ECG PRACTICAL APPROACH Dr. Hossam Hassan Consultant Emergency Medicine
Objectives To emphasize simplicities Practical approach Interpretation & clinical scenario are inseparable Systematic approach
Conduction System
Nomenclature
Magic numbers of Dr. Hossam
Systematic approach Rate Rhythm axis P-wave PR interval QRS complex ST segment T-wave
Rate The interval between 2 successive R-wave How many big squares? Divide 300 / # big squares Normal 60 – 100/min
Rhythm Sinus Rhythm Every P=wave is followed by QRS complex P-wave is upright in lead II
NSR
Types of Sinus Rhythm NSR Sinus Tachycardia Sinus Bradycardia Sinus arrhythmia
Sinus tachycardia
Axis Normal axis Right axis deviation Left axis deviation
RAD
LAD
P-wave Atrial depolarization Atrial contraction is a result Normally a dome-like structure
Abnormalities of P-wave Peaked p-pulmonle –Pulmonary HTN –PE –Pulmonary valve stenosis M-shaped M-mitrale –Mitral valve stenosis –Left atrial hypertrophy Inverted 2 nd atrial / junctional ectopy
P-pulmonale
PR interval Definition From the start of P to beginning of QRS Represent the delay in transmission in AV node Normally 0.12 – 0.20 msec
Abnormalities of PR interval Prolonged > 1 st degree HB Short < Pre-excitation syndromes –WPW Syndrome –LGL Syndrome Junctional rhythm
QRS Complex Amplitute Duration Shape Q-wave R-wave
QRS AMPLITUTE LVH By voltage criteria –S-wave in V 1 or V 2 + R-wave in V5 or V6
LVH & STRAIN PATTERN
Causes of LVH HTN Aortic stenosis HOCM Aortic regurgitation Mitral regurgitation
QRS DURATION Ventricular depolarization Ventricular contraction is a result Normally < 0.12 msec < small squares
Causes of wide QRS Ventricular tachycardia BBB –Left BBB –Right BBB
L BBB
R BBB
Shape Upstroke & downstroke of R-wave Delta wave
Q-wave 1 st negative deflection after the P-wave Normally 1mm wide & 2 mm deep Lead III, V5 & V6 Pathological Q-wave Wider & deeper >1/4 of the ensuing R-wave Old MI
+ve R-wave in V1
Causes +ve R-wave in V I RVH R BBB Posterior MI Type A WPW
ST-Segment From the end of S-wave to the beginning of T-wave Normally iso-electric Abnormalities –Elevated –depressed
Elevated ST segment Acute MI Pericarditis Early repolarization pattern in the young
Infarct localization Inferior –Lead II, III, aVF Septal –V I, V II Anterior –V3, V4
Lateral –Lead I, AVL,V5, V6 Posterior MI - Prominent R wave in V1,V2 with depressed ST segment
Acute inf MI
Anteroseptal MI
Anterior MI
Lateral MI
Depressed ST Segment Unstable angina Left ventricular strain pattern
LVH & strain pattern
T-wave Ventricular repolarization Dome like structure Abnormalities –Peaked / tented t-wave Hyperkalaemia Subendocadial ischemia –Inverted LV Strain pattern Dynamic t-wave changes of ischemia
DYNAMIC T-WAVE CHANGES
Hay….. wake up we are done