The INTRODUCTION. Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful.

Slides:



Advertisements
Similar presentations
Successful Scientific Writing
Advertisements

Results Introduction Tobacco use is the leading preventable cause of death in Wisconsin and the United States. Given the risk of smoking initiation during.
SUNBED USER’S MOTIVATIONS, KNOWLEDGE AND HABITS IN BUDAPEST, HUNGARY József Bakos, Judit Szabó, György Thuróczy Department of Non-ionizing Radiations,
Carcinogen Classification Criteria Patricia Richter Ph.D., DABT Tobacco Products Scientific Advisory Committee June 8, 2010.
The Health Implications of Raising the Minimum Age for Purchasing Tobacco Products Eric N. Lindblom Director, Office of Policy Center for Tobacco Products,
Case-Control Studies (Retrospective Studies). What is a cohort?
Arizona Adult Tobacco Survey Response to Health Professional Query Behavior Richard S. Porter, MS Bob Leischow, MPH Arizona Department of Health Services.
《 Promotion of Capability and Effectiveness for Tobacco Control Program among Rural Residents* 》 --Report On The Baseline Survey (Tobacco use status among.
Second-hand Smoke Study Sparks Controversy By Mike Wendling CNSNews.com London Bureau Chief May 16, 2003
Texas Tobacco Laws. Senate Bill Texas Legislature Tobacco-Free Schools Sec TOBACCO ON SCHOOL PROPERTY The Board of Trustees of a school.
Information on tobacco packages could encourage and help tobacco users to stop using tobacco or find least possible harmful ways of use for those unable.
Bullet points with meaning: Writing for a Deputy Minister Nancy Edwards UWISON June, 2014.
The success of anti-smoking campaigns Has the worldwide anti-smoking campaign caused a change in attitude of smokers?
What is Public Health? Allyson Hall, PhD
Lesson 2 Why is radon a health problem?. Slide 2-1 Radon and lung cancer Radon is largest source of radiation exposure to general public Radon is leading.
1 Minority SA/HIV Initiative MAI Training SPF Step 3 – Planning Presented By: Tracy Johnson, CSAP’s Central CAPT Janer Hernandez, CSAP’s Northeast CAPT.
COHORT STUDY DR. A.A.TRIVEDI (M.D., D.I.H.) ASSISTANT PROFESSOR
© Copyright 2011, Pearson Education, Inc. All rights reserved. Chapter 10 Nicotine and Tobacco Use.
Biology in Focus, HSC Course Glenda Childrawi, Margaret Robson and Stephanie Hollis A Search For Better Health Topic 11: Epidemiology.
Writing a Research Proposal
Higher Education Meets Public Health: Successful Collaborative Efforts to Change Tobacco Policies on Campus Maine Tobacco Free College Network Emily Rines,
International Telecommunication Union Committed to connecting the world 4 th ITU Green Standards Week Maria Feychting ICNIRP Vice Chair Professor of Epidemiology.
Outcomes of Public Health
Introduction to US Health Care
An Educational Perspective Based on Information Contained In The Indiana Prevention Resource Center Factline on Tobacco.
Dr. Abdulaziz BinSaeed & Dr. Hayfaa A. Wahabi Department of Family & Community medicine  Case-Control Studies.
Washington Communities for Tobacco Prevention Spokane Regional Health District Board of Health September 27, 2012.
Literature Review and Parts of Proposal
Conveyancing What role can it play in identifying and remediating homes above 200 Bq/m 3 ? Stephanie Long Senior Scientist, Radon Advice.
CDC’s Best Practices for Comprehensive Tobacco Control Programs Jerelyn Jordan Centers for Disease Control and Prevention Office on Smoking and Health.
Kansas Hospitals Are Tobacco-Free Kansas Public Health Association, Inc Fall Conference Cindy Samuelson Kansas Hospital Association.
 Smoking is the primary cause of avoidable death  Thousand of teens try cigarettes daily.  Smoking contributes to an astonishing number of diseases.
 2011 Johns Hopkins Bloomberg School of Public Health Jonathan M. Samet, MD, MS Director, USC Institute for Global Health Professor and Flora L. Thornton.
