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Common Pediatric Lower Limb Disorders
Dr.Kholoud Al-Zain Assistant Professors Consultant Pediatric Orthopedic Surgeons
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Topics Growing pain Limping In-toeing & out-toeing
Leg length inequality Genu varus & valgus Proximal tibia vara Club foot L.L deformities in C.P patients
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1) Growing Pain
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Leg Aches What is leg aches? “Growing pain” Benign
In 15 – 30 % of normal children F > M Unknown cause No functional disability, or limping Resolves spontaneously, over several years Growth spert
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Leg Aches Clinical features diagnosis by exclusion H/O: O/E:
At long bones of L.L (Bil) Dull aching, poorly localized Can be without activity At night Of long duration (months) Responds to analgesia O/E: Long bone tenderness nonspecific, large area, or none Normal joints motion
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Screening Examination
Leg Aches What is leg aches? Growing pain Benign No functional disability Resolves spontaneously Unknown cause Clinical features Diagnosis by exclusion History Screening Examination Tenderness Joint Motion
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Leg Aches D.D from serious problems, mainly tumor: Osteoid osteoma
Osteosarcoma Ewing sarcoma Leukemia SCA Subacute O.M
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Leg Aches Management: Reassurance Symptomatic: Analgesia (oral, local)
Rest Massage
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Leg Aches Differential Diagnosis from serious problems mainly tumor
Osteoid osteoma Osteosarcoma Ewing sarcoma Management Symptomatic Reassurance
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2) Limping
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Limping Definition It is: An abnormal gait Due to:
Pain Weakness (general or nerve or muscle) Or deformity (bone or joint) In one or both limbs
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Limping Diagnosis by: History age of onset Examination:
Gait in the clinic hallway Is it: Above pelvis Back (scoliosis) Below pelvis Hips, knees, ankles, & feet Neuro.Vascular
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Abnormal gait due to pain, weakness or deformity
Limp Abnormal gait due to pain, weakness or deformity Evaluation History (Mainly age of onset) Observation Evaluate the limp by studying the child’s gait while the child walks in the clinic hallway
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Limping Management: Generalization can’t be made
Treatment of the cause:
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3) In-toing & Out-toing
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In-toeing and Out-toeing
Terminology: Version: Describes normal variations of limb rotation It may be exaggerated Torsion: Describes abnormal limb rotation Internal or external
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In-toeing and Out-toeing
Evaluation: History Screening examination (head to toe) Foot-progression-angle Asses rotational profile: Hips Hips Rotational Profile Tibiae Foot Thigh Axis Feet Heal Bisector Line
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In-toeing and Out-toeing
Special tests Foot Propagation Angle (N= -5 to +15)
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In-toeing and Out-toeing
Special tests hips rotational profile
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In-toeing and Out-toeing
Special tests Foot Thigh Axis
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In-toeing and Out-toeing
Special tests Foot Thigh Axis (N= 10°-15°)
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In-toeing and Out-toeing
Special tests hips rotational profile
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A) In-toeing
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1) In-toeing: Femoral Anteversion
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2) In-toeing: Tibial Torsion
Supine position Sitting position
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2) In-toeing: Tibial Torsion
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2) In-toeing: Tibial Torsion
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3) In-toeing: Forefoot Adduction
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4) In-toeing: Adducted Big Toe
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D) In-toeing: Adducted Big Toe
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A) In-toeing Evaluation: History, Screening examination,
Asses rotational profile.
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B) Out-toeing
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B) Out-toeing Evaluation: History, Screening examination,
Asses rotational profile.
