II) Acute GN Definition (Hricik et al, 1998) Syndrome characterized by the abrupt onset of macroscopic hematuria; oliguria; acute renal failure; manifested.

Slides:



Advertisements
Similar presentations
EVALUATION OF ASYMPTOMATIC PROTEINURIA IN CHILDREN
Advertisements

PROTEINURIA AND HEMATURIA ASHIK HAYAT M.D.. Proteinuria and Nephrotic Syndrome Occurrence of proteinuria in a single urine is relatively common. Will.
Glomerulonephritis in children
Nephrotic Syndrome (NS)
Nephrotic/nephritic syndrome
Glomerular Diseases In Pediatrics.
Prepared by D. Chaplin Chronic Renal Failure. Prepared by D. Chaplin Chronic Renal Failure Progressive, irreversible damage to the nephrons and glomeruli.
Protein-, Mineral- & Fluid-Modified Diets for Kidney Diseases
Dr. Paula Blanco & Dr. Peter Magner
Proteinuria 24 th June 2011 Rachel Lennon. The Spectrum of Glomerular Disease Minimal change Proteinuria FSGS IgA nephropathy Membranous Diabetic nephropathy.
Pathology of the Kidney and Its Collecting System
Glomerular Diseases Dr. Atapour Differential diagnosis and evaluation of glomerular disease.
NEPHROTIC SYNDROME. Common Causes of Benign Proteinuria Dehydration Emotional stress Fever Heat injury Inflammatory process Intense activity.
Jack DeRuiter, PhD Department of Pharmacal Sciences April, 2000
Pathophysiology of Disease: Chapter 16 ( ) RENAL DISEASE: OVERVIEW AND ACUTE RENAL FAILURE Pathophysiology of Disease: Chapter 16 ( ) Jack.
This lecture was conducted during the Nephrology Unit Grand Ground by a Sub-intern under Nephrology Division, Department of Medicine in King Saud University.
This lecture was conducted during the Nephrology Unit Grand Ground by Medical Student rotated under Nephrology Division under the supervision of Prof.
This lecture was conducted during the Nephrology Unit Grand Ground by Medical Student under Nephrology Division under the supervision and administration.
Zehra Eren M.D. Nephrology Department. The Kidney in:  Congestive heart failure  Liver disease  Diabetes Mellitus  Systemic Vasculitis  İnfections.
Renal Pathology. Introduction: 150gm: each kidney 1700 liters of blood filtered  180 L of G. filtrate  1.5 L of urine / day. Kidney is a retro-peritoneal.
Continuity Clinic Proteinuria. Continuity Clinic.
Proteinuria DR Badi AlEnazi Consultant pediatric endocrinology and diabetologest.
Nephrotic syndrome. Nephrotic syndrome characterized by four components both clinical & biochemical *Generalized Oedema *Massive Proteinuria: above 1g/m.
Acute Renal Failure Hai Ho, M.D..
Ricki Otten MT(ASCP)SC
GLOMERULONEPHRITIS IN TYPE 2 DIABETES MELLITUS MOHAN DAS M.K, JACOB GEORGE,VIMALA.A. DEPARTMENT OF NEPHROLOGY, MEDICAL COLLEGE, THIRUVANANTHAPURAM 1.
Renal Pathology. Introduction Glomerular diseases Tubular and interstitial diseases Diseases involving blood vessels Cystic diseases Tumors Renal Pathology.
Nephrology Diseases & Chemotherapy. Idiopathic Nephrotic Syndrome (NS) Caused by renal diseases that increase the permeability across the glomerular filtration.
Nephrotic Syndrome (NS)
Glomerulonephritis Dr. Abdelaty Shawky Dr. Gehan mohamed.
2006 Renal Week Lecture 3 Hematuria and Glomerulonephritis Debbie Gipson UNC Kidney Cener website: password:
Nephrotic Syndrome Etiology Idiopathic nephrotic syndrome (90%)
URINARY SYSTEM PATHOLOGY Glomerulonephritis DR: NAWAL ALMOHAMMADI CLINICAL PHARMA STUDENTS 2015.
Glomerulonephritis Brian S. Pavey, DO, MS. Presentation Sudden onset – Hematuria – Hypertension – Edema – Acute kidney injury.
Pathology of the Urinary System Lecture-2. Recap.. Anatomy and physiology of kidney Structure of nephron and components Functional aspects Clinical aspects.
Associate professor of Internal Medicine
MLAB 2401: Clinical Chemistry Keri Brophy-Martinez
Lecture – 3 – Major renal syndromes Dr.Hazem.K.Al-Khafaji MBCHB.D.M.FICMS.
And Review of Acute nephritis Syndromes. Karyomegalic Tubulointerstitial Nephritis  Symptoms: Recurrent Pneumonias Renal failure leading invariably to.
POST INFECTIOUS GLOMERULONEPHRITIS (PIGN) Dr. Nariman Fahmi Ahmed Azat.
Hazem.K.Al-Khafaji FICMS Department of internal medicine College of medicine Al-Qadissyia University.
GLOMERULONEPHRITIS DR. HANY ELSAYED LECTURER OF PEDIATRICS.
Glomerular diseases typical case reports morphology Doc. MUDr. Zdeňka Vernerová, CSc., MUDr. Martin Havrda.
G LOMERULONEPHRITIS AND H EMATURIA. G LOMERULONEPHRITIS Glomerular injury may result from immunologic injury (poscstreptococcal acute glomerulonephritis,
Diseases of kidney Fatima Obeidat, MD Histopathologist/Neuropathologist.
Glomerulonephritis By Dr. Abdelaty Shawky Associate professor of pathology.
Gilead -Topics in Human Pathophysiology Fall 2009 Drug Safety and Public Health.
Nephrotic Syndrome mahmoud abu ajwa Prepared by : mahmoud abu ajwa 2016 Diagnostic clinical chemistry Supervisor : Mr.Naser Abu Sha’ban.
GLOMERULONEPHRITIS BY DR. Hayam Hebah Associate professor of Internal Medicine AL Maarefa college.
RENAL PATHOLOGY FOR REHABILITATION STUDENTS
Renal Pathology Kristine Krafts, M.D..
RENAL PATHOLOGY FOR DENTAL
ACUTE GLOMERULONEPHRITIS
INTERVENTIONS FOR CLIENTS WITH RENAL DISORDERS
The Nephrotic Syndrome
presentation: nephrotic syndrome
GLOMERULONEPHRITIS.
ACUTE & CHRONIC GLOMERULONEPHRITIS
Important notes: Dear students…
Jack DeRuiter, PhD Department of Pharmacal Sciences April, 2000
Acute poststreptococcal GN
Acute and Chronic Renal Failure
Overview of glomerular diseases
IgA Nephropathy Southwest Nephrology Symposium February 24th 2018.
Renal Pathology Kristine Krafts, M.D..
Nephrotic Syndrome.
CLINICAL PRESENTATION OF GN
Acute Glomerulonephritis
Kidney.
Urinalysis CPC (Renal block) Thursday Sep 5, 10-12am
Presentation transcript:

