How To Look To Patient Data DATA History Taking o Growth o Exercise Intolerance o Recurrent Chest Infection o Syncopal Attacks o Squatting.

Slides:



Advertisements
Similar presentations
CONGENITAL HEART DISEASE.
Advertisements

Sri Sathya Sai Institute of Higher Medical Sciences
Atrioventricular Canal Defect
Congenital Heart Defects Fred Hill, MA, RRT. Categories of Heart Defects Left-to-right shunt Cyanotic heart defects Obstructive heart defects.
 Cardiovascular System – Heart and Blood Vessels Topics in Human Pathophysiology Fall 2011 Gilead Drug Safety and Public Health.
Congestive Heart Failure
 Heart failure is a complex clinical syndrome Can result from:  structural or functional cardiac disorder  impairs the ability of the ventricle to.
Cardiovascular System
Ass. Professor of Cardiology
Atrial and Ventricular Enlargement
W. Kissinger Tintinalli Sixth Edition Chapter 52
Case scenarios Atrial switch Univentricular repair
CO - RELATION WITH ECG INTRA CARDIAC PRESSURES ASHOK MADRAS MEDICAL MISSION CHENNAI
Cardiomyopathies Dr. Hesham K. Rashid, MD Ass. Professor of Cardiology Benha University.
Cardioanaesthesia. Coronary artery disease O 2 delivery Coronary blood flow = directly related to coronary perfusion pressure (CPP) CPP = aortic diastolic.
Dr. Meg-angela Christi M. Amores
August 9, THE CARDIOVASCULAR SYSTEM IN CHILDHOOD EVALUATION AND TREATMENT SSA Conference, September 24, 2008 Joel Brenner, MD Director, Pediatric.
Congenital Heart Defects Functional Overview
Ventricular Diastolic Filling and Function
In the Name of Allah the Most Beneficent and Merciful C ardiomyopathies Prof. Dr. Muhammad Akbar Chaudhry M.R.C.P.( UK ), F.R.C.P.( E ) F.R.C.P. ( LONDON.
Common Clinical Scenarios *Younger people *Younger people _Functional murmur vs _Functional murmur vs _ MVP vs _ MVP vs _ AS _ AS *Older people _Aortic.
Cardiac Pathology: Valvular Heart Disease, Cardiomyopathies and Other Stuff.
Outline The critical physiological changes of pregnancy. The critical physiological changes of pregnancy. Predictors of cardiac events during pregnancy.
Homeostatic imbalances
Inflammatory and Structural Heart Disorders Valvular Heart Disease
© Continuing Medical Implementation ® …...bridging the care gap Cardiovascular Aging.
Mitral Valve Disease Prof JD Marx UFS January 2006.
Sudden Cardiac Death; Invasive Evaluation Alpay Çeliker MD Hacettepe University Department of Pediatric Cardiology Ankara, Türkiye.
Palliative Operation Seoul National University Hospital Department of Thoracic & Cardiovascular Surgery.
One & A Half Ventricle Repair
Differential Diagnosis. Many classes of disorders can result in increased cardiac demand or impaired cardiac function. Cardiac causes include: - arrhythmias.
Management of Adult Congenital Heart Disease Alpay Çeliker MD. Hacettepe University Department of Pediatric Cardiology.
Myocardial infarction My objectives are: Define MI or heart attack Identify people at risk Know pathophysiology of MI Know the sign & symptom Learn the.
MITRAL VALVE DISEASES. MITRAL VALVE DISEASES 1. Mitral valve stenosis. 2. Mitral valve regurge. 3. Mitral valve prolapse.
Clinical Symptoms of Atrial Fibrillation in Different Ranges of QRS Duration Burda I.Yu., Yabluchansky N.I. Medical Clinics Chair National University of.
Medical Disease in Pregnancy Cardiovascular Disease Cullen Archer, MD Department of Obstetrics and Gynecology.
Cardiovascular surgery, Congenital heart disease Dr. Robin Man Karmacharya, Lecturer, Department of Surgery, Dhulikhel Hospital.
Angina & Dysrhythmias. A & P OF THE CARDIAC SYSTEM Cardiac output  CO=SV(stroke volume) X HR(heart rate) Preload  Volume of blood in the ventricles.
1 بسم الله الرحمن الرحیم. Atrial and Ventricular Hypertrophy ECG Features and Common Causes ALI BARABADI University of Guilan.
Frank-Starling Mechanism
Definition and Classification of Shock
Aortic Insufficiency Acute and Chronic
Chapter 9 Heart. Review of Structure and Function The heart is divided into the systemic (left) and pulmonary (right) systems –The pulmonary system has.
Cardiovascular Blueprint PANCE Blueprint. Dilated Cardiomyopathy Defined as being characterized by enlargement of chambers and impaired systolic function.
Exercise Management Atrial Fibrillation Chapter 9.
How do you manage this patient?. Diagnostic An adequate diagnostic workup: Documents the presence and type of ASD(s) Determines the size (diameter) of.
Arrhythmias. Cardiac dysrhythmia Cardiac dysrhythmia (arrhytmia) Abnormal electrical activity in the heart.
First degree AV block Or PR prolongation. atrioventricular block:, AV block impairment of conduction of cardiac impulses from the atria to the ventricles,
Valvular Heart Disease
By M.elkhatib.  Equal  R = L  Q refers to flow  Therefore Qp = Qs  Blood flow to both the pulmonary & systemic circulations is balanced.  Homeostasis.
Heart Failure Heart failure (HF) is a common clinical syndrome. Heart failure (HF) is a complex clinical syndrome that can result from any structural or.
RJS How and why the heart goes wrong. RJS What there is to go wrong.
 By the end of this lecture the students are expected to:  Explain how cardiac contractility affect stroke volume.  Calculate CO using Fick’s principle.
INFANTS OF DIABETIC MOTHERS MUHAMMAD ALI Cardiology Division Department of Child Health University of Sumatera Utara.
Congestive Heart Failure MUHAMMAD ALI PEDIATRIC CARDIOLOGY DIVISION.
Atrial Septal Defect R3 이재연.
Echo conference R4 우종신 R4 우종신. Case 1 한 O 태 () Evaluation of severity Planimetry of mitral orifice Planimetry of mitral orifice –only direct measurement.
Cardiomyopathies Pavol Tomašov.
Cardiac Catheterization Complication
Heart & Thalassemia . R.Miri,MD, Interventional Cardiologist.
Pharmacotherapy Of Cardiovascular Disorders: Heart Failure
Dr. M. A. Sofi MD; FRCP (London); FRCPEdin; FRCSEdin
Of Cardiovascular diseases
Objectives 1-To discuss V.S.D.
Acromegalic cardiomyopathy: A case report
The Cardiovascular System
Cath-Lab Hemodynamics – I : Pressure tracings in the diseased heart
Part I Fetal Circulation, ASD, VSD
Definition and Classification of Shock
Khalid AlHabib Professor of Cardiac Sciences Cardiology Consultant
Presentation transcript:

