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كارشناس تغذیه بالینی معاونت درمان دانشگاه علوم پزشکی قزوین
آشنایی با فرآیند غربالگری و ارزیابی تغذیه ای بیماران بستری در بخش آی سی یو ارائه دهنده: ندا دهناد كارشناس تغذیه بالینی معاونت درمان دانشگاه علوم پزشکی قزوین 94/3/25
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آخرین ویرایش فرم ارزیابی تخصصی تغذیه
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Nutrition Care Process Steps
Nutrition Assessment Nutrition Diagnosis Nutrition Intervention Nutrition Monitoring and Evaluation
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Nutritional care process
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Nutritional Assessment
Anthropometric assessment Clinical evaluation Biochemical, laboratory assessment Dietary evaluation
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ESPEN guidelines Questions to be answered: What is the condition now?
Is the condition stable? Will the condition get worse? Will the disease process accelerate nutritional deterioration?
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Anthropometric methods in ICU
Weight Height estimation Mid-arm circumference Skin fold thickness Head circumference
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Weight
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Ideal Body Weight (kg) Men=48+ 2.3 for each inch over 152 m
Women= for each inch over 152 cm Correction for skeletal size:
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Ideal Body Weight (kg) Add 10% if SS is large
Subtract 10% if SS is small
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Alternative measurements Estimating Height from ulna length
Height in ICU patients Alternative measurements Estimating Height from ulna length
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Estimations of height
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Skin Fold Thickness معرف ميزان چربي زير پوستي و درنتيجه ميزان چاقي خواهد بود. محلهاي اندازه گيري: تريسپس، بايسپس،زير کتف و بالاي تيغه ايلياک . مشکلات عملي: خطاي در اندازه گيري. مشکلات اندازه گيري. وارياسيون توزيع چربي در افراد مختلف (فردي وجمعيتي). حساسيت کم.
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Skin Fold Thickness
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Mid arm circumference measured with a nonstretch measuring tape
midway between the acromion and olecranon of the nondominant arm ≤ 15 cm: severe depletion of muscle mass 16–19 cm: moderate depletion 20–22 cm: mild depletion
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Mid arm circumference
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If MUAC is <23.5 cm, BMI is likely to be <20 kg/m2
BMI estimation If MUAC is <23.5 cm, BMI is likely to be <20 kg/m2 If MUAC is >32.0 cm, BMI is likely to be >30 kg/m2
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CLINICAL ASSESSMENT
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General: muscle wasting
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Wasting in the hands
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The Shoulder and Elbow The shoulder Normal: rounded or sloped
Abnormal: square, can see acromion process The elbow well padded and not showing joint
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The Arm Bend arm and pinch at triceps. Only pinch the fat, not the muscle. Normal: fingers don’t meet Abnormal: fingers meet
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The Legs showing muscle wasting
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Essential fatty acid deficiency syndromes (EFADs)
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Zinc deficiency
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Zinc deficiency
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Vitamine C Deficiency
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Biochemical Assessment
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Serum Protein levels are not reliable during inflammation
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Biochemical markers of nutrition status:
MUST TOOL Is recommended by ESPEN for community 4 steps. Biochemical markers of nutrition status:
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Biochemical markers of nutrition status:
Plasmatic proteins with short biologic half-life Albumin -syntetizate in liver, half-life time is 21 days Normal: 35-45g/l. Decrease of alb: malnutrition Trends of changes alb.levels during realimentation are criterium of succesfull terapy. Acute decrease: acute phase response.
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Biochemical markers of nutrition status:
Transferin: syntesized in liver, biolog HL: 8days. Value 2-4g/l, RBP: syntesized in liver Biolog half-life : 12h., Normal value: 0,03-0,006g/l. Acute phase reactant (negative)
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Biochemical markers of nutrition status:
Prealbumin-syntesized in liver, biolog.half-life:1,5 days. Normal Value 0,15-0,4g/l. Decrease in failure of proteosyntesis-indicator of acute protein malnutrition.
