Arrhythmias of heart Department of propedeutic of internal medicine.

Slides:



Advertisements
Similar presentations
Updated March 2006: D. Tucker, RPh, BCPS
Advertisements

Normal ECG waves & ARRYHTHMIAS
PREAPRED BY; Moneer al-aliowh SUPERVISED BY; D, yshmen al-fholy
By Dr.Ahmed Mostafa Assist. Prof. of anesthesia & I.C.U.
UNC Emergency Medicine Medical Student Lecture Series
Cardiovascular 2 Phase 2 Michelle Mair
The electrocardiogram (ECG or EKG)
MALIGNANT ARRHYTHMIAS: ECG IDENTIFICATION DR.SIVAKUMAR ARDHANARI MD
ECG Rhythm Interpretation
ECG Rhythm Interpretation
Cardiovascular course 4th year - Pathophysiology
Electrocardiography Arrhythmias Review
Arrhythmia Rhythm refers to the regularity or spacing of the ECG waves. Any variation from the normal rhythm and sequence of excitation of the heart is.
Fast & Easy ECGs, 2nd E – A Self-Paced Learning Program
Presentation Information
Normal ECG: Rate and Rhythm
Clk. Alexander L. Gonzales II December 14, EKG Characteristics: Regular narrow-complex rhythm Rate bpm Each QRS complex is proceeded by a.
Junctional Rhythms / A-V Nodal Rhythm. Aims and Objectives.  Investigate common types of Junctional and AV nodal tachycardias.  Understand underlying.
Junctional Dysrhythmias
Arrythmia Interpretation (cont’d) Rates of automaticity – Too fast (tachycardia) – Too slow (bradycardia) – Too irritable (Premature) – Absent (block)
Natalia Fernandez, PT, MS, MSc, CCS University of Michigan Health Care System Department of Physical Medicine and Rehabilitation.
 Any atrial area may originate an impulse.  Rhythms have upright P waves preceding each QRS complex.  Not as well-rounded  Heart rates usually from.
ECG/EKG. ECG ECG stands for Electrocardiogram Sooo smart students, what do you think it measures?
Your heart is a muscle that works continuously like a pump Each beat of your heart is set in motion by an electrical signal from within your heart muscle.
Elsevier items and derived items © 2006 by Elsevier Inc. Chapter 37 Interventions for Clients with Dysrhythmias.
Supraventricular Arrhythmias Claire B. Hunter, M.D.
Due to certain diseases or body disorders, heart start beating abnormally. Due to this abnormal beating their is great variation in ECG. In this lecture.
Department faculty and hospital therapy of medical faculty and department internal diseases of medical prophylactic faculty. Cardiac arrhythmia Docent.
Fast & Easy ECGs – A Self-Paced Learning Program
Disease of Cardiac System
Abnormal Sinus Rhythms
Lecture Objectives Describe sinus arrhythmias Describe the main pathophysiological causes of cardiac arrhythmias Explain the mechanism of cardiac block.
Normal electrocardiogram
EKG Interpretation: Arrhythmias Mustafa Salehmohamed, D.O. Assistant Clinical Instructor Department of Medicine N.Y. College of Osteopathic Medicine October.
Junctional Dysrhythmias
The Basics of ECG Interpretation Dr Tim Smith. Summary Cardiac conducting system and the ECG waveform Cardiac conducting system and the ECG waveform The.
For more presentations FOR MORE FREE MEDICAL POWERPOINT PRESENTATIONS VISIT WEBSITE blogspot.com.
 Cardiac dysrhythmia (arrhythmia and irregular heartbeat) is a large and heterogeneous group of conditions in which there is abnormal electrical activity.
Causes of cardiac arrhythmias
Chapters 11, 12, 13 Electrocardiogram Dr. Marko Ljubković Department of Physiology.
Cardiac Arrhythmias. Classification. Extrasystoly. Paroxismal tachycardia. Heart blocks. Atrial fibrillation. Etiology. Pathogenesis. Clinical pattern.
Normal EKG – P wave: Atrial depolarization – PR interval: < 0.20 sec – QRS complex: ventricular depolarization – QRS interval < 0.10 sec SA 0.10 – 0.12.
ECG Part II. Rate-measure of frequency of occurrence of cardiac cycles(b/m) < 60 beats/min is a bradycardia beats/min is normal >100 beats/min.
ECG intereptation Abdualrahman ALshehri Lecturer King Saud University
ELECTROCARDIOGRAM An electrocardiogram (EKG or ECG) is a graphic representation of the heart’s electrical activity.
SCN EKG Review and Strip
EKG’s Kelly Marchant RN July 28, 2015 Adapted from NURO 438
MECHANISMS OF CARDIAC ARRHYTHMIAS. DR AMNA TAHIR PHYSIOLOGY DEPARTMENT. KEMU.
Disorders of heart rhythm D.D., Professor Denefil Olha Volodymyrivna.
SIGNS AND SYMPTOMS 1-Chest pain 2-Dyspnea 3-Palpitation 4-Syncopal attach.
Arrhythmias. Cardiac dysrhythmia Cardiac dysrhythmia (arrhytmia) Abnormal electrical activity in the heart.
First degree AV block Or PR prolongation. atrioventricular block:, AV block impairment of conduction of cardiac impulses from the atria to the ventricles,
Arrhythmias An arrhythmia is… – disturbance of the electrical rhythm of the heart. Classification – Supraventicular (sinus, atrial, junctional) and..
Electrocardiography – Abnormalities (Arrhythmias) 7
Dr. Mona Soliman, MBBS, MSc, PhD Associate Professor Department of Physiology Chair of Cardiovascular Block College of Medicine King Saud University.
ARRHYTHMIA Objectives At the end of this session students should be able to:  Distinguish the normal from abnormal rhythms.  Understand the pathophysiologic.
Lecture Objectives Describe sinus arrhythmias Describe the main pathophysiological causes of cardiac arrhythmias Explain the mechanism of cardiac block.
ARRHYTHMIAS Jamil Mayet. Arrhythmias - learning objectives –Mechanisms of action of antiarrhythmic drugs –Diagnosis To differentiate the different types.
Disorder of heart rhythm Ph.D., MD, Assistant Professor Potikha N.Ya.
Arrhythmias and EKGs.
8 Introducing the Atrial Rhythms 1.
Heart Blocks Leaugeay Webre BS, CCEMT-P, NREMT-P.
ECG RHYTHM ABNORMALITIES
ECG Conduction Abnormalities
Objective 12 Electrocardiograms
Lecturer: Prof.Rustamova M.T.
Cardiovascular System Block Cardiac Arrhythmias (Physiology)
ARRHYTHMIA DR MANSOUR ALQURASHI
Antiarrhythmic drugs [,æntiə'riðmik] 抗心律失常药
Presentation transcript:

Arrhythmias of heart Department of propedeutic of internal medicine

Etiology. Violations of rhythm of cardiac activity cause such defeats of myocardium: organic: IHD,defects of heart, AH, myocarditis, cardiomyopathy; · toxic: medicines, alcohol; · hormonal : thyrotoxicosis, myxedema, pheochromocytoma, climax; · functional : neurogenic, sporting; · anomalies of development of heart - more frequent all WPW.

Pathogenesis Theories of origin of arrhythmias: Pathological automatism (presence of ectopic focuses) Mechanism of re-entry trigern (starting) activity.

Classification of violations of rhythm and conductivity of heart I. Violations of formation of impulse Sinus tachycardia (more than 90 complexes are for a minute) Sinus bradycardia (less than 60 complexes are for a minute ) Sinus arrhythmia Stop (refuse) of sinus node Migration of supraventricular driver of rhythm

Extrasystolia (a synonym is premature depolarization): auricle (atrial) auricle-ventrical (atrioventricular) ventrical

Tachycardia: supraventricular: sino - auricle (sinoatrial) auricle (atrial) auricle - ventrical (atrioventrical) ordinary(to 30 in a hour) frequent (30 and anymore in a hour) allorythmia(bi-, thre-, quadrigeminia) polymorphic twin early(R on T) chronic paroxysmal noudle

with the additional ways of leadthrough ortodromal antidromal ventrical: unsteady (from 3 ventrical complexes to 30) steady (more than 30 seconds) monomorphic polymorphic

ІІ. VIOLATION OF LEADTHROUGH OF IMPULSE - Sinoauricular of blockade - Atrioventricular blockades: І st. ІІ st. ІІІ st.

