1. Chapter 108: Periodontal Diseases and Oral Bone Loss
Keith L. Kirkwood

2. From the Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 7th Edition.
Table 1. General Classification of Periodontal Diseases and Conditions*

3. Table 2. Diagnosis of Periodontal Diseases
From the Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 7th Edition.
Table 2. Diagnosis of Periodontal Diseases
© 2008 American Society for Bone and Mineral Research

4. Table 3. Nonsurgical Treatment of Periodontitis
From the Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 7th Edition.
Table 3. Nonsurgical Treatment of Periodontitis
© 2008 American Society for Bone and Mineral Research

5. Table 4. Surgical Treatment of Periodontitis
From the Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 7th Edition.
Table 4. Surgical Treatment of Periodontitis
© 2008 American Society for Bone and Mineral Research

6. From the Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 7th Edition.
Figure 1
Figure 1 Etiology of periodontal disease progression and factors that impact the disease progression. Periodontal diseases can occur when periodontal pathogenic bacteria is present in a susceptible host. Genetic and environmental factors modify the host immune response toward bacteria initiating tissue and bone destruction manifested as periodontitis.
Figure 1 Etiology of periodontal disease progression and factors that impact the disease progression. Periodontal diseases can occur when periodontal pathogenic bacteria is present in a susceptible host. Genetic and environmental factors modify the host immune response toward bacteria initiating tissue and bone destruction manifested as periodontitis.
© 2008 American Society for Bone and Mineral Research

7. From the Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 7th Edition.
Figure 2
Figure 2 Anatomy of periodontal tissues and cellular biology of inflammatory bone loss. The periodontal tissues surrounding the tooth include epithelium, connective tissue, periodontal ligament (PDL), and alveolar bone. Inset depicts cell responses to bacterial plaque biofilm on the root surface. Polymorphonuclear lymphocytes (PMNs) and macrophages (M) secrete inflammatory cytokines and matrix metalloproteinases (MMPs) to increase the immune response and degrade connective tissue matrix. Osteoclastogenesis is induced to these stimuli to increase bone resorption through RANKL/RANK system.
Figure 2 Anatomy of periodontal tissues and cellular biology of inflammatory bone loss. The periodontal tissues surrounding the tooth include epithelium, connective tissue, periodontal ligament (PDL), and alveolar bone. Inset depicts cell responses to bacterial plaque biofilm on the root surface. Polymorphonuclear lymphocytes (PMNs) and macrophages (M) secrete inflammatory cytokines and matrix metalloproteinases (MMPs) to increase the immune response and degrade connective tissue matrix. Osteoclastogenesis is induced to these stimuli to increase bone resorption through RANKL/RANK system.
© 2008 American Society for Bone and Mineral Research