Self-reported cognitive and emotional effects and lifestyle changes shortly after preventive cardiovascular consultations in general practice Dea Kehler.
Writing research proposal/synopsis
Protecting Children through Los Angeles’ Tobacco Enforcement Program Nora Manzanilla Office of the Los Angeles City Attorney ROCKARD J. DELGADILLO TOBACCO.
Tobacco Sales To Youth Presented to { } by { } Date.
Take a Stand Murphree, Bella Period 1B Abrams March 29, 2012.
Tobacco Use In Kansas Healthy Kansans 2010 Steering Committee Meeting May 12, 2005.
Environmental Tobacco Smoke (ETS) and the Risk of Head and Neck Cancer: INHANCE Consortium Yuan-Chin Amy Lee Gene-Environmental Epidemiology Group International.
Chronic Disease Risk Factors. Risk Factors What are the risk factors for chronic disease? #1?? #2?? Others….
Critical Appraisal of the Scientific Literature
Evaluating Local Tobacco Control Organizations. David Ahrens, Research Program Manager Research conducted by: Barbara.
Why Indicators Matter Using Outcome Indicators to Plan and Evaluate Comprehensive Tobacco Control Programs Todd Rogers, PhD California Tobacco Control.
Section 24.3 Public Health Slide 1 of 18 Objectives Summarize the main goal of public health programs today. Describe how the United States’ public health.
Texas Tobacco Laws – for Schools AISD Policy and Student Code of Conduct Compliance, Education & Enforcement 11/15/
Saftey Agencies in Healthcare Practice OSHA CDCP CLIA FDA ISO WHO NIH USDHHS.
The Role of Harm Reduction in Tobacco control – An Update of Pros and Cons Lars M. Ramström Institute for Tobacco Studies Stockholm, Sweden 53rd International.
Meeting of the UNEP Advisory group on Endocrine Disrupting Chemical,Geneva, September 2015 Caroline N. Wamai Senior Principal Chemist Ministry of.
What is a Public Health Assessment? “The evaluation of data and information on the release of harmful substances into the environment in order to assess.
 2007 Johns Hopkins Bloomberg School of Public Health Section B ASSIST Evaluation.
The Impact of Epidemiology in Public Health Robert Hirokawa, DrPH Epidemiologist, Science and Research Group HHI / TSP, Hawaii Department of Health.
Smoking and the Movies Jonathan M. Samet, MD, MS MPAA, Hollywood, California February 23, 2007 Department of Epidemiology.
U.S. DEPARTMENT OF HEALTH AND HUMAN SERVICES Centers for Disease Control and Prevention National Center for Health Statistics Occupational exposure to.
What is Public Health? Allyson Hall, PhD Department of Health Services Research, Management, and Policy College of Public Health and Health Professions.
Behavioral Science HEALTH BELIEF MODEL (HBM) Dr. G.U Ahsan, Ph.D.
National Program for Tobacco Control in the Republic of Uzbekistan for Mr.Mamutov R.Sh. Director, National Centre for Tobacco Control.
Introduction What do we mean by Public Health? How has the Approach to Public Health Changed over Time? What is Meant by Population Health? What are the.
LITERATURE REVIEW ARCHELLE JANE C. CALLEJO, PTRP,MSPH.
Comprehensive Tobacco Action Group Summary December 16, 2005.
TM Substance Use Transitions from Initial Use to Regular Use to Discontinuance Ralph S. Caraballo, Ph.D., MPH Office on Smoking and Health, CDC, Atlanta.
THE OCCUPATIONAL SAFETY AND HEALTH ACT Brendon Clair.
Chapter 7 Smoking and occupational exposures as risk factors for asthma.
1 Access to and use of aids to smoking cessation in the UK Robert West University College London Austin, Texas February 2007.
1 Impact of the ‘smoking ban’ on smoking prevalence in England Cancer Research UK Health Behaviour Research Centre Department of Epidemiology and Public.
Utilizing Research: Putting Research Evidence Into Nursing Practice Prepare by /Dr. AmiraYahia.
Case control & cohort studies
IAEA International Atomic Energy Agency IAEA Safety Standards and Public Exposure to Radon Trevor Boal Radiation Protection Unit - NSRW.
EU-OSHA Workshop: Workplace Risks to Reproductive Function
Defining An Effectiveness Standard
Presentation transcript:

The INTRODUCTION

Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful Components of the Introduction

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful Components of the Introduction ● Identify a gap in knowledge or knowhow (“Study problem”).

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful Components of the Introduction ● Identify a gap in knowledge or knowhow (“Study problem”). - Provide key background (scope/nature/magnitude of the gap).

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful Components of the Introduction ● Identify a gap in knowledge or knowhow (“Study problem”). - Provide key background (scope/nature/magnitude of the gap). - Be clear that filling this gap will be useful.

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful Components of the Introduction ● Identify a gap in knowledge or knowhow (“Study problem”). - Provide key background (scope/nature/magnitude of the gap). - Be clear that filling this gap will be useful. - Describe the relevant limitations of previous studies.

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful Components of the Introduction ● Identify a gap in knowledge or knowhow (“Study problem”). - Provide key background (scope/nature/magnitude of the gap). - Be clear that filling this gap will be useful. - Describe the relevant limitations of previous studies. ● Present your approach to filling the gap (“Study purpose”).

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful Components of the Introduction ● Identify a gap in knowledge or knowhow (“Study problem”). - Provide key background (scope/nature/magnitude of the gap). - Be clear that filling this gap will be useful. - Describe the relevant limitations of previous studies. ● Present your approach to filling the gap (“Study purpose”). - Be clear that your approach is new.

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful Components of the Introduction ● Identify a gap in knowledge or knowhow (“Study problem”). - Provide key background (scope/nature/magnitude of the gap). - Be clear that filling this gap will be useful. - Describe the relevant limitations of previous studies. ● Present your approach to filling the gap (“Study purpose”). - Be clear that your approach is new. - Emphasize that your approach addresses the limitations of previous studies in a logical/compelling way.

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful Components of the Introduction ● Identify a gap in knowledge or knowhow (“Study problem”). - Provide key background (scope/nature/magnitude of the gap). - Be clear that filling this gap will be useful. - Describe the relevant limitations of previous studies. ● Present your approach to filling the gap (“Study purpose”). - Be clear that your approach is new. - Emphasize that your approach addresses the limitations of previous studies in a logical/compelling way.

The INTRODUCTION Purpose: to convince the reader that your study will yield knowledge or knowhow that is new and useful Components of the Introduction ● Identify a gap in knowledge or knowhow (“Study problem”). - Provide key background (scope/nature/magnitude of the gap). - Be clear that filling this gap will be useful. - Describe the relevant limitations of previous studies. ● Present your approach to filling the gap (“Study purpose”). - Be clear that your approach is new. - Emphasize that your approach addresses the limitations of previous studies in a logical/compelling way. (often requires just three paragraphs)

Elements of the Introduction GAP G B – Key BACKGROUND to the gap. G U – Why filling the gap will be USEFUL. G L – Key LIMITATION(S) of previous studies responsible for the gap. APPROACH A B – BACKGROUND to the approach (not always necessary) A N – What is NEW about the approach taken in the current study. A L – How does this new approach address the LIMITATIONS of previous studies.

[G U : If radiofrequency exposure from mobile phone use increases the risk of cancer, acoustic neuroma would be of potential concern.] [G B : The exposure from mobile phones is concentrated in the head close to the handset; exposure is relatively high only for the glial and meningeal tissue closest to the surface of the head, the parotid gland, and the vestibular portion of the eighth cranial nerve where acoustic neuromas arise.] [Gap: Six studies have investigated the association between mobile phone use and acoustic neuroma, with inconsistent results.] [G L : All available studies are limited by a small number of exposed cases and a short follow-up since the time hand-held mobile phones first became available.]

[A B : In Sweden, mobile phone use became common in the general population relatively early; handheld mobile phones were introduced at the end of the 1980s with an exponential increase of users during the 1990s. Slightly less than 6% of the population used mobile phones in 1990, 23% in 1995, and over 80% today.] Therefore, [Approach: a study based on the Swedish population (is this new?)] [A L : will have a large proportion of long-term users, which is crucial for the possibility of detecting any increased risk of tumors related to long-term mobile phone use.]

[G U : If radiofrequency exposure from mobile phone use increases the risk of cancer, acoustic neuroma would be of potential concern.] [G B : The exposure from mobile phones is concentrated in the head close to the handset; exposure is relatively high only for the glial and meningeal tissue closest to the surface of the head, the parotid gland, and the vestibular portion of the eighth cranial nerve where acoustic neuromas arise.] [Gap: Six studies have investigated the association between mobile phone use and acoustic neuroma, with inconsistent results.] [G L : All available studies are limited by a small number of exposed cases and a short follow-up since the time hand-held mobile phones first became available.] [A B : In Sweden, mobile phone use became common in the general population relatively early; handheld mobile phones were introduced at the end of the 1980s with an exponential increase of users during the 1990s. Slightly less than 6% of the population used mobile phones in 1990, 23% in 1995, and over 80% today.] Therefore, [Approach: a study based on the Swedish population] [A L : will have a large proportion of long-term users, which is crucial for the possibility of detecting any increased risk of tumors related to long-term mobile phone use.]

