PTC shock Lt. col. Dr. Zaman Ranjha Associate prof. of Surgery
PTC Shock Objectives To understand the structured approach to cerculatory problems To recognize and manage shock
PTC Shock Inadequate organ perfusion and tissue oxygenation Most often due to hypovolaemia in surgery and trauma
PTC Shock Assessment Blood pressure Heart rate Capillary refill Peripheral temperature Peripheral colour Urine output
PTC Types of Shock Hypovolaemic Cardiogenic Obstructive Neurogenic Endocrine Anaphylactic septic
PTC Severity of shock Compensated Vasomotor response At the cost of skin, muscles and GIT. Acidosis beyond 12 Hrs- MOD Decompensated 30-40% volume loss Cadiopulmonary and renal compensation is knocked out
PTC Shock Pathophysiology Cellular Autodigestive enzyme-cell lysis Microvascular o2 free radical- endothelial damage Mode of death rapid-cadiopulmonary delayed-organ ischemia/reperfusion
PTC Hypovolumic shock Fluid loss less intake, increased loss- vomiting, GIT, Renal third space- pancreatitis Blood loss
PTC Shock Sites of blood loss Closed Femoral #1.5-2 litres Closed Tibial # 500 ml Pelvic # 3 litres Rib # (each)150 ml Haemothorax 2 litres Hand sized wound 500 ml Fist sized clot500 ml
PTC Shock Concealed blood loss Abdominal Cavity Pleural Cavity Femoral Shaft Pelvic Fractures Scalp (children)
PTC Types Of Bleeding Compressible - usually peripheral Non-compressible - e.g. intra-abdominal - Surgery required
PTC Shock systemic effects CVS.-Sympathomymatic tachycardia-vasoconstriction Resp. -compensatory respiratory alkalosis Renal. Reduced perfusion, GFR, Urine Na, H2o, conservation Endocrine.Adrenal,cortisone =Na +water catecholamine Hypothalamus- vasopressin
PTC Shock Clinical Signs Altered mental state : anxiety to coma Pulse present ? - radial systolic > 80 mmHg - femoral systolic >70 mmHg - carotidsystolic > 60 mmHg Tachycardia Pulse pressure narrowed
PTC Shock Clinical Signs Skin - cold, pale, sweaty, cyanosed Capillary refill time > 2 seconds Blood pressure JVP Urine output < 0.5 ml/kg/hr Respiratory rate
PTC Clinical Signs In Shock
PTC Blood Loss < 750ml
PTC Blood Loss ml
PTC Blood Loss >1500ml
PTC Cardiogenic Shock myocardial contusion cardiac tamponade tension pneumothorax penetrating wound of heart myocardial infarction Valvular heart disease arrhythmya
PTC Shock Obstructive shock Cardiac temponade Tension pneumothorax Pulmonary embolism Reduced preload Reduced cardiac out put Engorged neck veins + oedma
PTC Shock Endocrine shock May be combination of three Adrenal- hypovolumic Hypothyroid- neurogenic Hyperthyroid – high out put
PTC Shock Distributive shock No volume depletion 1-Septic shock Endotoxin-vasodilation-AV shunting- cellular hypoxia 2- Anaphylactic shock Histamine- vasidilatation 3- Neurogenic- vasomotor
PTC Shock ?
PTC Shock Management A + B, oxygen (if available) Two large bore intra-venous cannulae Stop obvious bleeding Fluid replacement Maintain temperature Analgesia
PTC Shock Stop bleeding Chest –Drain tube and re-expand lung –Emergency thoracotomy rarely Abdomen –Laparotomy if hypotensive after fluids Limbs –Pressure dressing –Tourniquet is last resort
PTC Shock Fluid replacement Warm fluids if possible Colloids or crystalloids? Consider hypotensive resuscitation if haemostasis not secure- parallel with surgery Consider oral resuscitation Resuscitation beneficial –dehydration
PTC Shock Fluid replacement - How much? ml 0.9% Saline or Ringer’s Reassess ml 0.9% Saline or Ringer’s Reassess Consider blood Consider surgery Aim for systolic BP>90 + HR <100
PTC Shock Consider blood Tx Haemodynamic instability in spite of fluids Haemoglobin <7g/dl and patient still bleeding
PTC Shock ?
PTC Shock Summary Careful assessment Stop the bleeding Replace volume Correct the cause