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Intestinal Obstruction
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Introduction Intestinal obstruction is said to have occurred when the peristaltic movements of the intestines stop and the passage of food through the lumen stops.
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Pathophysiology Bowel distends – altered motility – below the obstruction has normal motility – absorption until it is empty- later contracts and becomes immobile. Proximal to the obstruction peristalsis increases to overcome obstruction – later paralysed - intestine distends Causes of gas : aerobic and anaerobic organisms gas production – O2 and CO2 absorbed – nitrogen and hydrogen sulphide remains in the gut
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Causes of Fluid : digestive juices – absorption of fluid stops – dehydration and electrolyte loss – decreased intake – lack of absorption - vomiting - sequestration of fluid in bowel lumen. Stangulation : - blood supply gets cut off in a few cases. – hernial orifices adhesions , bands – interruption of mesenteric flow like volvulus, intussusception, - rising intraluinal pressure ( closed loop obstruction) – mesenteric vascular obstruction.
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Classification of Intestinal Obstruction
Dynamic Adynamic
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Dynamic Obstruction Peristalsis is working against a mechanical obstruction Extramural : intraperitoneal bands and adhesions, hernias, volvulus, intussusception Intramural : malignant or inflammatory strictures Intraluminal : impacted faeces, foreign bodies, bezoar, gall stones Trichobezoar intussusception Volvulus adhesion band maligstricture
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Adynamic Obstruction Two forms Peristalsis absent (Paralytic ileus)
Peristalsis present; but in a nonpropulsive form (mesenteric vascular occlusion or pseudo-obstruction) In both types a mechanical element is absent
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Dynamic obstruction Abdominal pain of colicky type
Abdominal distension Vomiting Absolute constipation
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strangulation Stangulation – viability at risk – due to external compression(henial ofifices/adhesions/bands)
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Clinical Types high Small bowel obstruction Large bowel obstruction
low Large bowel obstruction
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In high small bowel obstruction
vomiting occurs early Profuse Rapid dehydration Distension is minimal Little evidence of fluid levels on X-Ray
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In low small bowel obstruction
Pain is predominant Central distension Vomiting is delayed Multiple central fluid levels in X-Ray
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In large bowel obstruction
Distension is early Pronounced distension Pain is mild Vomiting and dehydration are late
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In high small bowel obstructions In low small bowel obstructions
In large bowel obstruction Vomiting occurs early, profuse Vomiting is delayed Vomiting is late Rapid dehydration Pain is predominant dehydration is late Pain is mild Distension is minimal Central distension Distension is early and pronounced Little evidence of fluid levels on X-ray Multiple central fluid levels in X-ray Distension is early Pronounced distension Pain is mild Vomiting and dehydration are late
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Nature of Presentation
Acute obstruction –usually small bowel – sudden onset of severe colicky central abdominal pain, distension with early vomiting and constipation Chronic obstruction – usually large bowel – lower abdominal colic and absolute constipation distension Acute on chronic obstruction – long history of pain Incomplete obstruction
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Acute Intestinal Obstruction
Clinical Features Pain – colicky - severe pain strangulation- pain does not occur in paralytic ileus. Severe pain indicative of strangulation Vomiting – the more distal the obstruction the later it appears Distension – the more distal the more is the distension Constipation – neither faeces nor flatus is passed – this does not apply in Richter’s hernia, gall stone obstruction, mesenteric vascular obstruction, obstruction associated with pelvic abscess, faecal impaction, neoplasm Late manifestations are : dehydration Oliguria Hypovolaemic shock
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Pyrexia Septicaemia Respiratory embarassment Peritonism In all cases of intestinal obstruction all the hernial orifices must be examined
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Clinical Features Pain colicky, abdominal tenderness
Abdominal distension Absolute constipation Discharge of blood in the stools Dehydration , hypotension, shock Fever Dehydration Oliguria Septicaemia Respiratory embarassment Peritonism Constipation - not applicable in Richter’s hernia, gall obturation, mesenteric vascular occlusion, obstruction associated with a pelvic abscess, partial obstruction (faecal impaction /colonic neoplasm)
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Strangulation : shock, pain is never completely absent, symptoms commence suddenly and recur regularly, rigidity and rebound tenderness Generalized tenderness and the presence of rigidity are indicative of the need for early laparotomy. In hernia the lump is tense, tender, irreducible, there is no expansile cough impulse and it has recently increased in size.
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Abdominal distension
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Abdominal distension in intestinal obstruction
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Congen i t a l Megaco L on
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Small and large bowek obstruction
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Small bowel obstruction
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Small bowel obstruction with multiple fluid levels
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Small bowel obstruction upright view
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Hirschprung’s disease
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Congenital megacolon
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In ischaemic colitis
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Intussusception
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Mechanical ileus
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Gas shadow supine view
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Gas-shadowing
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Adynamic ileus
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Investigations Supine abdominal film
Small bowel : straight segments, central, lie transversely no gas in colon Jejunum : valvulae conniventes concertina or ladder effect Ileum : featureless Caecum : a rounded gas shadow in RIF Large bowel : except caecum the rest show haustrations Volvulus of the sigmoid – grossly dilated loop of colon, with or without visible haustrae which arises from the pelvis and extends obliquely across fthe spine to the upper abdomen
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Six
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Six
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six
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Treatment Three main measures: Gastrointestinal drainage
Fluid and electrolytic replacement Relief of obstruction, usually surgical Antibiotics
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Intestinal volvulus – with gangrenous loop
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Volvulus of the intestine
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Hirschprung’s disease
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Congenital megacolon – resected specimen
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Hirscprung’s disease
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intestine worms
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Surgeon’s fingers Surgeon’s fingers The adhesive band is seen at the centre of the circle. Under which an instrument has been passed to make the band prominent. The bands develop between one loop of intestine and the abdominal wall. Six
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Tricho bezoar SIX
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