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In-toeing and Out-toeing
Management principles: Establishing correct diagnosis Allow spontaneous correction (observational management) Control child’s walking, sitting or sleeping is extremely difficult and frustrating Shoe wedges or inserts are ineffective Bracing with twister cables limits child’s activities Night splints have no long term benefit
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In-toeing and Out-toeing
Annual clinic F/U asses degree of deformity Femoral anti-version sit cross legged Tibial torsion spontaneous improvement Forefoot adduction anti-version shoes, or proper shoes reversal Adducted big toe spontaneous improvement
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In-toeing and Out-toeing
Usually does not improve spontaneously Will need an operation: After the age 8y Foot propagation angle >30°
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In-toeing and Out-toeing
Operative correction indicated for children: (> 8) years of age With significant cosmetic and functional deformity <1%
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4) Limb Length Inequality
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Limb Length Inequality
True vs. apparent Etiology:
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH Developmental as Blount’s
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH Developmental as Blount’s Traumatic as oblique # (short), or multifragmented (long)
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH Developmental as Blount’s Traumatic as oblique # (short), or multifragmented (long) Infection stunted growth or dissolved part of bone
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH Developmental as Blount’s Traumatic as oblique # (short), or multifragmented (long) Infection stunted growth or dissolved part of bone Metabolic as rickets
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH Developmental as Blount’s Traumatic as oblique # (short), or multifragmented (long) Infection stunted growth or dissolved part of bone Metabolic as rickets (unilateral) Tumor affecting physis
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Limb Length Inequality
True and apparent Etiology Congenital Developmental Traumatic Infection Metabolic Tumor
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Limb Length Inequality
Adverse effects & clinical picture: Gait disturbance Equinus deformity Pain: back, leg Scoliosis (secondary)
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Limb Length Inequality
Evaluation: Screening examination Clinical measures of discrepancy Imaging methods (Centigram)
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Limb Length Inequality
Clinical measures of discrepancy: Measuring tape Giliazi test
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Limb Length Inequality
Adverse effects Gait disturbance Equinous deformity Back pain Scoliosis Evaluation Screening examination Clinical measures of discrepancy Imaging methods (Centigram)
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Limb Length Inequality
Management depends on the severity (>2cm): For shorter limb: Shoe raise Bone lengthening For longer limb: Epiphysiodesis (temporary or permanent) Bone shortening
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Limb Length Inequality
Management principles Severity Lifts Shortening Epiphysiodesis Lengthening
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5) Genu Varus & Valgus
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Genu Varum and Genu Valgum
Definition: Bow legs Knock knees
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Genu Varum and Genu Valgum
Etiology: Physiologic Pathologic
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Genu Varum and Genu Valgum
Note: Physiological is usually bilateral. Pathological can be unilateral.
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Genu Varum and Genu Valgum
Definitions Bow legs Knock knees Etiology Physiologic Pathologic
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Genu Varum and Genu Valgum
Evaluation History Examination (signs of Rickets) Laboratory Re take the rickets picture
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Genu Varum and Genu Valgum
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Genu Varum and Genu Valgum
Evaluation: Imaging Put mechanical axsis
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Genu Varum and Genu Valgum
Evaluation Imaging
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Genu Varum and Genu Valgum
Management principles: Non-operative: Physiological usually Pathological must treat underlying cause, as rickets Epiphysiodesis Corrective osteotomies
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Genu Varum and Genu Valgum
Management principles: Non-operative ? Epiphysiodesis Corrective osteotomies
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6) Proximal Tibia Vara
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Proximal Tibia Vara “Blount disease”: damage of tibial proximal medial growth plate of unknown cause Usually: Overweight Dark skinned
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Proximal Tibia Vara Types:
Infantile < 3y of age, & usually early walkers Juvenile y, combination Adolescent > 10y, & usually unilateral Is it physis defect
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Proximal Tibia Vara Classification (radiological): XR (M.D.A)
M.D.A < 11° observe closely M.D.A > 15° operate
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Proximal Tibia Vara MRI is mandatory: When: Why? Sever cases
Recurrence Why? Is it physis defect
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Tibia Vara Blount disease
Damage of proximal medial tibial growth plate of unknown cause MRI is mandatory …. Why? Is it physis defect
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Proximal Tibia Vara Bilateral Unilateral
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7) Club Foot
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Clubfoot Etiology Postural fully correctable
Idiopathic (CTEV) partially correctable Secondary (Spina Bifida) rigid deformity
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Clubfoot Etiology Postural Idiopathic (CTEV) Secondary (Spina Bifida)
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Clubfoot Diagnosis by exclusion: Neurological lesion that can cause:
As Spina Bifida, excluded by spine XR Abnormalities that can explain: As Arthrogryposis With other congenital anomalies: As tibial hemimelia, exclude by XR Syndromatic clubfoot: As: Larsen’s Syndrome Amniotic Band Syndrome Why this seqwence Why #2 & #4 are not the same
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Clubfoot Diagnosis by exclusion Exclude
Neurological lesion that can cause the deformity “Spina Bifida” (excluded by spine x-rays) Other abnormalities that can explain the deformity “Arthrogryposis, Myelodysplasia” Presence of concomitant congenital anomalies “Proximal femoral focal deficiency” Syndromatic clubfoot “Larsen’s syndrome, Amniotic band Syndrome”
