Figure 18.1 Motor components of the human basal ganglia

Figure 18.1 Motor components of the human basal ganglia (Part 1)

Figure 18.1 Motor components of the human basal ganglia (Part 2)

Figure 18.2 Anatomical organization of the inputs to the basal ganglia

Figure 18.3 Neurons and circuits of the basal ganglia

Figure 18.3 Neurons and circuits of the basal ganglia (Part 1)

Figure 18.3 Neurons and circuits of the basal ganglia (Part 2)

Figure 18.4 Regions of the cerebral cortex that project to the corpus striatum

Figure 18.5 Functional organization of intrinsic circuitry and outputs of basal ganglia

Figure 18.5 Functional organization of intrinsic circuitry and outputs of basal ganglia (Part 1)

Figure 18.5 Functional organization of intrinsic circuitry and outputs of the basal ganglia (Part 2)

Figure 18.6 A chain of nerve cells arranged in a disinhibitory circuit

Figure 18.6 A chain of nerve cells arranged in a disinhibitory circuit (Part 1)

Figure 18.6 A chain of nerve cells arranged in a disinhibitory circuit (Part 2)

Figure 18.7 Basal ganglia disinhibition and the generation of saccadic eye movements

Figure 18.7 Basal ganglia disinhibition and the generation of saccadic eye movements (Part 1)

Figure 18.7 Basal ganglia disinhibition and the generation of saccadic eye movements (Part 2)

Figure 18.8 Disinhibition in the direct and indirect pathways through the basal ganglia

Figure 18.8 Disinhibition in the direct and indirect pathways through the basal ganglia (Part 1)

Figure 18.8 Disinhibition in the direct and indirect pathways through the basal ganglia (Part 2)

Figure 18.9 Center-surround functional organization of the direct and indirect pathways

Figure Neurological diseases provide insights into function of the basal ganglia

Figure Hypo- and hyperkinetic disorders alter the balance of inhibitory signals in the direct and indirect pathways

Figure Hypo- and hyperkinetic disorders alter the balance of inhibitory signals in the direct and indirect pathways (Part 1)

Figure Hypo- and hyperkinetic disorders alter the balance of inhibitory signals in the direct and indirect pathways (Part 2)

Box 18C Deep Brain Stimulation

Box 18C Deep Brain Stimulation (Part 1)

Box 18C Deep Brain Stimulation (Part 2)

Figure Inactivation of tonically active cells of substantia nigra pars reticulata causes saccades

Box 18D Basal Ganglia Loops and Non-Motor Brain Functions

Box 18D Basal Ganglia Loops and Non-Motor Brain Functions (Part 1)

Box 18D Basal Ganglia Loops and Non-Motor Brain Functions (Part 2)