Inflammation p.1 SYLLABUS: Robbins and Cotran, Pathologic Basis of Disease, Chapter: Acute and Chronic Inflammation (all)

Slides:

Advertisements

Similar presentations

PRESS F1 FOR GUIDEANCE PATHOLOGY 1st Practical (Slides) MFSH 2003.

Advertisements

Periapical Granuloma Chronic inflammatory reaction in response to a pulpal infection with bacteria. All we can see here are tiny blue dots sprinkled.

CELL RESPONSE TO INJURY AND ADAPTIVE CELL REACTION

F 1+7/12 yrs CC: cough and mild cyanosis Diffuse pulmonary opacity.

Cell injury p.2 and cell death SYLLABUS: Robbins and Cotran, Pathologic Basis of Disease, Chapter: Cellular Responses to Stress and Toxic Insults: Adaptation,

Practical Pathology Inflammation Dr : Hala El-sayed Mahmoud Lecturer of pathology.

BY Dr Abiodun Mark Akanmode.. Identify the slide.

 Parasitic disease caused by several species of flatworm  Affects many in developing countries  a disease caused by parasitic worms. The organisms.

The lymphoreticular system is involved in the defence of the body against microorganisms and foreign substances – i.e. the immune response. Consists of.

Lab 1 : Inflammation & Repair Lab 1 : Inflammation & Repair Pathology Department of Microbiology and Immunology Dr. Manal Maher Hussein.

Inflammation. Acute inflammation The cardinal signs of inflammation are rubor (redness), calor (heat), tumor (swelling), dolor (pain), and loss of function.

INFLAMMATION Acute And Chronic. The cardinal signs of inflammation.

CELL INJURY & INFLAMMATION - II PRACTICAL 2 I - Acute Inflammation I - Acute Inflammation Foundation Block Pathology Dept, KSU.

GRANULOMATOUS INFLAMMATION

1 I N F L A M M A T I O N Assistant of professor Nechiporenko G. V.

Inflammation. Inflammation definition Inflammation – what for?

Liver and pancreas SYLLABUS: RBP(Robbins Basic Pathology) Chapters: The Liver and the Biliary Tract The Pancreas.

Female reproductive system 89Chronic cervicitis 302Naboth cysts 141Cervical squamous cell carcinoma 45Endometrial hyperplasia 129Endometrial carcinoma.

Kidney: Candidasis Lab 10, Case 1. Multifocal punctate lesions visible on the serosal surface (arrows) Don’t confuse these small yellow punctate lesions.

Lung Capillary lumen Type I pneumocyte Type I pneumocyte

Inflammation and repair Dr Shaesta Naseem

Respiratory practical Dr. Shaesta Naseem

Digestive pathology I. Chronic peptic ulcer From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig Deep loss of substance, often single, round or.

Respiratory system SYLLABUS: RBP(Robbins Basic Pathology) Chapters: The Lung and the Upper Respiratory Tract.

Respiratory practical block Pathology practical I and II Dr Shaesta Naseem Zaidi.

Revision respiratory practical block. A closer view of the lobar pneumonia demonstrates the distinct difference between the upper lobe and the consolidated.

Bronchial asthma By Dr. Abdelaty Shawky Assistant professor of pathology.

Pathology Inflammation-2 By Prof. Dr. SALAH FAYED.

Inflammation 5 Dr Heyam Awad FRCPath. topics to be covered in this lecture Outcome of acute inflammation. Morphology of acute inflammation. Chronic inflammation.

Acute inflammation Dr.Nazar Taher.

Inflammation and repair Pathology Practical class

Inflammation p.2 SYLLABUS: RBP(Robbins Basic Pathology) Chapter: Acute and Chronic Inflammation PBD (Pathologic Basis of Disease) Infectious Diseases.

classification of acute inflammation : according to the inflammatory fluid exudates, -Exudates are any fluid that filters from the circulatory system.

Microscopically, this abscess has a mixture of inflammatory cells, but the wall of the abscess is "organizing" with ingrowth of capillaries (filled with.

Gastrointestinal system SYLLABUS: RBP(Robbins Basic Pathology) Chapter: The Oral Cavity and the Gastrointestinal Tract.

INFLAMMATION 1. Cellulitis * Definition: Acute diffuse suppurative inflammation. * Cause: Streptococcus haemolyticus. The organism produces two enzymes:

INFLAMMATION By Dr. Gehan Mohamed Dr. Abdelaty Shawky 1.

