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Published byTracy Boyd Modified over 8 years ago
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Cervical Instability
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Normal Anatomy Normal stability of any joint is made of 2 aspects – Static Stabilisers – osseous configuration, capsules and ligaments – Dynamic Stabilisers- muscle function through dynamic ligament tension, force couples, joint compression and/or neuromuscular control Approximately 50% of rotation occurs at C1/2 Ligaments provide the primary source of stability Vast amount of neurological and vascular structures
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Atlantoaxial Joint Transverse Ligament of the Atlas
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Atlantoaxial Joint Tectorial Membrane
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Atlantoaxial Joint Alar Ligament and Transverse Ligament
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Pathophysiology Excessive movement at the upper cervical spine Can be the result of bony fracture, ligamentous laxity or rupture or neuromuscular deficits Can result in pain, neurological or vascular compromise
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Mechanism Of Injury Traumatic – Whiplash – Fractures, Dislocations – Surgery Systemic – Upper Respiratory Infection Congenital – Down Syndrome – RA – Os Odontoideum – Klippel-Feil Syndrome – Hypermobility Syndrome
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Associated Pathologies Cervical Artery Dysfunction Cervical Myelopathy Cervicogenic Headaches
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Subjective History of trauma or congenital/systemic disease Neck pain Intolerance to prolonged positions Feeling need to support the head Sharp pain or catch with movements
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Subjective Signs of neurological or vascular compromise – Drop attacks – Facial or lip paraesthesia – Bilateral or quadrilateral symptoms – Nystagmus – Dizziness – Blurred vision – Metallic taste in mouth – Lump back of throat – Think neuro, think vascular, think cranial nerves, think CNS
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Objective Based on Subjective History May not be appropriate in some cases Start with cranial nerve and BP testing
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Objective Serious (Static Stability) Reduced sensation Reduced power Reflex changes Cranial Nerve Changes Significant muscle spasm Reluctance to move Non Serious (Neuromuscular) Sensorimotor changes – Smooth Pursuit Neck Torsion – Saccadic Eye Testing – Joint Position Error Full range of movement with painful stretching end of range Painful catch/ unsmooth movements Increased joint play
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Special Tests Sharp Purser – Sitting relocation of C1 on C2 Alar Ligament Testing – Supine testing of rotation and lateral flexion of Upper Cx with fixation of C2 Transverse ligament Testing – Supine with fingers around patients head and between occiput and C2. Lift head and C1 anteriorly and hold for 20-30 mins
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Further Investigation MRI X-ray Open Mouth X-Ray
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Management Referral to a specialist if signs of neurological or vascular compromise Conservative management for those with congenital or neuromuscular reasons for instability Surgery nearly always for traumatic instability
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Conservative - Management Sensorimotor rehabilitation Cervical and scapular rehabilitation Manual Therapy to Thoracic Spine Acupuncture for pain relief
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Surgical - Management Depends on pathology causing instability
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