II) Acute GN Definition (Hricik et al, 1998) Syndrome characterized by the abrupt onset of macroscopic hematuria; oliguria; acute renal failure; manifested by a sudden decrease in GFR; and fluid retention, manifested by edema and hypertension. Definition (Hricik et al, 1998) Syndrome characterized by the abrupt onset of macroscopic hematuria; oliguria; acute renal failure; manifested by a sudden decrease in GFR; and fluid retention, manifested by edema and hypertension.

Acute GN Classically post-streptococcal although other infectious etiologies possible Acute pharyngitis followed d later by acute nephritis (sodium and water retention, hypertension, smoky coloured urine, fall in GFR Usually associated with low complement Treatment is supportive Prognosis generally good with spontaneous resolution Classically post-streptococcal although other infectious etiologies possible Acute pharyngitis followed d later by acute nephritis (sodium and water retention, hypertension, smoky coloured urine, fall in GFR Usually associated with low complement Treatment is supportive Prognosis generally good with spontaneous resolution

III) Nephrotic Syndrome Case 79 year old man, history of ureteric stenosis requiring stenting 8 years ago New onset edema, HTN 24 hour urine – 13 g protein, cholesterol 11, creatinine 135 Diagnosis?Case 79 year old man, history of ureteric stenosis requiring stenting 8 years ago New onset edema, HTN 24 hour urine – 13 g protein, cholesterol 11, creatinine 135 Diagnosis?

ProteinuriaProteinuria Mechanisms Damage to glomerular BM/visceral epithelium Impaired reabsorption by tubular cells Secretion of protein from tubular cells Filtration of an abnormal protein that is smallMechanisms Damage to glomerular BM/visceral epithelium Impaired reabsorption by tubular cells Secretion of protein from tubular cells Filtration of an abnormal protein that is small

Nephrotic Syndrome

Definition (Screiner, 1971) proteinuria edemahypoalbuminemia hyperlipidemiaA clinical entity having multiple causes and characterized by increased glomerular permeability manifested by massive proteinuria and lipiduria. There is a variable tendency towards edema, hypoalbuminemia and hyperlipidemia. Protein excretion rates are usually in excess of 3.5 g/day/1.73m 2 body surface in the absence of a depressed GFR. Definition (Screiner, 1971) proteinuria edemahypoalbuminemia hyperlipidemiaA clinical entity having multiple causes and characterized by increased glomerular permeability manifested by massive proteinuria and lipiduria. There is a variable tendency towards edema, hypoalbuminemia and hyperlipidemia. Protein excretion rates are usually in excess of 3.5 g/day/1.73m 2 body surface in the absence of a depressed GFR.

Nephrotic Syndrome Differential Diagnosis Minimal change FSGS Membranous MPGN IgA nodular – amyloidosis, DM, fibrillary Differential Diagnosis Minimal change FSGS Membranous MPGN IgA nodular – amyloidosis, DM, fibrillary

Nephrotic Syndrome

Minimal Change Disease

FSGSFSGS

Membranous Nephropathy