1. Specific Injuries Ankle Injuries: Sprains –Single most common injury in athletics caused by sudden inversion or eversion moments Inversion Sprains –Most common and result in injury to the lateral ligaments –Anterior talofibular ligament is injured with inversion, plantar flexion and internal rotation –Occasionally the force is great enough for an avulsion fracture to occur w/ the lateral malleolus

2. Severity of sprains is graded (1-3) With inversion sprains the foot is forcefully inverted or occurs when the foot comes into contact w/ uneven surfaces

4. Grade 1 Inversion Ankle Sprain –Etiology Occurs with inversion plantar flexion and adduction Causes stretching of the anterior talofibular ligament –Signs and Symptoms Mild pain and disability; weight bearing is minimally impaired; point tenderness over ligaments and no laxity –Management RICE for 1-2 days; limited weight bearing initially and then aggressive rehab Tape may provide some additional support Return to activity in 7-10 days

5. Grade 2 Inversion Ankle Sprain –Etiology Moderate inversion force causing great deal of disability with many days of lost time –Signs and Symptoms Feel or hear pop or snap; moderate pain w/ difficulty bearing weight; tenderness and edema Positive talar tilt and anterior drawer tests Possible tearing of the anterior talofibular and calcaneofibular ligaments –Management RICE for at least first 72 hours; X-ray exam to rule out fx; crutches 5-10 days, progressing to weight bearing

6. –Management (continued) Will require protective immobilization but begin ROM exercises early to aid in maintenance of motion and proprioception Taping will provide support during early stages of walking and running Long term disability will include chronic instability with injury recurrence potentially leading to joint degeneration Must continue to engage in rehab to prevent against re-injury

7. Grade 3 Inversion Ankle Sprain –Etiology Relatively uncommon but is extremely disabling Caused by significant force (inversion) resulting in spontaneous subluxation and reduction Causes damage to the anterior/posterior talofibular and calcaneofibular ligaments as well as the capsule –Signs and Symptoms Severe pain, swelling, hemarthrosis, discoloration Unable to bear weight Positive talar tilt and anterior drawer

8. –Management RICE, X-ray (physician may apply dorsiflexion splint for 3-6 weeks) Crutches are provided after cast removal Isometrics in cast; ROM, PRE and balance exercise once out Surgery may be warranted to stabilize ankle due to increased laxity and instability

9. Eversion Ankle Sprains - (Represent 5-10% of all ankle sprains) Etiology –Bony protection and ligament strength decreases likelihood of injury –Eversion force results in damage to deltoid ligament and possibly fx of the fibula –Deltoid can also be impinged and contused with inversion sprains

10. –Etiology (continued) Due to severity of injury, it may take longer to heal Foot that is pronated, hypermobile or has a depressed medial longitudinal arch is more predisposed to eversion sprains –Signs and Symptoms Pain may be severe; unable to bear weight; and pain with abduction and adduction but not direct pressure on bottom of foot –Management RICE; X-ray to rule out fx; no weight bearing initially; posterior splint tape; NSAID’s Follows the same course of treatment as inversion sprains Grade 2 or higher will present with considerable instability and may cause weakness in medial longitudinal arch resulting in excessive pronation or fallen arch

11. Syndesmotic Sprain –Etiology Injury to the distal tibiofemoral joint (anterior/posterior tibiofibular ligament) Torn w/ increased external rotation or dorsiflexion Injured in conjunction w/ medial and lateral ligaments –Signs and Symptoms Severe pain, loss of function; passive external rotation and dorsiflexion cause pain Pain is usually anterolaterally located –Management Difficult to treat and may requires months of treatment Same course of treatment as other sprains, however, immobilization and total rehab may be longer

12. Ankle Fractures/Dislocations –Etiology Number of mechanisms –Signs and Symptoms Swelling and pain may be extreme with possible deformity –Management RICE to control hemorrhaging and swelling Once swelling is reduced, a walking cast or brace may be applied, w/ immobilization lasting 6-8 weeks

15. Osteochondritis Dissecans –Etiology Occur in superior medial articular surface of the talar dome One or several fragments of articular cartilage, w/ underlying subchondral bone partially or completely detached and moving within the joint space Mechanism may be single trauma or repeated traumas –Signs and Symptoms May be a complaint of pain and effusion with signs of atrophy May also be catching, locking, or giving way

16. Osteochondritis Dissecans –Management Diagnosis through X-ray or MRI Incomplete and non-displaced injuries can be immobilized with early motion and delayed weight bearing If fragments are displaced, surgery is necessary Surgery will minimize risk of nonunion

