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Common Pediatric Lower Limb Disorders
Dr.Kholoud Al-Zain Assistant Professor Consultant, Pediatric Orthopedic Surgeon Dec- 2016 Acknowledgement: Dr.Abdalmonem Alsiddiky Dr.Khalid Bakarman Prof. M. Zamzam
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Topics to Cover In-toeing Genu (varus & valgus), & proximal tibia vara
Club foot L.L deformities in C.P patients Limping & leg length inequality Leg aches
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1) Intoeing
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Intoeing- Evaluation Detailed history
Onset, who noticed it, progression Fall a lot How sits on the ground Screening examination (head to toe) Pathology at the level of: Femoral anteversion Tibial torsion Forefoot adduction Wandering big toe
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Intoeing- Asses rotational profile
Pathology Level Special Test Femoral anteversion Tibial torsion Forefoot adduction Wandering big toe Hips rotational profile: Supine Prone Inter-malleolus axis: Foot thigh axis Heel bisector line
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Intoeing- Special Test
Foot Propagation Angle normal is (-10°) to (+15°)
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Intoeing- Femoral Anteversion
Hips rotational profile, supine IR/ER normal = 40-45/45-50°
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Intoeing- Femoral Anteversion
Hips rotational profile prone
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Intoeing- Tibial Torsion
Inter-malleolus axis Supine position Sitting position Inter-malleolus axis
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Intoeing- Tibial Torsion
Foot Thigh Axis normal (0°) to (-10°)
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Intoeing- Tibial Torsion
Foot Thigh Axis normal (0°) to (-10°)
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Intoeing- Forefoot Adduction
Heel bisector line normal along 2 toe
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Intoeing- Adducted Big Toe
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Intoeing- Treatment Establish correct diagnosis Parents education
Annual clinic F/U asses degree of deformity Femoral anti-version sit cross legged Tibial torsion spontaneous improvement Forefoot adduction anti-version shoes, or proper shoes reversal Adducted big toe spontaneous improvement
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Intoeing- Treatment Operative correction indicated for children:
(> 8) years of age With significant cosmetic and functional deformity <1%
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2) Genu Varus & Valgus
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Genu Varum and Genu Valgum
Definition: Bow legs Knock knees
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Normal Genu Varum and Genu Valgum
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Genu Varum and Genu Valgum
Types: Physiological is usually bilateral Pathological can be unilateral
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Genu Varum and Genu Valgum
Types: Physiologic Pathologic
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Genu Varum and Genu Valgum
Evaluation History (detailed) Examination (signs of Rickets) Laboratory Re take the rickets picture
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Genu Varum and Genu Valgum
Re take the rickets picture
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Genu Varum and Genu Valgum
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Genu Varum and Genu Valgum
Evaluation: Imaging Rickets Put mechanical axsis
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Genu Varum and Genu Valgum
Management principles: Non-operative: Physiological usually Pathological must treat underlying cause, as rickets Epiphysiodesis Corrective osteotomies
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“Proximal Tibia Vara”
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Proximal Tibia Vara “Blount disease”: damage of proximal medial tibial growth plate of unknown cause Usually: Overweight Dark skinned Types: Infantile < 3y of age, & usually early walkers Juvenile y, combination Adolescent > 10y, & usually unilateral
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Blount Disease Bilateral Unilateral Types:
Infantile usually in over weight & early walkers Adolescent usually over weight & unilateral
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Blount Disease Staging:
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Blount Disease
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Blount Disease MRI is mandatory: When: Why? Sever cases Recurrence
Is it physis defect
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3) Club Foot
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Clubfoot Etiology Postural fully correctable
Idiopathic (CTEV) partially correctable Secondary (Spina Bifida) rigid deformity, pt needs workup
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Clubfoot Clinical examination Characteristic Deformity : Hind foot:
Equinus (Ankle joint) Varus (Subtalar joint) Mid & fore foot: Forefoot Adduction Cavus
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Clubfoot
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Clubfoot Clinical examination: Deformities don’t prevent walking
Calf muscles wasting Internal torsion of the leg Foot is smaller in unilateral affection Callosities at abnormal pressure areas Short Achilles tendon Heel is high and small No creases behind Heel Abnormal crease in middle of the foot
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A cosmetically pleasing appearance is also an important goal sought by
Clubfoot Management: The goal of treatment for is to obtain a foot that is plantigrade, functional, painless, and stable over time A cosmetically pleasing appearance is also an important goal sought by surgeon and family
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Clubfoot Manipulation and serial casts:
Validity up to 12 months soft tissue becomes more tight Technique “Ponseti” 3 stages, weekly basis (usually by 6-8w)
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Clubfoot Manipulation and serial casts:
Maintaining correction “Dennis Brown Splint” 3-4y old
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Clubfoot Manipulation and serial casts:
Follow up watch and avoid recurrence, till 9y old Avoid false correction by going in sequence When to stop ? not improving, pressure ulcers All pictures put better / Why only to 12 m / when stop
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Clubfoot Indications of surgical treatment:
Late presentation (>12 months of age) Complementary to conservative treatment (residual forefoot adduction) Failure of conservative treatment Recurrence after conservative treatment
