Project: Ghana Emergency Medicine Collaborative Document Title: Systematic Evaluation to Non-Traumatic Head CTs Author(s) Rashmi U. Kothari, MD (KCMS/MSU), 2012 License: Unless otherwise noted, this material is made available under the terms of the Creative Commons Attribution Share Alike-3.0 License: We have reviewed this material in accordance with U.S. Copyright Law and have tried to maximize your ability to use, share, and adapt it. These lectures have been modified in the process of making a publicly shareable version. The citation key on the following slide provides information about how you may share and adapt this material. Copyright holders of content included in this material should contact with any questions, corrections, or clarification regarding the use of content. For more information about how to cite these materials visit Any medical information in this material is intended to inform and educate and is not a tool for self- diagnosis or a replacement for medical evaluation, advice, diagnosis or treatment by a healthcare professional. Please speak to your physician if you have questions about your medical condition. Viewer discretion is advised: Some medical content is graphic and may not be suitable for all viewers. 1
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Systematic Evaluation to Non-Traumatic Head CTs Rashmi U. Kothari, MD KCMS/MSU Source Undetermined
Why do you need to be able to evaluate a CT Radiology report is not immediately available Need immediate intervention Don’t trust anyone
Course Outline Basic principles of CT Basic anatomy Systematic approach CT Potpourri
Course Goals Learn “Blood Can Be Very Bad” approach to reading CTs Identify classic CT findings
Disclaimer Make you a neuroradiologist Teach you cause of finding of abnormality Help you with contrast CTs
Basic Principles of CT Imaging Source Undetermined
X-rays Absorbed Differently by Different Tissues Radiodense Bone Metal Calcium Blood Grey matter Radiolucent Air Spinal fluid Ischemic infarct Edema White matter Source Undetermined
Attenuation (amount of radiation blocked by tissue) Air Blood Bone HU HU HU=Hounsfield Units HU
Windowing Blood Brain Blood Bone Source Undetermined
CT Anatomy Six levels of cuts Cortical sulci Lateral Ventricles Basal Ganglia 3 rd Ventricle Midbrain Pons Source Undetermined
CT Anatomy: Cortical Sulci & Lat. Ventricle Falx Cortical sulci Frontal lobe Parietal lobe Lateral ventricles Occipital lobe Source Undetermined
CT Anatomy: Basal Ganglia & 3 rd Ventricle Anterior horns Insular ribbon Sylvian fissure 3 rd ventricle Quadrigeminal cistern Anterior horns Choroid plexus. Source Undetermined
CT Anatomy: Midbrain & Pons Frontal sinus Suprastellar cistern Pons 4 th ventrical Sylvian fissure Insular ribbon Ambient cistern (cirummesenphalic cistern) Source Undetermined
Systematic Approach to Head CTs Perron et al: Carolina’s Medical Center “Blood Can Be Very Bad” pnemonic Course reviewing 12 scans & short histories Pre-test 60% to Post-test 78% _peds/perron_ich _acep_2005_peds_ course.pdf
“Blood Can Be Very Bad” Blood Cisterns Brain Ventricles Bone Perron et al: Ann Emerg Med 1998:32:
“Blood Can Be Very Bad” Acute blood = hyperdense (white) HU As it ages it becomes hypodense At 1-2 weeks it is isodense with brain
“Blood Can Be Very Bad” Source Undetermined
“Blood Can Be Very Bad” 4 cisterns: Suprasellar Quadrigeminal Slyvian Ambient Source Undetermined
Andrew D. Perron, MD, FACEP 21 Cisterns: Is there blood? Are they open? Source Undetermined
“Blood Can Be Very Bad” Brain Source Undetermined
“Blood Can Be Very Bad” Ventricle Source Undetermined
Andrew D. Perron, MD, FACEP “Blood Can Be Very Bad” Bone Source Undetermined
Classic CT Findings Source Undetermined
Subdural Concave shape Venous bleeds Crosses suture line Epidural Lens shape 85% arterial bleeds Middle meningeal art Lucid period Source Undetermined
Intracerebral Hemorrhage 10% of all strokes 2 major causes Hypertension Blacks & Asians 50% basal ganglia Pons Cerebellum Amyloid Caucasians Lobar Recurrent Source Undetermined
Subarachnoid Hemorrhage 5-10% of all strokes Aneurysms, AVMs, trauma Hyperdense, fuzzy Locations of blood C- –Sulci –Sylvian fissure –Circle of Willis –Falx –Tentorium Source Undetermined
ICHNormalICH SAHSAHSAH Source Undetermined
Findings Suggestive of ICH Normal Calcification Basal ganglia Choroid plexus Pineal gland C- Source Undetermined