17. Acute Achilles Strain –Etiology Common in sports and often occurs with sprains or excessive dorsiflexion –Sign and Symptoms Pain may be mild to severe Most severe injury is partial/complete avulsion or rupturing of the Achilles –Management Pressure and RICE should be applied After hemorrhaging has subsided an elastic wrap should continue to be applied Conservative treatment should be used as Achilles problems generally become chronic A heel lift should be used and stretching and strengthening should begin soon

18. Achilles Tendinitis –Etiology Inflammatory condition involving tendon, sheath or paratenon Tendon is overloaded due to extensive stress Presents with gradual onset and worsens with continued use Decreased flexibility exacerbates condition –Signs and Symptoms Generalized pain and stiffness, localized proximal to calcaneal insertion Warm and painful with palpation, also presents with thickening May limit strength May progress to morning stiffness

19. Crepitus with active plantar flexion and passive dorsiflexion Chronic inflammation may lead to thickening –Management Resistant to quick resolution due to slow healing nature of tendon Must reduce stress on tendon, address structural faults (orthotics, mechanics, flexibility) Use anti-inflammatory modalities and medications Cross friction massage may be helpful in breaking down adhesions Strengthening must progress slowly in order to not aggravate the tendon

21. Achilles Tendon Rupture –Etiology Occurs w/ sudden stop and go; forceful plantar flexion w/ knee moving into full extension Commonly seen in athletes > 30 years old Generally has history of chronic inflammation –Signs and Symptoms Sudden snap (kick in the leg) w/ immediate pain which rapidly subsides Point tenderness, swelling, discoloration; decreased ROM Obvious indentation and positive Thompson test Occurs 2-6 cm proximal the calcaneal insertion

23. Achilles Tendon Rupture (continued) –Management Usual management involves surgical repair for serious injuries (return of 75-80% of function) Non-operative treatment consists of RICE, NSAID’s, analgesics, and a non-weight bearing cast for 6 weeks, followed up by a walking cast for 2 weeks (75-90% return to normal function) Rehabilitation lasts about 6 months and consists of ROM, PRE and wearing a 2cm heel lift in both shoes

24. Peroneal Tendon Subluxation/Dislocation –Etiology Occurs in sports with dynamic forces being applied to the ankle May also be caused by dramatic blow to posterior lateral malleolus, or moderate/severe inversion ankle sprain resulting in tearing of peroneal retinaculum –Signs and Symptoms Complain of snapping in and out of groove with activity Eversion against manual resistance replicates subluxation Recurrent pain, snapping and instability Present with ecchymosis, edema, tenderness, and crepitus over the tendon

25. Peroneal Subluxation (continued) –Management Conservative approach should be used first, including compression with felt horseshoe Reinforce compression pad with rigid plastic or plaster until acute signs have subsided RICE, NSAID’s and analgesics Conservative treatment time 5-6 weeks followed by gradual rehab program Surgery if conservative plan fails

26. Anterior Tibialis Tendinitis –Etiology Commonly occurs after extensive down hill running –Signs and Symptoms Point tenderness over anterior tibialis tendon –Management Rest or at least decrease running time and distance, avoid hills In more serious cases, ice & stretch before and after running to reduce symptoms Daily strengthening should be conducted Oral antiinflammatory medication may be required

27. Posterior Tibialis Tendinitis –Etiology Common overuse condition in runners with foot hypermobility or over pronation Repetitive microtrauma –Signs and Symptoms Pain and swelling in area of medial malleolus Edema, point tenderness and increased pain during resistive inversion and plantar flexion –Management Initially, RICE, NSAID’s and analgesics Non-weight bearing cast w/ foot in inversion may be used Correct problem of over pronation with taping or orthotic

28. Peroneal Tendinitis –Etiology Not common, but can be found with athletes that have pes cavus due to excessive supination placing stress on peroneal tendon –Signs and Symptoms Pain behind lateral malleolus during push-off or when rising on ball of foot Pain along distolateral aspect of calcaneus and beneath the cuboid –Management RICE, NSAID’s, elastic taping, appropriate warm- up and flexibility exercises LowDye taping or orthotics to help support foot

30. Shin Contusion –Etiology Direct blow to lower leg (impacting periosteum anteriorly) –Signs and Symptoms Intense pain, rapidly forming hematoma w/ jelly like consistency –Management RICE, NSAID’s and analgesics as needed Maintaining compression for hematoma (which may need to aspirated) Fit with doughnut pad and orthoplast shell for protection If not managed appropriately may develop into osteomyelitis (deterioration of bone)