III. COMBINED DYSPOIESISS AND LEADTHROUGHS OF IMPULSE · auricle Parasystole · from AV of connection · ventrical specified at possibility

IV. DISEASE, SYNDROMES And PHENOMENA –Syndromes and ECG phenomena of pre-excitation of ventricles: syndrome of WPW syndrome of the shortened interval of PR (Launa - Ganonga - Livayna) syndrome of early repolarisation of ventricles innate purchased Syndrome of the prolonged interval of QT

Syndrome of weakness of sinus noudle Syndrome of Morganii-Adams-Stoks Syndrome of Frederic Cardiac sudden death (arrhythmical) asystole fibrilation of ventricles electromechanics dissociation Heart arest

V. ARRHYTHMIAS ARE AT NORMAL OR BROKEN FUNCTION OF CARDIOSTIMULATORS OF DIFFERENT TYPE Note: In a diagnosis it is needed to specify cardiac surgery interferences and devices, applied for treatment of arrhythmias and asequences heart (with pointing of method and to give interference) - kateterni (radio frequency et al) destruction, implantation of drivers of rhythm and cardioverters - defibrilyatoriv, leadthrough of cardioversion or defibrilyacii (the date of the last is specified) and others like that.

Description of normal sinus rhythm correct rhythm with frequency of heartbeats per 1 min. the P wave is positive in II, III, AVF leads, negative - in the AVR leads, permanent form of P wave a complex QRS follows by every P waveR (if there is not а-v-blockade). Interval of P-Q>0.12 (if there are not additional ways of leadthrough).

Sinus tachycardia ECG is criteria: correct rhythm sinus P waves are ordinary configuration beats per 1 min. gradual beginning and completion Reasons: physical and emotional loading, pain, fever, hypovolumia, hypotension, anaemia, thyrotoxicosis, action of certain matters (coffeine, alcohol)

Treatment liquidation of etiologic factor B-adrenoblockers (propranolol 10-40mg, oksprenolol 20mg 3 times per days, pindolol 5 mg 3 times on days, nadolol 20 mg 2 times per days, metaprolol 50 mg 2 times per days) sedative facilities

Sinus bradycardia ECG is criteria: correct rhythm less than 60 beats per 1 min sinus P waves interval of PQ >0,12 sec. Reasons: increase of parasympatic tonus, myocarditis, myxedema, hypothermia, mechanical icterus, syndrome of weakness of sinus knot.

Treatment to the atropine sulfate of 0,5-1 mg or 0,1% solution 1-2 times per day plathyphylinum hydrotartratum 0,5-1 mg or 0,2% solution 1-2 times per day extract of beladonnae -0,02 gr. 3-4 times per days izadrin 5-10 mg sublingval

The first aid is at the attacks of Morgani-Adams-Stoks to the atropine sulfate of 0,5-1 mg or 0,1% solution intravenously izadrin 5-10 mg sublingval (at a necessity to repeat) alupent for mg on 500 ml of izotonic soluble-sodium chloride electro-cardiostimulation (temporal or permanent)

Sinus arrhythmia ECG is criteria: P wave is sinus origin interval of PQ >0,12 s beats per 1 min the rhythm of heart is wrong with the difference of the longest and shortest interval of R-R 0.16 and anymore Treatment: does not require

a-normal sinus rhythm б- sinus tachycardia в- sinus bradycardia г- sinus arrhythmia

Arest of sinus knot EKG is criteria: complete asystole (absence of P and QRST) the pause of R-R does not make two ordinary intervals, and anymore. Treatment: how to the syndrome of Morgani-Adams-Stoks

Syndrome of weakness of sinus knot EKG is criteria: sinus bradycardia is expressed refuse of sinus knot with periods asystoles a duty of sinus bradycardia is with the attacks of auricle tachycardia, fibrilation (syndrome of «tachy-brady») a sino-atrial blockade is with extra contractions heart beats slow renewal of function of sine knot.

Treatment effective treatment does not exist medical therapy is at the attacks of Morgani-Adams-Stoks implantation of permanent electro- cardyostimulator

Extrasystolia  premature excitation and reduction of heart or his separate parts is as a result of increase of activity of hearths of ectopic automatism.