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NUTRITIONAL ASSESSMENT
Urine urea nitrogen (UUN): to evaluate degree of hypermetabolism (stress level): 0 –5 g/d= normometabolism 5 – 10 g/d = mild hypermetabolism (level 1 stress) 10 – 15 = moderate (level 2 stress) >15 = severe (level 3 stress)
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Biochemical parameters
Na,K,Cl,Ca,P,Mg, osmolality - blood, urine Acidobasis, lactate urea, creatinin, creatinin clearence, Nitrogen balance bilirubine, ALT, AST, LDH, amylase, lipase cholesterol, triglycerides, glucose – blood, urine
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Other biochemical parameters
Trace elements /Zn,Se../ Vitamins Drugs /methotrexate, antiepileptics, antibiotics.../ Aminogram /glutamin../ Interleucins,TNF… Hormones /cortisol, glucagone, adrenaline../.
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Monitoring
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Enteral Nutrition Monitoring: Gastric Residuals
Clinically assess the patient for abdominal distension, fullness, bloating, discomfort Place the pt on his/her right side for minutes before checking a RV to avoid cascade effect Seek transpyloric access of feeding tube Raise threshold for RV to mL Consider stopping RV checks in stable pts
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Some Lab tests
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Na serum levels Hypernatremia: Na over 150 mmol/l
hyperaldosteronism hypovolemia renin-angiot-aldost. Hypothalamic damage Hypertonic hyperhydration Diabetes insipidus Brain death
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K serum levels Hypokalemia: K under 3,5mEq/L
Low intake, bigger uptake, or both Emesis, diarrhoe / intestinal loss/ Diuretics Chemotherapy, antimycotics /renal tubules failure/ Anabolic phasis Hyperaldosteronism Acute metabolic alcalosis
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BUN (5-20 mg/dl) Consider hydration and Nutrition. High level of urea
high intake of N, increase catabolism polytrauma-muscele loss GIT bleeding dehydration low output- renal failure, Low level – malnutrition,serious hepatic failure- ureosyntetic cycle and gluconeogenesis dysfunction, pregnancy- increase ECF
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ALT(SGPT) N V: M: 7-46 F:4-35 U/L High level – hepatopathologia,
steatosis, hepatitis, cell damage,
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AST(SGOT) High level – hypoperfusion, hepatitis, cell necrosis, muscles damage both aminotransferases increase during damage of hepatic cells during inf.hepatitis.
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TG(10-190 mg/dl) TG-increase Glycemia, serum, urine,
during sepsis, mainly on the begining, monitorate during parenterál nutrition with lipid emulsion Glycemia, serum, urine, Hypoglycemia below 2,5mmol/l-vital danger hyperglycemia- insulin.rezistence, recomendation level of glycemia 4,5-8,2 /2006/ better survive in ICU patient
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Glucose Glycemia, serum, urine,
Hypoglycemia below 2,5mmol/l-(45 mg/dl) vital danger hyperglycemia- insulin.rezistence Recomendation level of glycemia 4,5-8,2 ( mg/dl)
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P– serum levels(2.7-4.5 mg/dl)
Hypophosphataemia: under 1,9 mg/dl Acute wastage of energy after succesfully resuscitation, overfeeding sy, anabolism (energetic substrates without K,Mg,P). Hyperphosphataemia – over 5,8 mg/dl Renal failure Cell damage
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Urea Urea in urine Increase – catabolism, prerenal failure
Decrease – chronic malnutrition, acute renal failure
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Creatinine( mg/dl) Serum levels of creatinine evaluation together with muscle mass, age, gender Increase bigger offer- destruction of muscle mass, low output-renal failure Decrease- low offer-low muscle mass malnutrition Creatinine clearence, excretion fraction -renal function N-balance – catabolism – the need of nitrogen Uratic acid – cell damage, arthritis uratica
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Monitoring
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Monitoring in PN therapy
Variable to be monitored Initial Later period Weight Daily Weekly serum glucose 3/wk Electrolytes (Na+, K+, Cl-) 1-2//wk BUN Ca+, P,mg Liver function Enzymes Serum triglycerides weekly CBC
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Nutritional screening tools
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Monitoring in PN therapy
Variable to be monitored Initial Later period Clinical status Daily Catetheter site Temperature Intake &Output
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