Lung Cancer Risk and Workplace Exposure to Environmental Tobacco Smoke Group Exercise What is the Gap (G)? Does the study approach address the limitations of previous studies in a logical/compelling way? Why will filling this gap be useful (Gu)?

Exposure to environmental tobacco smoke (ETS) has been recognized as a cause of human cancer by the US Surgeon General, 1 the National Institute for Occupational Safety and Health, 2 the US Environmental Protection Agency, 3 the California Environmental Protection Agency, 4 the National Health and Medical Research Council of Australia, 5 the Great Britain Department of Health, 6 and most recently, the International Agency for Research on Cancer. 7 Evidence for this association has come primarily from studies of nonsmokers who are married to a smoker, and meta-analyses of these studies have demonstrated strong and consistent evidence for an association. 3,8,9 Demonstrating an association between workplace ETS exposure and lung cancer risk has been more difficult. Early meta-analyses failed to demonstrate an association between workplace ETS exposure and lung cancer risk among nonsmokers, 10–14 but a statistically significant association has been reported in the 3 most recently published meta-analyses. 15–17 We sought to extend the previous meta-analyses by including additional studies and by conducting analyses stratified by level of exposure, which was not performed in the previous meta-analyses. What is the Gap (G)?

Exposure to environmental tobacco smoke (ETS) has been recognized as a cause of human cancer by the US Surgeon General, 1 the National Institute for Occupational Safety and Health, 2 the US Environmental Protection Agency, 3 the California Environmental Protection Agency, 4 the National Health and Medical Research Council of Australia, 5 the Great Britain Department of Health, 6 and most recently, the International Agency for Research on Cancer. 7 Evidence for this association has come primarily from studies of nonsmokers who are married to a smoker, and meta-analyses of these studies have demonstrated strong and consistent evidence for an association. 3,8,9 Demonstrating an association between workplace ETS exposure and lung cancer risk has been more difficult. Early meta-analyses failed to demonstrate an association between workplace ETS exposure and lung cancer risk among nonsmokers, 10–14 but a statistically significant association has been reported in the 3 most recently published meta-analyses. 15–17 We sought to extend the previous meta-analyses by including additional studies and by conducting analyses stratified by level of exposure, which was not performed in the previous meta-analyses. What is the Gap (G)?

Exposure to environmental tobacco smoke (ETS) has been recognized as a cause of human cancer by the US Surgeon General, 1 the National Institute for Occupational Safety and Health, 2 the US Environmental Protection Agency, 3 the California Environmental Protection Agency, 4 the National Health and Medical Research Council of Australia, 5 the Great Britain Department of Health, 6 and most recently, the International Agency for Research on Cancer. 7 Evidence for this association has come primarily from studies of nonsmokers who are married to a smoker, and meta-analyses of these studies have demonstrated strong and consistent evidence for an association. 3,8,9 Demonstrating an association between workplace ETS exposure and lung cancer risk has been more difficult. Early meta-analyses failed to demonstrate an association between workplace ETS exposure and lung cancer risk among nonsmokers, 10–14 but a statistically significant association has been reported in the 3 most recently published meta-analyses. 15–17 We sought to extend the previous meta-analyses by including additional studies and by conducting analyses stratified by level of exposure, which was not performed in the previous meta-analyses. Does the study approach address the limitations of previous studies in a logical/compelling way?

Exposure to environmental tobacco smoke (ETS) has been recognized as a cause of human cancer by the US Surgeon General, 1 the National Institute for Occupational Safety and Health, 2 the US Environmental Protection Agency, 3 the California Environmental Protection Agency, 4 the National Health and Medical Research Council of Australia, 5 the Great Britain Department of Health, 6 and most recently, the International Agency for Research on Cancer. 7 Evidence for this association has come primarily from studies of nonsmokers who are married to a smoker, and meta-analyses of these studies have demonstrated strong and consistent evidence for an association. 3,8,9 Demonstrating an association between workplace ETS exposure and lung cancer risk has been more difficult. Early meta-analyses failed to demonstrate an association between workplace ETS exposure and lung cancer risk among nonsmokers, 10–14 but a statistically significant association has been reported in the 3 most recently published meta-analyses. 15–17 We sought to extend the previous meta-analyses by including additional studies and by conducting analyses stratified by level of exposure, which was not performed in the previous meta-analyses. Does the study approach address the limitations of previous studies in a logical/compelling way?