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Clubfoot Types of surgery: Soft tissue
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Clubfoot Types of surgery: Bony New pictures
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Clubfoot Types of surgery: If sever, rigid, and in an older child
New pictures
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Clubfoot Types of surgery:
If sever, rigid, and in an older child (salvage)
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4) L.L Deformities in C.P Patients
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Lower Limb Deformities in CP Child
C.P is a non-progressive brain insult that occurred during the peri-natal period. Causes skeletal muscles imbalance that affects joint’s movements. Can be associated with: Mental retardation (various degrees) Hydrocephalus and V.P shunt Convulsions Its not-un-common
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Lower Limb Deformities in CP Child
Physiological classification: Spastic Athetosis Ataxia Rigidity Mixed Topographic classification: Monoplegia Diplegia Paraplegia Hemiplegia Bilateral hemiplegia Triplegia Quadriplegia or tetraplegia
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Lower Limb Deformities in CP Child
Hip Flexion Adduction Internal rotation Knee Ankle Equinus Varus or valgus Gait Intoeing Scissoring
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Lower Limb Deformities in CP Child
Assessment: Gait
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Lower Limb Deformities in CP Child
Assessment: Hips
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Lower Limb Deformities in CP Child
Assessment: Knees
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Lower Limb Deformities in CP Child
Assessment: Ankles
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Lower Limb Deformities in CP Child
Management is multidisciplinary: Parents education Pediatric neurology diagnosis, F/U, treat fits P.T (home & center) joints R.O.M, gait training Orthotics maintain correction, aid in gait Social / Government aid Others: Neurosurgery (V.P shunt), Ophthalmology (eyes sequent), …etc.
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Lower Limb Deformities in CP Child
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Lower Limb Deformities in CP Child
Indications of Orthopedic surgery: Sever contractures preventing P.T P.T plateaued due to contractures Perennial hygiene (sever hips adduction) In a non-walker to sit confortable in wheelchair Prevent: Neuropathic skin ulceration (as feet) Joint dislocation (as hip)
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Lower Limb Deformities in CP Child
Options of Surgery: Tenoplasty Tenotomy Neurectomy Tendon Transfer Bony surgery Osteotomy/Fusion
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5) Limping
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Limping Definition Limping an abnormal gait, Due to:
Pain (where), Deformity (bone or joint), or Weakness (general or nerve or muscle) In one or both limbs
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Limping Diagnosis by: History (detailed) Examination:
Gait good analysis Is it: Above pelvis Back (scoliosis) Below pelvis Hips, knees, ankles, & feet Neuro.Vascular
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Limping Management: Generalization can’t be made.
Treatment of the cause:
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If The Cause Was MSK That Led To Limb Length Inequality
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Limb Length Inequality
True vs. apparent Etiology:
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH Developmental as Blount’s
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH Developmental as Blount’s Traumatic as oblique # (short), or multifragmented (long)
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH Developmental as Blount’s Traumatic as oblique # (short), or multifragmented (long) Infection stunted growth or dissolved part of bone
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH Developmental as Blount’s Traumatic as oblique # (short), or multifragmented (long) Infection stunted growth or dissolved part of bone Metabolic as rickets
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Limb Length Inequality
True vs. apparent Etiology: Congenital as DDH Developmental as Blount’s Traumatic as oblique # (short), or multifragmented (long) Infection stunted growth or dissolved part of bone Metabolic as rickets (unilateral) Tumor affecting physis
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Limb Length Inequality
Adverse effects & clinical picture: Gait disturbance Equinus deformity Pain: back, leg Scoliosis (secondary) Evaluation: Screening examination Clinical measures of discrepancy Imaging methods (Centigram)
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Limb Length Inequality
Management depends on the severity (>2cm): For shorter limb: Shoe raise Bone lengthening For longer limb: Epiphysiodesis (temporary or permanent) Bone shortening
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6) Leg Aches
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Leg Aches What is leg aches? “Growing pain” Benign
In 15 – 30 % of normal children F > M Unknown cause No functional disability, or limping Resolves spontaneously, over several years Growth spert
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Leg Aches Clinical features diagnosis by exclusion H/O: O/E:
At long bones of L.L (Bil) Dull aching, poorly localized Can be without activity At night Of long duration (months) Responds to analgesia O/E: Long bone tenderness nonspecific, large area, or none Normal joints motion
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Leg Aches D.D from serious problems, mainly tumor: Osteoid osteoma
Osteosarcoma Ewing sarcoma Leukemia SCA Subacute O.M
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Leg Aches Management Reassurance Symptomatic: Analgesia (oral, local)
Rest Massage
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Any Question ?
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Remember
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Take Home Message Intoeing is one of 4 causes, treatment depends on the level, mainly observe, operate >8y old Genu varus & valgus phys vs. patho, rickets, when operate Blount early walkers, treatment mainly surgery CTEV 3 types, treat as young as possible, Ponseti better to avoid surgery L.L in C.P mainly treat spastic, PT importance, surgery indications Limping due (pain- week- deformed), above or below pelvis L.L.I proper assess (cause & level), treated >2cm, options of treat Leg aches symptomatic treatment
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