Lead II
موج P: بزرگی دهلیز چپ
بزرگی دهلیز چپ
CHD Tricuspid Stenosis Pulmonary Stenosis COPD Pulmonary HTN Pulmonary Embolus Mitral Regurgitation Mitral Stenosis بزرگی دهلیز راست : علل
Tall, peaked P wave > 2.5 mm in any lead Most prominent P waves in leads I, II and aVF بزرگی دهلیز چپ : معیار
بزرگی دهلیز راست
Bundle Branch Blocks بلوک شاخه ای دسته ای
Normal variant Idiopathic degeneration of the conduction system Cardiomyopathy Ischemic heart disease Aortic Stenosis Hyperkalemia Left Ventricular Hypertrophy بلوک شاخه ای دسته ای چپ : علل
بلوک شاخه ای دسته ای چپ : معیار Bizarre QRS Morphology High voltage S wave in V1, V2 & V3 Tall R wave in leads I, aVL and V5-6 Often LAD QRS Interval ST depression in leads I, aVL, & V5-V6 T wave inversion in I, aVL, & V5-V6
Left Bundle Branch Block
بلوک شاخه ای دسته ای راست : علل Idiopathic degeneration of the conduction system Ischemic heart disease Cardiomyopathy Massive Pulmonary Embolus Ventricular Hypertrophy Normal Variant
بلوک شاخه ای دسته ای راست : معیار QRS morphology Wide S wave in leads I and V4-V6 RSR’ pattern in leads V1, V2 and V3 QRS duration ST depression in leads V1 and V2 T wave inversion in leads V1 and V2
بلوک شاخه ای دسته ای راست
ایسکمی و انفارکتوس
Normal Complexes and Segments
J Point
اندازه گیری
ایسکمی T wave inversion, ST segment depression Acute injury: ST segment elevation Dead tissue: Q wave
EKG Changes: Ischemia → Acute Injury→ Infarction
لیدهای قفسه سینه
محل لیدهای قدامی V1 Right 4 th ICS V2 Left 4 th ICS V3 Left Sternal border Between V2 and V4 V4 Left MCL 5 th ICS V5 Anterior axillary line 5 th ICS V6 Mid axillary line 5 th ICS
خونرسانی به قلب Left Anterior Descending (LAD) artery Bulk of LV Anterior wall Apex Part of lateral wall Part of septum
خونرسانی به قلب Right Coronary Artery (RCA) Right Ventricle Sinus Node 60% of the time Right Atrium Posterior Descending Artery (PAD) 90% of the time
خونرسانی به قلب Left Circumflex (LCFLX) artery Lateral Wall & Posterior Wall of LV & LA SA node 40% of the time PDA 10% of the time Posterior Descending Artery (PAD) Off RCA 90%/LCFLX 40% AVN, prox BB, IW/PW, basal septum
View of the Leads II, III, aVF Inferior Wall//RV RCA distribution I, aVL Lateral Wall LCFLX/distal LAD distribution aVR R side of heart V1 & V2 Intraventricular septum Proximal LAD V3 & V4 Anterior wall Mid LAD V5 & V6 Lateral wall Distal LAD
Standard EKG
Anterior MI
Anterior-septal MI
Inferior AMI
Right Sided Leads
Right Ventricular AMI
Lateral MI
Posterior Leads
Posterior AMI
Inferior-RV-Posterior AMI
تغییرات موج ST-T
Strain in Hypertrophy
Strain in LVH
Strain in RVH
Strain vs. Infarction
پریکاردیت
تغییرات دیوگسین
موج T
خسته نباشید