31. Muscle Contusions –Etiology Contusion of leg, particularly in the region of the gastrocnemius –Signs and Symptoms Bruise may develop, pain, weakness and partial loss of limb function Palpation will reveal hard, rigid, inflexible area due to internal hemorrhaging and muscle guarding –Management Stretch to prevent spasm; apply cold compression and ice If superficial therapy and massage do not return athlete to normal in 2-3 days, ultrasound would be indicated Wrap or tape will help to stabilize the area

32. Leg Cramps and Spasms - Sudden, violent, involuntary contraction, either clonic (intermittent) or tonic (sustained) in nature –Etiology Difficult to determine; fatigue, loss of fluids, electrolyte imbalance, inadequate reciprocal muscle coordination –Signs and Symptoms Cramping with pain and contraction of calf muscle –Management Try to help athlete relax to relieve cramp Firm grasp of cramping muscle with gentle stretching will relieve acute spasm Ice will also aid in reducing spasm If recurrent may be fatigue or water/electrolyte imbalance

33. Gastrocnemius Strain –Etiology Susceptible to strain near musculotendinous attachment Caused by quick start or stop, jumping –Signs and Symptoms Depending on grade, variable amount of swelling, pain, muscle disability May feel like being “hit in leg with a stick” Edema, point tenderness and functional loss of strength –Management RICE, NSAID’s and analgesics as needed Grade 1 should apply gentle stretch after cooling Weight bearing as tolerated; use heel wedge to reduce calf stretching while walking Gradual rehab program should be instituted

35. Acute Leg Fractures –Etiology Fibula has highest incidence of fracture, occurring primarily in the middle third Tibial fractures occur predominantly in the lower third Result of direct blow or indirect trauma –Signs and Symptoms Pain, swelling, soft tissue insult Leg will appear hard and swollen (Volkman’s contracture) –Management X-ray, reduction, casting up to 6 weeks depending on the extent of injury

37. Medial Tibial Stress Syndrome (Shin Splints) –Etiology Pain in anterior portion of shin Catch all for stress fractures, muscle strains, chronic anterior compartment syndrome Accounts for 10-15% of all running injuries, 60% of leg pain in athletes Caused by repetitive microtrauma Weak muscles, improper footwear, training errors, varus foot, tight heel cord, hypermobile or pronated feet and even forefoot supination can contribute to MTSS May also involve, stress fractures or exertional compartment syndrome

38. Shin Splints (continued) –Signs and Symptoms Four grades of pain –Pain after activity –Pain before and after activity and not affecting performance –Pain before, during and after activity, affecting performance –Pain so severe, performance is impossible –Management Physician referral for X-rays and bone scan Activity modification Correction of abnormal biomechanics Ice massage to reduce pain and inflammation Flexibility program for gastroc-soleus complex Arch taping and or orthotics

39. Compartment Syndrome –Etiology Rare acute traumatic syndrome due to direct blow or excessive exercise –Signs and Symptoms Excessive swelling compresses muscles, blood supply and nerves Increase in fluid accumulation could lead to permanent disability Chronic cases appear as gradual build-up that dissipates following activity; generally bilateral and becomes predictable; can remain elevated producing ischemia and pain or ache w/ rare neurological involvement; increased pressure involvement Weakness with foot and toe extension and occasionally numbness in dorsal region of foot

40. Compartment Syndrome (continued) –Management If severe acute or chronic case, may present as medical emergency that requires surgery to reduce pressure or release fascia RICE, NSAID’s and analgesics as needed Surgical release is generally used in recurrent conditions Return to activity after surgery - light activity- 10 days later

41. Stress Fracture of Tibia or Fibula –Etiology Common overuse condition, particularly in those with structural and biomechanical insufficiencies Runners tends to develop in lower third of lower leg (dancers middle third) Often occur in unconditioned, non-experienced individuals Often training errors are involved Component of female athlete triad –Signs and Symptoms Pain more intense after exercise than before Point tenderness; difficult to discern bone and soft tissue pain Bone scan results (stress fracture vs. periostitis)

42. Management –Discontinue stress inducing activity 14 days –Use crutches for walking –Weight bearing may return when pain subsides –Cycling before running –After pain free for 2 weeks athlete can gradually return to running –Biomechanics must be addressed