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Clubfoot Clinical examination Characteristic Deformity : Hind foot:
Equinus (Ankle joint) Varus (Subtalar joint) Mid & fore foot: Forefoot Adduction Cavus
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Clubfoot
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Clubfoot Clinical examination: Deformities don’t prevent walking
Calf muscles wasting Internal torsion of the leg Foot is smaller in unilateral affection Callosities at abnormal pressure areas Short Achilles tendon Heel is high and small No creases behind Heel Abnormal crease in middle of the foot
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A cosmetically pleasing appearance is also an important goal sought by
Clubfoot Management: The goal of treatment for is to obtain a foot that is plantigrade, functional, painless, and stable over time A cosmetically pleasing appearance is also an important goal sought by surgeon and family
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Clubfoot Manipulation and serial casts:
Validity up to 12 months soft tissue becomes more tight Technique “Ponseti” 3 stages, weekly basis (usually by 6-8w)
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Clubfoot Manipulation and serial casts:
Maintaining correction “Dennis Brown Splint” 3-4y old
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Clubfoot Manipulation and serial casts:
Follow up watch and avoid recurrence, till 9y old Avoid false correction by going in sequence When to stop ? not improving, pressure ulcers All pictures put better / Why only to 12 m / when stop
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Clubfoot Manipulation and serial casts:
Validity up to 12 months soft tissue becomes more tight Technique “Ponseti” 3 stages, weekly basis (usually by 6-8w) Maintaining correction “Dennis Brown Splint” 3-4y old Follow up watch and avoid recurrence, till 9y old Avoid false correction by going in sequence When to stop ? not improving, pressure ulcers All pictures put better / Why only to 12 m / when stop
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Manipulation and serial casts
Clubfoot Manipulation and serial casts Validity, up to 12 months ! Technique “Ponseti” Avoid false correction When to stop ? Maintaining the correction Follow up to watch and avoid recurrence
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Clubfoot Indications of surgical treatment:
Late presentation (>12 months of age) Complementary to conservative treatment (residual forefoot adduction) Failure of conservative treatment Recurrence after conservative treatment
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Clubfoot Types of surgery: Soft tissue
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Clubfoot Types of surgery: Bony New pictures
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Clubfoot Types of surgery: If sever, rigid, and in an older child
New pictures
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Clubfoot Indications of surgical treatment Types of surgery
Late presentation, after 12 months of age ! Complementary to conservative treatment Failure of conservative treatment Recurrence after conservative treatment Types of surgery Soft tissue Bony Salvage
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Clubfoot Types of surgery:
If sever, rigid, and in an older child (salvage)
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Clubfoot
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8) L.L Deformities in C.P Patients
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Lower Limb Deformities in CP Child
C.P is a non-progressive brain insult that occurred during the peri-natal period. Causes skeletal muscles imbalance that affects joint’s movements. Can be associated with: Mental retardation (various degrees) Hydrocephalus and V.P shunt Convulsions Its not-un-common
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Lower Limb Deformities in CP Child
Physiological classification: Spastic Athetosis Ataxia Rigidity Mixed Topographic classification: Monoplegia Diplegia Paraplegia Hemiplegia Bilateral hemiplegia Triplegia Quadriplegia or tetraplegia
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Lower Limb Deformities in CP Child
Hip Flexion Adduction Internal rotation Knee Ankle Equinus Varus or valgus Gait Intoeing Scissoring
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Lower Limb Deformities in CP Child
Assessment: Hips
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Lower Limb Deformities in CP Child
Assessment: Knees
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Lower Limb Deformities in CP Child
Assessment: Ankles
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Lower Limb Deformities in CP Child
Assessment: Hips Knees Ankles New pictures
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Lower Limb Deformities in CP Child
Management is multidisciplinary: Parents education Pediatric neurology diagnosis, F/U, treat fits P.T (home & center) joints R.O.M, gait training Orthotics maintain correction, aid in gait Social / Government aid Others: Neurosurgery (V.P shunt), Ophthalmology (eyes sequent), …etc.
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Lower Limb Deformities in CP Child
Indications of Orthopedic surgery: Sever contractures preventing P.T P.T plateaued due to contractures Perennial hygiene (sever hips adduction) In a non-walker to sit confortable in wheelchair Prevent: Neuropathic skin ulceration (as feet) Joint dislocation (as hip)
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Lower Limb Deformities in CP Child
Options of Surgery: Tenotomy Tenoplasty Muscle lengthening Neurectomy Tendon Transfer Bony surgery Osteotomy/Fusion
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Lower Limb Deformities in CP Child
Management principles Multidisciplinary Options of Surgery Neurectomy Tenotomy Tenoplasty Muscle lengthening Tendon Transfer Bony surgery Osteotomy/Fusion
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Remember …
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Conclusion Leg aches of exclusion, D.D, long bone, activity ±, symptomatic treatment Limping due (pain- week- deformed), uni or bi, , proper assessment In & out toeing torsion vs. version, proper assessment to know cause & level, mainly observe, operate >8y old L.L.I true vs. apparent, proper assessment to know cause & level, if not treated, >2cm, options of treat Genu varus & valgus physiological vs. pathological, rickets, when operate Blount medial physis, D.M.A, MRI, types, surgery CTEV 3 types, diagnosis of exclusion, clinical picture, Ponseti, better to avoid surgery L.L in C.P muscle imbalance, of different types, proper pt assessment, PT ± surgery
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