Exposure to environmental tobacco smoke (ETS) has been recognized as a cause of human cancer by the US Surgeon General, 1 the National Institute for Occupational Safety and Health, 2 the US Environmental Protection Agency, 3 the California Environmental Protection Agency, 4 the National Health and Medical Research Council of Australia, 5 the Great Britain Department of Health, 6 and most recently, the International Agency for Research on Cancer. 7 Evidence for this association has come primarily from studies of nonsmokers who are married to a smoker, and meta-analyses of these studies have demonstrated strong and consistent evidence for an association. 3,8,9 Demonstrating an association between workplace ETS exposure and lung cancer risk has been more difficult. Early meta-analyses failed to demonstrate an association between workplace ETS exposure and lung cancer risk among nonsmokers, 10–14 but a statistically significant association has been reported in the 3 most recently published meta-analyses. 15–17 We sought to extend the previous meta-analyses by including additional studies and by conducting analyses stratified by level of exposure, which was not performed in the previous meta-analyses. Why will filling this gap be useful?

Reducing the Illegal Sale of Cigarettes to Minors Group Exercise Read Abstract and Introduction What is the gap?

What gap are the authors trying to fill? Knowledge or Knowhow?

[Gap: Although most states, including California, have laws regulating the access of minors to tobacco, they are rarely enforced. By all accounts, tobacco is readily accessible to minors.] [G B : In field trials, minors have successfully purchased tobacco from stores and vending machines 70% to 100% of the time. National survey data indicate that 57% of high school seniors who report daily smoking began by the age of 14 years.] [G U : Stopping the sale of tobacco to minors through enforcement of existing laws is, therefore, a critical step in any effort to prevent tobacco use.] What about the limitations of previous studies (G L )?

[Gap: Although most states, including California, have laws regulating the access of minors to tobacco, they are rarely enforced. By all accounts, tobacco is readily accessible to minors.] [G B : In field trials, minors have successfully purchased tobacco from stores and vending machines 70% to 100% of the time. National survey data indicate that 57% of high school seniors who report daily smoking began by the age of 14 years.] [G U : Stopping the sale of tobacco to minors through enforcement of existing laws is, therefore, a critical step in any effort to prevent tobacco use.] [A B : We are aware of only two published studies that measured the effect of efforts to enforce existing laws against the sale of cigarettes to minors. In Decatur, IL, a voluntary merchant education program reduced the ability of minors to purchase cigarettes from stores by 18%. In Avon, England, a voluntary merchant education program combined with a media campaign reduced the ability of minors to purchase tobacco from 91% of tobacconists to 44%. ] [Approach: The purpose of the current study was to determine the effectiveness of a program to reduce illegal sales of cigarettes to minors that includes [A N(L) : three components]: voluntary merchant education, a media campaign, and grassroots community organization.]

Original ~ 500 words Revision ~ 200 words

Passive voice exercise On the basis of evidence from clinical trials, the Advisory Committee on Immunization Practices (ACIP) recommended in February 2000 that a 4- dose PCV7 regimen should be given to all children aged <2 years. Try to convert this to active voice without changing the meaning.

Passive voice exercise On the basis of evidence from clinical trials, the Advisory Committee on Immunization Practices (ACIP) recommended in February 2000 that a 4- dose PCV7 regimen should be given to all children aged <2 years.

Passive voice exercise On the basis of evidence from clinical trials, the Advisory Committee on Immunization Practices (ACIP) recommended in February 2000 that a 4- dose PCV7 regimen should be given to all children aged <2 years. On the basis of evidence from clinical trials, the Advisory Committee on Immunization Practices (ACIP) recommended in February 2000 that all children aged <2 years should receive a 4-dose PCV7 regimen should be given to.

Passive voice exercise On the basis of evidence from clinical trials, the Advisory Committee on Immunization Practices (ACIP) recommended in February 2000 that a 4- dose PCV7 regimen should be given to all children aged <2 years. On the basis of evidence from clinical trials, the Advisory Committee on Immunization Practices (ACIP) recommended in February 2000 that all children aged <2 years should receive a 4-dose PCV7 regimen should be given to. Can you find an extraneous word in this revision?

On the basis of evidence from clinical trials, the Advisory Committee on Immunization Practices (ACIP) recommended in February 2000 that a 4- dose PCV7 regimen should be given to all children aged <2 years. On the basis of evidence from clinical trials, the Advisory Committee on Immunization Practices (ACIP) recommended in February 2000 that all children aged <2 years should receive a 4-dose PCV7 regimen should be given to.