 Pathogen:  --hemolytic streptococci anaerobe mixed infection  Pathology: catarrhal purulent (follicular+lacunar)

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Presentation transcript:

 Pathogen:  --hemolytic streptococci anaerobe mixed infection  Pathology: catarrhal purulent (follicular+lacunar)

 Signs and symptoms:  Fever  Sore throat  Tender cervical lymphadenopathy  Dysphagia  Erythematous tonsils with exudates

 Same signs and symptoms as acute  Occurring in 4-7 separate episodes per year  5 episodes per year for 2 years  3 episodes per year for 3 years

1. chronic / abscess 2. hypertrophy 3. focus 4. keratosis / diphtheria 5. tumor

 Current clinical indicators of AAO-HNS:  3 or more infections per year despite adequate medical therapy  Hypertrophy causing dental malocclusion or adversely affecting orofacial growth documented by orthodontist  Hypertrophy causing upper airway obstruction, severe dysphagia, sleep disorder, cardiopulmonary complications

 Peritonsillar abscess unresponsive to medical management and drainage documented by surgeon, unless surgery performed during acute stage  Persistent foul taste or breath due to chronic tonsillitis not responsive to medical therapy  Chronic or recurrent tonsillitis associated with streptococcal carrier state and not responding to beta-lactamase resistant antibiotics  Unilateral tonsil hypertrophy presumed neoplastic

1. acute attack 2. blood disorder 3. general disorder 4. infectious disease 5. menstruation/pregnancy 6. immune deficiency

 acute tonsillitis  peritonsillar cellulitis  abscess   --hemolytic streptococci…  types: anterior-superior posterior-superior

 Abscess formation outside tonsillar capsule  Signs and symptoms:  Fever(>3-5d)  Sore throat  muffled voice  Dysphagia/odynophagia  Drooling  Trismus  Unilateral swelling of soft palate/pharynx with uvula deviation

 Thought to be extension of tonsillitis to involve surrounding tissue with abscess formation  Recently described to be an infection of small salivary glands in the supratonsillar fossa called Weber’s glands  Would explain superior pole involvement and the usual absence of tonsillar erythema/exudates

 puncture and incision (diagnosis & therapy)  antibiotics  tonsillectomy

Thank you !