Cardiovascular system

Infectious Diseases.

HISTOLOGY OF LARGE INTESTINE

Kyung Hee University, Seoul, Korea Conference LGI Conference Presented by Byeong-Joo Noh Supervised by Youn-Wha Kim Kyung Hee University, Seoul, Korea.

Chief Complaint Abdominal distension o/s) 내원 2 주전 Present Illness 52/F, 2014 년 9 월 Crohn's disease 진단받고 F/U 중인 자, 외래에서 Adalimumab(last : ) 복용.

Practical Pathology.

-Dr Sowmya Srinivas. INTRODUCTION  When circulating blood reaches the capillaries, part of its fluid content passes into the surrounding tissues as tissue.

Inflammation and repair

Diseases of Vermiform Appendix

Typhoid Fever and Acute Appendicitis: A Rare Association Not Yet Fully Formed - DOI: / Fig. 1. H&E staining postlaparoscopic appendectomy.

Pleomorphic adenoma –the tumour at the left side is white gray firm lobulated mass without hemorrhage or necrosis. note the normal lobulated gland at the.

Inflammation Case Presentation

Diagrammatic representation of vascular (vasodilatation) and exudative changes (humeral and cellular) involved in inflammatory process.

Inflammation I - Acute Inflammation

(I) IBD CROHN DISEASE (granulomatous colitis) ULCERATIVE COLITIS

GIT BLOCK PATHOLOGY PRACTICAL Dr Abdullah Basabein

Digestive pathology 2.

Acute Lobar Pneumonia (48)

Endocrine Pathology, Case 4

Immunopathological characterization of selected mouse models of inflammatory bowel disease: Comparison to human disease Yava L. Jones-Hall, Matthew B.

The cardinal signs of inflammation are rubor (redness), calor (heat), tumor (swelling), dolor (pain), and loss of function. Seen here is skin with erythema,

Dr. Samina Qamar FCPS Histopathology

AJKD Atlas of Renal Pathology: Acute Pyelonephritis

Here is chronic endometritis with lymphocytes and plasma cells in the endometrial stroma. In general, the inflammatory infiltrate of chronic inflammation.

Tumors of the nose, sinuses, and nasopharynx

Fulminant hepatic failure as the initial presentation of acute autoimmune hepatitis William R Kessler, Oscar W Cummings, George Eckert, Naga Chalasani,

Nodular Lymphoid Hyperplasia of the Lung: A Very Rare Disease Entity

Pathologic features of interstitial nephritis associated with inflammatory bowel disease. Pathologic features of interstitial nephritis associated with.

Acute Pyelonephritis American Journal of Kidney Diseases

Nonspecific interstitial pneumonia fibrosing pattern in a case of connective tissue disease. a) The alveolar walls are thickened by interstitial fibrosis.

Acute Inflammation.

Presentation transcript:

Inflammation p.1 SYLLABUS: Robbins and Cotran, Pathologic Basis of Disease, Chapter: Acute and Chronic Inflammation (all)

Inflammation p Lobar pneumonia 106 Appendicitis 155 Nasal polyp 216 Pericarditis (fribrionous) 50Follicular hyperplasia of the lymph node 119 Candidiasis (esophagus)

- alveoli filled with fibrinous exsudate (fibrin stain) Lobar pneumonia

-neutrophilic inflammatory infiltration of all the layers of appendix - possible mucosal ulcerations, inflammatory exsudate in the lumen of appendix, necrosis of the wall extending to serosa Appendicitis

-edematous mucosa with a loose stroma -often hyperplastic or cystic mucous glands - a variety of inflammatory cells, including neutrophils, eosinophils, and plasma cells with occasional clusters of lymphocytes Nasal polyp

- fibrinous exsudate (eosinophilic meshwork of threads and amorphous masses) covering pericardium Pericarditis (fibrinous inflammation)

Follicular hyperplasia of the lymph node (Reactive lymphoid hyperplasia, follicular pattern) -increase in number and size of germinal centers, due to expansion of follicular center, occasionally of mantle as well - follicular polarization retained

Follicular hyperplasia of the lymph node

Candidiasis (esophagus) Organisms: a mixture of budding yeast forms (blastoconidia) and pseudohyphae, tubular structures separated by narrowed areas Tissue reaction: usually purulent but may be minimal or granulomatous

Candidiasis (esophagus)