1. Prepared By Group A

2. Group Members

3. Definition APH  Bleeding from the genital tract in pregnancy between 20 to 24 week’s gestation and the onset of labour.  It affects 4% of all pregnancies.  It is associated with increased risks of fetal and maternal morbidity and mortality.

4. Causes  Placental: Abruptio placenta. Placenta previa.  Non-placental: Vasa previa. Bloody show. Trauma. Uterine rupture. Cervicitis. Carcinoma. Idiopathic.

5. ABRUPTIO PLACENTA

6. Introduction Definition: It is the separation of the placenta from its site of implantation before delivery of the fetus.  Incidence:  1 in 200 deliveries.

7. Types of Placental Abruption  Revealed placental abruption: causes vaginal bleeding.  Concealed placental abruption: internal bleeding

8. Risk Factors  Increased age & parity.  Hypertensive disorders.  Preterm ruptured membranes.  Multiple gestation.  Polyhydramnios.  Smoking.  Cocaine use.  Uterine fibroid.  Trauma

9. Clinical Presentation  Vaginal bleeding.  Uterine tenderness or back pain.  Fetal distress.  High frequency contractions.  Uterine hyper tonus.  IUFD.  Nausea and vomiting

10. Classification  Asymptomatic, External vaginal bleeding Uterine tetany and tenderness may be present No signs of maternal shock No evidence of fetal distress Grade 0Grade 1

11.  External vaginal bleeding may or may not be present  Uterine tender and tentany  No signs of maternal shock  Signs of fetal distress present  External bleeding may or may not be present  Marked uterine tetany  Maternal shock  Fetal death or distress  Coagulopathy in 30% of the cases Grade 2.Grade 3. Cont.

12. Diagnosis  Physical examination to determine the uterine rigidity or tenderness.  Abdominal Ultrasound  CBC  Fetal Monitoring  Pelvic Exam  Vaginal Ultrasound

13. Management  Fetal Monitoring for the fetal heart rate  Blood Transfusion if its need  Administer Rh immune globulin if the patient is Rh-  Vaginal Delivery  Blood plasma replacement to maintain fibrinogen level  Cesarean Delivery is often necessary for fetal and maternal stabilization

14. Prevention  Do not drink any alcohol such as beer and wine  Do not smoke or use recreational drugs during pregnancy  Get early and regular prenatal care  Early recognizing and managing conditions in the mother such as diabetes and high blood pressure also decrease the risk of placental abruption

15. Complications  Hypovolemic shock  DIC (Disseminated intravascular coagulation)  Renal failure.  Death.  Uterine rupture  Hypoxia.  Brain Damage  IUGR.  stillbirth  Anemia MaternalFetal

16. PLACENTA PREVIA

17. Introduction Definition:  The presence of placental tissue overlying or proximate to the internal cervical os after viability. Incidence:  Complicates approximately 1 in 300 pregnancies.

18. Predisposing factors  Multiparty  Increased maternal age  Previous placenta previa, recurrence rate 4-8%  Multiple gestation  Previous cesarean section  Uterine anomalies  Maternal smoking

19. Placenta praevia Grades:  Grade 1: the placental edge is in the lower uterine segment but does not reach the internal os (low implantation).  Grade 2: the placental edge reaches the internal os but does not cover it.  Grade 3: the placenta covers the internal os when it is close and is asymmetrically situated (partial).  Grade 4: the placenta covers the internal os and is centrally situated (complete)

20. Clinical presentation  Bright red vaginal bleeding without pain  Premature contractions  Baby is breech in transverse position

21. Diagnosis  History taking  Abdominal examination  Leopold's Maneuvers  Fetal Heart Monitoring  Vaginal Examination is avoiding

22. Management  Admit to hospital  Corticosteroids  Blood volume replacement to maintain blood pressure  Avoiding intercourse

23. Complications of Placenta praevia  APH  PPH  Increase risk of puerperal sepsis  Malpresentation; breech, oblique, transverse.  IUGR  Premature delivery  Death Maternal Fetal

24. VASA PREVIA

25. Introduction  Is a complication of pregnancy in which babies blood vessels cross or run near the internal opening of the uterus  These vessels are at risk of rupture when the supporting membranes rupture.  The term of Vasa previa is derived from the Latin word  Vasa means Vessel  Pre means Before  Via means Way  The incidence is 1 in 2000 – 3000 deliveries.

26. Associated Conditions  Low-lying placenta.  Bilobed placenta.  Multi-lobed placenta.  Succenturiate-lobed placenta.  Multiple pregnancies.  IVF.

27. Clinical Presentations  Painless vaginal bleeding  Rupture of membranes  Fetal bradycardia

28. Diagnosis  The diagnosis of vasa previa is considered if vaginal bleeding occurs upon rupture of the membranes.  Fetal hemoglobin test  Concomitant fetal heart rate abnormalities.  Ultrasound

29. Antenatal Management  Consider hospitalization in the third trimester to provide proximity to facilities for emergency cesarean delivery.  Fetal surveillance to detect compression of vessels.  Antenatal corticosteroids to promote lung maturity.

30. Antepartum Management  Immediate C/S.  Avoid amniotomy as the risk of fetal mortality is 60-70% with rupture of the membranes.

31. UTERINE RUPTURE

32.  Reported in 0.03-0.08% of all delivering women, but 0.3-1.7% among women with a history of a uterine scar (from a C/S for example)  13% of all uterine ruptures occur outside the hospital  The most common maternal morbidity is hemorrhage  Fetal morbidity is more common with extrusion

33. Cont.  Classic presentation includes vaginal bleeding, pain, cessation of contractions, absence/ deterioration of fetal heart rate, loss of station of the fetal head from the birth canal, easily palpable fetal parts, and profound maternal tachycardia and hypotension.  Patients with a prior uterine scar should be advised to come to the hospital for evaluation of new onset contractions, abdominal pain, or vaginal bleeding.

34. Risk Factors  The most common risk factor is a previous C/S or uterine surgery.  Placenta previa  Plastenta accreta.  Trauma.

35. Presentation  Sudden severe fetal heart decelerations.  Abdominal pain ( <10%).  Excessive vaginal bleeding  Rapid heart rate of mother  Lowe blood pressure  Cessation of uterine contractions.

36. Prognosis  Fetal death 50-75%.  Maternal mortality is high if not diagnosed & managed promptly.  Maternal morbidity: hemorrhage & infection.

37. Management  stabilization of maternal hemodynamics.  Blood transfusion  Prompt C/S with either repair of the uterine defect or hysterectomy.  Antibiotics.

38. Complications  CPD.  Abnormal presentation.  Unusual fetal enlargement (hydrocephalus).  Difficult forceps.  Breech extraction.  Internal podalic version. Labor complications:Delivery complications:

39. Reference  https://en.wikipedia.org/wiki/Antepartum_haemorrhage https://en.wikipedia.org/wiki/Antepartum_haemorrhage  https://www.glowm.com/pdf/AIP%20Chap5%20APH.pdf https://www.glowm.com/pdf/AIP%20Chap5%20APH.pdf

40. Thank You