Findings Suggestive of ICH Metal Very hypodense “Sparks” Clips, bullets, metallic catheters C- Source Undetermined
Findings Suggestive of ICH C- ? Source Undetermined
Volume Averaging (Technical Issues Mimicking ICH) Orbital roof Petrous portion of temporal bone Pituitary fossa Brainstem Source Undetermined
Findings Suggestive of ICH/SAH ? ? C- Source Undetermined
Motion Artifact (Technical Issues Mimicking ICH or SAH) Streaky Hyperdense Boney prominence Source Undetermined
Evolution of an Infarct Ultra-Acute 0-3 hours Acute-Subacute 6hrs-days Chronic 1 year Source Undetermined
Ultra-Early CT Findings Normal Sulcal effacement Loss of insular ribbon Loss of grey-white interface Acute hypodensity Source Undetermined
Sulcal Effacement Source Undetermined
Loss of Insular Ribbon Source Undetermined
Loss of Sulci & Acute Hypodensity Source Undetermined
Acute Hypodensity Source Undetermined
Acute-Subacute Stroke (hours-days) Hypodense Well demarcated Mass effect Midline shift Loss of sulci Source Undetermined
Old Infarct (months to years) Density of CSF Well demarcated Ventrical enlargement Sulci enlargement No sulcal effacement No mass effect Source Undetermined
Suggestive of an Infarct? Source Undetermined
Suggestive of an Infarct? Tumor Stroke Source Undetermined
Case Presentations
Thalamic ICH Source Undetermined
Normal Source Undetermined
Chronic Frontal Subdural Source Undetermined
Subacute Right Parietal Infarct Source Undetermined
continued Source Undetermined
SAHNormal Source Undetermined
Acute Subdural Source Undetermined
Normal Source Undetermined
Closed Ventricles Source Undetermined
Andrew D. Perron, MD, FACEP Cisterns: Are they open? Source Undetermined
Metallic Artifact Source Undetermined
Brainstem SAH Source Undetermined
Chronic MCA Infarct Source Undetermined
Left IVH Source Undetermined
Epidural Source Undetermined
Rt Subacute Epidural Source Undetermined
Sagital Sinus Source Undetermined
Subacute Infarct Source Undetermined
Renal Cell Metastasis Source Undetermined
continued Source Undetermined
SAH Normal Source Undetermined
48 hr old Right Temporal Infarct Source Undetermined
Acute on Chronic Subdural Source Undetermined
SAH Source Undetermined
Rt Parietal Fx with Air Source Undetermined
Brain Abscess Source Undetermined
Calcification Basal Ganglia Source Undetermined
continued Source Undetermined
hours 3-4 days 7-10 days months Source Undetermined
Trauma with Air Source Undetermined
Dense MCA Sign Source Undetermined
Subacute Brainstem Infarct Source Undetermined
Atrophy Source Undetermined
Trauma with SAH Source Undetermined
Bitemporal Edema (Herpes) Source Undetermined
Meningioma Source Undetermined
Caudate Infarct Source Undetermined
IVH Left Lateral Horn Source Undetermined
Ultra-Early Right Parietal Infarct Right Sulcal Effacement Source Undetermined
Continued Source Undetermined
Subacute Infarct (Rt Temporal Lobe) Source Undetermined
Periventricular White Matter Disease Source Undetermined
Chronic Rt Occipital Infarct Source Undetermined
Subacute Subdural Source Undetermined
Traumatic Petechae Source Undetermined
Loss of Sulci & Sylvian Fissure Source Undetermined
Old Lt Lacunar Infarct Source Undetermined
Subacute Lt Subdural Source Undetermined
Rt MCA Infarct with Hemorrhage Source Undetermined
Lt Sagital Vein Thrombosis Source Undetermined
SAH with Blood along Falx & in Ventricle Source Undetermined
Tumor Source Undetermined
Tumor Source Undetermined
CT Ground Rule Radiodense Bone Blood Calcium Grey matter Metal Radiolucent Spinal fluid Ischemic infarct Edema White matter Air Source Undetermined
“Blood Can Be Very Bad” Blood Cisterns Brain Ventricles Bone Perron et al: Ann Emerg Med 1998:32:
Intracerebral Hemorrhage Appearance Hyperdense Well demarcated Globular Location Intraparenchyma l Mimics Normal Calcification Basal ganglia Choroid plexus Pineal gland Artifacts Metal Catheters Volume Averaging Motion Source Undetermined
Subarachnoid Hemorrhage Appearance Hyperdense Fuzzy Locations of blood Sulci Sylvian fissure Circle of Willis Falx Tentorium Mimics Contrast Calcified Falx Normal Tentorium Motion artifact Source Undetermined
Ultra-Early Infarct Normal Sulcal effacement Loss of insular ribbon Loss of grey-white interface Acute hypodensity Acute-Subacute Hypodense Well demarcated Mass effect Midline shift Loss of sulci Old Infarcts Density of CSF Well demarcated Ventrical enlargement Sulci enlargement No sulcal effacement No mass effect Source Undetermined