How To Look To Patient Data DATA

History Taking o Growth o Exercise Intolerance o Recurrent Chest Infection o Syncopal Attacks o Squatting

ECG, Echo & Cardiac Cath. Systolic & Diastolic Dysfunction Reduced Fractional Shortening Systolic Dysfunction

Diastolic Dysfunction Ventricular Hypertrophy Obstructive Volume Before Repair e.g valvular & outflow obst. Before Repair e.g valvular & outflow obst. After Repair e.g Homograft conduit After Repair e.g Homograft conduit Before Repair e.g Lt. to Rt. shunt Before Repair e.g Lt. to Rt. shunt After Repair e.g Pulmonary valve regurge ( F4 ) MV repair After Repair e.g Pulmonary valve regurge ( F4 ) MV repair ConcentricEccentric

Anaesthetic considerations : Consider determinants of coronary perfusion & myocardial oxygen balance Heart rate changes Hypotension Myocardial contractility

Anaesthetic considerations Cardiomyopathy increase wall thickness coronary filling becomes diastolic coronary perfusion depends on bl. p. & hr Maintain heart rate to decrease regurgitant fraction Syst. Dysfunction In Dialted type RVLV anaesthetic myocardial depression Decrease driving filling pressure of coronary arteries Coronary ischemia Diast. Dysfunction In Hypertrophic & restrictive type

Residual Shunts : o Occasionally present after repair of ASD, VSD & F4 o Small patch leaks are hemodynamically benign

Dysrhythmias : Atrial & ventricular types increase mortality and morbidity Arrhythmias Associated With Specific Surgical Procedures Ostium secondum ASD : P-R interval is prolonged in 20-30% of patients AF, atrial flutter with advancing age

VSD : RBBB Atrial ectopic, junctional beats, premature ventricular beat Late onset of complete heart block or ventricular arrhythmias are rare Repair of F4 : RBBB & complete heart block Mustard or Senning operation : Sinus nodal dysfunction Bradycardia A-V block, AF

Severity of hypertension of base line PAH correlated with the incidence of major complications ( pulmonary hypertensive crisis or cardiac arrest ) Pulmonary hypertension

Cardiovascular risk of PAH Major perioperative hemodynamic deterioration mainly pulmonary hypertensive crisis and acute right ventricular failure and cardiac arrest. Data to look for : o Mean pulmonary artery pressure > 25 mmHg o Severity of base line PH : Subsystemic PAP < 70% of syst. bl. pressure Systemic PAP = 70 – 100 of syst. bl. pressure Suprasystemic PAP > 70 of syst. bl. pressure ( based on mean pressures )

ANAESTHETIC CONSIDERATIONS Avoid Factors Rapidly Increasing PVR

Laboratory data Hematocrit value Decompansated Erythrocytosis HCT. Increase Erythropoitin Level Increase Red Cell Mass Increase More Blood Viscocity Hyperviscosity symptoms Decreased oxygen delivery

Blood Indicies : Microspherocytosis Iron Deficiency Anaemia Rigid Cell Membrane Low Hemoglobin Concentration Increase Blood Viscosity Hyperviscosity Symptoms At Lower Hematocrit Value

Phlebotomy Done to relieve hyperviscosity symptoms with hematocrit > 65 % in absence of iron deficiency anaemia or signs of dehydration

Hemostatic values Prolonged PT, PTT, APTT values most frequently seen in cyanotic patients Thrombocytopenia is related to degree of polycythemia.

Summary General associated risk factors in CHD  Severe form of isolated lesion  Complex lesions  Concurrent infectious disease  Congestive heart failure  Acute hemodynamic deterioration  Previous palliative or corrective procedures

Summary Risk criteria of hemodynamic critical impairment in perioperative period in CHD Arterial saturation < 75 % Hematocrit > 65 % Qp / Qs > 2 : 1 LV outflow tract gradient > 50 mmHg RVOT gradient > 50 mmHg PVR > 6 wood units

THANK YOU