This article and any supplementary material should be cited as follows: Oleson CV, Seidel BJ, Zhan T. Association of vitamin D deficiency, secondary hyperparathyroidism,

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This article and any supplementary material should be cited as follows: Oleson CV, Seidel BJ, Zhan T. Association of vitamin D deficiency, secondary hyperparathyroidism, and heterotopic ossification in spinal cord injury. J Rehabil Res Dev. 2013;50(9):XX–XX. Slideshow Project DOI: /JRRD JSP Association of vitamin D deficiency, secondary hyperparathyroidism, and heterotopic ossification in spinal cord injury Christina V. Oleson, MD; Benjamin J. Seidel, DO; Tingting Zhan, PhD

This article and any supplementary material should be cited as follows: Oleson CV, Seidel BJ, Zhan T. Association of vitamin D deficiency, secondary hyperparathyroidism, and heterotopic ossification in spinal cord injury. J Rehabil Res Dev. 2013;50(9):XX–XX. Slideshow Project DOI: /JRRD JSP Aim – Explore relationship between low vitamin D, secondary hyperparathyroidism, and heterotopic ossification (HO) in patients with spinal cord injury. Relevance – Many subjects with deficient vitamin D exhibit HO in bones below neurological level of injury.

This article and any supplementary material should be cited as follows: Oleson CV, Seidel BJ, Zhan T. Association of vitamin D deficiency, secondary hyperparathyroidism, and heterotopic ossification in spinal cord injury. J Rehabil Res Dev. 2013;50(9):XX–XX. Slideshow Project DOI: /JRRD JSP Method For 96 subjects: – Measured levels of serum vitamin D25(OH), calcium, and intact parathyroid hormone (PTH). – Obtained nutritional patterns and fracture history. – Ascertained evidence of current or previous HO through chart review.

This article and any supplementary material should be cited as follows: Oleson CV, Seidel BJ, Zhan T. Association of vitamin D deficiency, secondary hyperparathyroidism, and heterotopic ossification in spinal cord injury. J Rehabil Res Dev. 2013;50(9):XX–XX. Slideshow Project DOI: /JRRD JSP Results 12 subjects exhibited HO. – 11 with serum vitamin D25(OH) between 5–17 ng/mL. 9 subjects exhibited secondary hyperparathyroidism (72–169 pg/mL). Significant correlations found between: – Hyperparathyroidism and HO. – Hyperparathyroidism and vitamin D deficiency (<20 ng/mL).

This article and any supplementary material should be cited as follows: Oleson CV, Seidel BJ, Zhan T. Association of vitamin D deficiency, secondary hyperparathyroidism, and heterotopic ossification in spinal cord injury. J Rehabil Res Dev. 2013;50(9):XX–XX. Slideshow Project DOI: /JRRD JSP Conclusion No direct correlation between HO and low vitamin D, but hyperparathyroidism may increase risk. Patients with low vitamin D and elevated PTH should be screened for HO and begin vitamin supplementation. – Initiating early treatment of low vitamin D to restore therapeutic levels may prevent HO development.