Auricle extrasystolia EKG of sign: premature reduction after which incomplete scray pause the P wave is changed, negative a complex QRS is not changed or aberrant. Treatment: it is not needed, at presence of clinical symptoms of b-blockers, verapamil, digitalis.

a- from the overhead departments of auricle б- from the middle departments of auricle в- from the lower departments of auricle г- is blocked auricle extrasystole

А-V- extrasystoles with simultaneous excitation of atriums and ventricles. EKG-signs: the P wave is not determined an extraordinary complex QRS is not extended incomplete scray pause

with previous excitation of ventricles EKG-signs: an extraordinary complex QRS is not extended P wave is after QRS complete scray pause Treatment: like, as at auricle extrasystolia.

Ventrical extrasystolia EKG of sign: complex QRS wide without a previous P wave complete scray pause Treatments need frequent monotopic, politopic, group and early as R/T extrasystoles.

a- sinistroventrical extrasystole b- dextraventrical extrasystole

Paroxysmal tachycardia Supraventricular tachycardia EKG is signs: frequency of reductions of atriums per 1 min. auricle complexes are preceded the complexes of QRS the complexes of QRS are not changed

Atrioventricular tachycardia EKG-signs: heart beats per 1 min. retrograde P wave (negative) after QRS or accumulates on him

a- auricle paroxysmal tachycardia б - atrioventricular tachycardia with previous excitation of ventricles в - atrioventricular tachycardia with simultaneous excitation of atriums and ventricles

Treatment of paroxysmal supraventricular tachycardia at leadthrough to the impulse through an a-v knot: massage of sinus carotic izoptin 0,25%-4ml obzіdan 0,1% - 5ml dіgoxin 0,025% - 1ml v/v cordaron 5% - 6 ml

Ventrical tachycardia EKG is criteria: - >140 hearts beats per 1 min. - the complexes of QRS are extended

 EKG is at paroxysmal ventrical tachycardia

Trembling and fibrilation of atriums EKG is criteria of trembling: frequency of auricle waves per 1 min. ( waves of f) EKG is criteria of fibrilation: waves of f wrong rhythm (different R-R) absence of P wave

Trembling and fibrilation of ventricles EKG is criteria: Sinus wave curve with frequent, rhythmic, wide and high waves, excitation of ventricles with frequency per 1 min. it is not possible to distinguish the elements of ventrical complex Treatment: electric cardioversion Dzh. At unefficiency the repeated cardioversion

a- trembling of ventricles b- blinking and fibrilation of ventricles

Sinoauricular blockade EKG is criteria: periodic fall of cardiac cycles increase of pause between the waves of Р-Р in 2 times

Inwardly auricle blockade EKG is criteria: increase of duration and breaking up of wave Р

Atrioventricular blockade І st permanent lengthening of interval of P-Q anymore as on 0,20s a- auricle form б- key form в- distal form blockade

А-v blockade ІІ ст the periodic stopping of leadthrough of impulse is from an auricle to the ventricles. There are three types: I type (Mobit I) is the gradual lengthening of interval of P-Q with the subsequent fall of QRST ( periods of Samoylov-Venkenbach) ІІ type (Mobit ІІ) is a fall of complexes of QRST without the gradual lengthening of interval of P-Q ІІІ type (Mobit ІІІ) of fall every second, or 2 and more complexes successively

А-v blockade ІІІ ст  complete autonomy of reduction of auricle and ventricles.The intervals of P-P and R-R are permanent, but R-R>P-P.

EKG is at the complete blockade of right bundle of bunch of Hiss

EKG is at the complete blockade of left bundle of bunch of Hiss

Syndrome of Wolf-Parkinson- White reduction of interval of P-Q (R) a presence in composition a complex QRS of additional wave of excitation is d-wave deformation and increase of duration of complex QRS discordant displacement of RS-T and change of polarity of wave T (inconstant sign)

EKG is at the syndrome of WPW

Conclusions It is necessary to know for successful diagnostics and treatment of arrhythmias: basic nosotropic mechanisms of their development · to own the methods of diagnostics of arrhythmias · to distinguish of high quality, potentially malignant and malignant arrhythmias · to understand algorithms diagnostic at the therapeutic going near the different types of arrhythmias